A limited speech recognition system 2 Final report

Limited speech recognition system for computer voice lin

Speech Communication

Contains report on one research project.U. S. Air Force (Electronics Systems Division) under Contract AF 19(628)-5661National Institutes of Health (Grant 2 ROI NB-04332-06)Joint Services Electronics Programs (U. S. Army, U. S. Navy, and U. S. Air Force) under Contract DA 28-043-AMC-02536(E

Speech Communication

Contains reports on two research projects.National Science Foundation (Grant GK-31353)National Institutes of Health (Grant 5 RO1 NS04332-10)U. S. Navy - Office of Naval Research (Contract N00014-67-A-0204-0069

A limited speech recognition system Final report

Systems analysis, recognition algorithm, and design of limited speech recognition syste

Speech Communication

Contains reports on three research projects.U. S. Air Force Cambridge Research Laboratories under Contract F19628-69-C-0044National Institutes of Health (Grant 5 RO1 NS04332-09)M.I.T. Lincoln Laboratory Purchase Order CC-57

Speech Communication

Contains research objectives, summary of research and reports on one research project.U.S. Air Force Cambridge Research Laboratories, Office of Aerospace Research, under Contract F19628-69-C-0044National Institutes of Health (Grant 2 RO1 NB-04332-07

Speech Communication

Contains reports on four research projects.U. S. Air Force Cambridge Research Laboratories under Contract F19628-69-C-0044National Institutes of Health (Grant 5 RO1 NS 04332-08

Gamma heavy chain disease in a patient with rheumatoid arthritis – a laboratory evaluation

Introduction: Heavy chain diseases (HCD) are neoplastic proliferations of B cells which secrete truncated immunoglobulin heavy chains without associated light chains. Being rare and probably underdiagnosed diseases the aim of this report is to show an additional case of gamma heavy chain disease in a 48 year old female patient with rheumatoid arthritis focusing on the laboratory presentation. Materials and methods: Laboratory work-up included agarose gel electrophoresis (AGE), capillary zone electrophoresis (CZE), immunofixation and nephelometrically determined immunoglobulin and immunoglobulin subclasses of the patient’s serum. Urine samples were also subjected to immunofixation and to a SDS-PAGE with consecutive immunoblot. Results: Nephelometrically measured elevated IgG concentrations were noted in combination with a decreased gamma globulin region and an increased beta globulin region on AGE. A definite monoclonal spike was not identified on AGE but at least suspected on CZE; finally serum and urine immunofixation demonstrated a monoclonal gamma heavy chain devoid of any corresponding light chains confirming the diagnosis of HCD. Analysis of the gamma heavy chain (HC) with means of SDS-PAGE revealed proteins of 40 kD and 80 kD most likely presenting a truncated HC in its monomeric and dimeric form and possibly leading to the failure of IgG-subclass typing with the applied IgG subclass antisera. Conclusion: This case report illustrates a new case of gamma HCD demonstrating variable laboratory manifestations and therefore the need for heightened awareness concerning this disease when confronted with abnormal and discrepant protein profiles in routinely applied laboratory tests

Gneiss-charnockite transformation at Kottavattam, Southern Kerala (India)

At Kottavattam, leucocratic granitic garnet-biotite gneisses (age less than 2 Ga) were partially transformed to coarse-grained charnockite along a system of conjugate fractures (N70E and N20W) and the foliation planes (N60 to 80W; dip 80 to 90 SW) about 550 m.y. ago. To examine and quantify changes in fabric, mineralogy, pore fluids and chemical composition associated with this process, large rock specimens showing gneiss-charnockite transition were studied in detail. The results of the present study corroborate the concept that charnockite formation at Kottavattam is an internally-generated phenomenon and was not triggered by the influx of carbonic fluids from a deep-seated source. It is suggested that charnockitization was caused by the following mechanism: (1) near-isothermal decompression during uplift of the gneiss complex led to an increase of the pore fluid pressure (P sub fluid greater than P sub lith) which - in a regime of anisotropic stress - triggered or at least promoted the development of conjugate fractures; (2) the simultaneous release of pore fluids from bursting fluid inclusions and their escape into the developing fracture system resulted in a drop of fluid pressure; and (3) the internal generation and buffering of the fluids and their, probably, limited migration in an entirely granitic rock system explains the absence of any significant metasomatic mass transfer