Graph Analysis Using a GPU-based Parallel Algorithm: Quantum Clustering

The article introduces a new method for applying Quantum Clustering to graph structures. Quantum Clustering (QC) is a novel density-based unsupervised learning method that determines cluster centers by constructing a potential function. In this method, we use the Graph Gradient Descent algorithm to find the centers of clusters. GPU parallelization is utilized for computing potential values. We also conducted experiments on five widely used datasets and evaluated using four indicators. The results show superior performance of the method. Finally, we discuss the influence of $\sigma$ on the experimental results

Content Adaptive NN-Based In-Loop Filter for VVC

The most recent video coding standard VVC contains five in-loop filters to reduce compression artifacts that come from the common drawbacks of block-based hybrid compression framework. However, those traditional in-loop filters are insufficient to deal with the complicated compression artifacts. The emergence of Neural Networks (NNs) has brought significant advancements in the realm of image and video processing, offering a promising avenue for improving video compression. Many prior studies in this domain have focused on training models on large datasets to achieve generalization, rather than catering to specific content characteristics. In this work, we introduced a content-adaptive in-loop filter for Versatile Video Coding (VVC) working with other in-loop filters. The content adaptation is achieved by over-fitting a pre-trained model at the encoder side on the test data. To reduce the bitrate overhead, the Neural Network Compression and Representation (NNR) standard has been introduced which focuses on compressing NNs efficiently. Furthermore, rather than over-fitting all parameters within the NN model, we introduce a set of learnable parameters known as multipliers, which serve to further reduce the bitrate overhead. The proposed model takes auxiliary information including Boundary Strength (BS) and Quantization parameter (QP) as input. Additionally, we have conducted a comprehensive series of experiments to identify the optimal combination of hyperparameters for this approach. The results indicate coding gains of -2.07% (Y), -5.54% (Cb), -1.95% (Cr) Bjøntegaard Delta rate (BD-rate) for Class B and -1.34% (Y), -1.88% (Cb), -0.52% (Cr) Bjøntegaard Delta rate (BD-rate) for Class D with respect to the Peak Signal-to-Noise Ration (PSNR) on top of the Versatile Video Coding (VVC) Test Model (VVC) 12.0 with NN-based Video Coding (NNVC) 5.0, in Random Access (RA) configuration

Generic Event Extraction Using Markov Logic Networks

Master'sMASTER OF SCIENC

Adiponectin protects against paraquat-induced lung injury by attenuating oxidative/nitrative stress.

The specific mechanisms underlying paraquat (PQ)-induced lung injury remain unknown, which limits understanding of its cytotoxic potential. Although oxidative stress has been established as an important mechanism underlying PQ toxicity, multiple antioxidants have proven ineffective in attenuating the deleterious effects of PQ. Adiponectin, which shows anti-oxidative and antinitrative effects, may have the potential to reduce PQ-mediated injury. The present study determined the protective action of globular domain adiponectin (gAd) on PQ-induced lung injury, and attempted to elucidate the underlying mechanism or mechanisms of action. BALB/c mice were administered PQ, with and without 12 or 36 h of gAd pre-treatment. The pulmonary oxidative/nitrative status was assessed by measuring pulmonary O2(•-), superoxide dismutase (SOD), malondialdehyde (MDA), nitric oxide (NO) and 8-hydroxy-2-dydeoxy guanosine (8-OHdG) production, and blood 3-Nitrotyrosine (3-NT). At a dose of 20 mg/kg, PQ markedly increased O2(•-), SOD, MDA, NO and 8-OHdG production 3 h post-administration, but did not significantly increase 3-NT levels until 12 h. gAd inhibited these changes in a dose-dependent manner, via transient activation of MDA, followed by attenuation of MDA formation from 6 h onwards. Histological analysis demonstrated that gAd decreased interstitial edema and inflammatory cell infiltration. These results suggest that gAd protects against PQ-induced lung injury by mitigating oxidative/nitrative stress. Furthermore, gAd may be a potential therapeutic agent for PQ-induced lung injury, and further pharmacological studies are therefore warranted