NEXUS Network: Connecting the Preceding and the Following in Dialogue Generation

Sequence-to-Sequence (seq2seq) models have become overwhelmingly popular in building end-to-end trainable dialogue systems. Though highly efficient in learning the backbone of human-computer communications, they suffer from the problem of strongly favoring short generic responses. In this paper, we argue that a good response should smoothly connect both the preceding dialogue history and the following conversations. We strengthen this connection through mutual information maximization. To sidestep the non-differentiability of discrete natural language tokens, we introduce an auxiliary continuous code space and map such code space to a learnable prior distribution for generation purpose. Experiments on two dialogue datasets validate the effectiveness of our model, where the generated responses are closely related to the dialogue context and lead to more interactive conversations.Comment: Accepted by EMNLP201

Improving Variational Encoder-Decoders in Dialogue Generation

Variational encoder-decoders (VEDs) have shown promising results in dialogue generation. However, the latent variable distributions are usually approximated by a much simpler model than the powerful RNN structure used for encoding and decoding, yielding the KL-vanishing problem and inconsistent training objective. In this paper, we separate the training step into two phases: The first phase learns to autoencode discrete texts into continuous embeddings, from which the second phase learns to generalize latent representations by reconstructing the encoded embedding. In this case, latent variables are sampled by transforming Gaussian noise through multi-layer perceptrons and are trained with a separate VED model, which has the potential of realizing a much more flexible distribution. We compare our model with current popular models and the experiment demonstrates substantial improvement in both metric-based and human evaluations.Comment: Accepted by AAAI201

KN and KbarN Elastic Scattering in the Quark Potential Model

The KN and KbarN low-energy elastic scattering is consistently studied in the framework of the QCD-inspired quark potential model. The model is composed of the t-channel one-gluon exchange potential, the s-channel one-gluon exchange potential and the harmonic oscillator confinement potential. By means of the resonating group method, nonlocal effective interaction potentials for the KN and KbarN systems are derived and used to calculate the KN and KbarN elastic scattering phase shifts. By considering the effect of QCD renormalization, the contribution of the color octet of the clusters (qqbar) and (qqq) and the suppression of the spin-orbital coupling, the numerical results are in fairly good agreement with the experimental data.Comment: 20 pages, 8 figure

Life fingerprints of nuclear reactions in the body of animals

Nuclear reactions are a very important natural phenomenon in the universe. On the earth, cosmic rays constantly cause nuclear reactions. High energy beams created by medical devices also induce nuclear reactions in the human body. The biological role of these nuclear reactions is unknown. Here we show that the in vivo biological systems are exquisite and sophisticated by nature in influence on nuclear reactions and in resistance to radical damage in the body of live animals. In this study, photonuclear reactions in the body of live or dead animals were induced with 50-MeV irradiation. Tissue nuclear reactions were detected by positron emission tomography (PET) imaging of the induced beta+ activity. We found the unique tissue "fingerprints" of beta+ (the tremendous difference in beta+ activities and tissue distribution patterns among the individuals) are imprinted in all live animals. Within any individual, the tissue "fingerprints" of 15O and 11C are also very different. When the animal dies, the tissue "fingerprints" are lost. The biochemical, rather than physical, mechanisms could play a critical role in the phenomenon of tissue "fingerprints". Radiolytic radical attack caused millions-fold increases in 15O and 11C activities via different biochemical mechanisms, i.e. radical-mediated hydroxylation and peroxidation respectively, and more importantly the bio-molecular functions (such as the chemical reactivity and the solvent accessibility to radicals). In practice biologically for example, radical attack can therefore be imaged in vivo in live animals and humans using PET for life science research, disease prevention, and personalized radiation therapy based on an individual's bio-molecular response to ionizing radiation