Synthesis of perbromates

Salts of heptavalent bromine were synthesized by a hot atom process, the beta decay of radioactive selenium-83 incorporated into a selenate. Formation of an unreactive perbromate ion led to preparation of macro amounts of perborate. A rubidium salt was isolated

A production trial with layers of four hybrids under different environmental conditions in the Eastern Province of Saudi Arabia.

161 Australorp X Rhode Island Red (RIR) X New Hampshire (NH) and 166 White Leghorn (WL) imported fowls were kept in insulated housing; 161 Australorp X RIR X NH, 166 WL, 154 RIR X WL and 148 RIR X NH X RIR fowls were kept in open houses. In the 6 groups resp., live weight at 126 days of age averaged 1602, 1227, 1578, 1234, 1397 and 1539 g, food consumption 4.02, 4.49, 4.03, 4.47, 4.25 and 4.17 kg/kg gain, overall laying percentage 56.6, 56.4, 52.5, 53.4, 55.0 and 37.5, egg production/hen 202, 201, 188, 191, 196 and 134, and egg weight 56.3, 56.3, 55.0, 55.8, 55.0 and 57.5 g. Egg mass averaged 1335, 1323, 1191, 1151, 1126 and 796 kg and food consumed/kg eggs produced 3.68, 3.53, 3.71, 3.25, 3.37 and 4.74 kg. Live weight at 16 mth of age averaged 2894, 2040, 2665, 1874, 2132 and 2517 g, and mortality 12.8, 11.1, 33.6, 43.5, 52.8 and 50.4%. (Abstract retrieved from CAB Abstracts by CABI’s permission

Feeding layers with different levels of energy and protein in the Eastern Province of Saudi Arabia.

Three hundred day-old Single Comb White Leghorn hybrid chicks were kept in insulated units with ventilators and 930 in traditional open-air units under a corrugated galvanised sheet roof in Saudi Arabia. All were fed on a standard diet. Growth in both groups was almost equal, but feed conversion was slightly more favourable in the insulated house. Overall mortality was considered to be favourable. The remaining 995 pullets, when of laying age, were divided into 8 groups, 2 of which were kept in the insulated units, and the rest in open-air units. During the laying period, 4 groups were fed on a high-energy (HC) mash and 4 on low-energy (LC) mash. A 17-h lighting schedule was maintained. Feed intake and energy and protein conversion were higher in the LC groups, while gross feed conversion was better in the HC group and the hens tended to be slightly heavier. There was no difference in egg production between feed groups, but that of hens in insulated units was higher than that of hens in open units. Mortality was higher in the HC group, the main cause being visceral lymphomatosis, and higher in the open housed groups, where 33 died from heat stroke in July, than in the insulated units. The authors conclude that the favourable aspects of the HC feeding were offset by the higher mortality and the higher feeding costs, and so the LC mash might be more profitable. I. F. la C. (Abstract retrieved from CAB Abstracts by CABI’s permission

Sofosbuvir and Daclatasvir Combination Therapy in a Liver Transplant Recipient With Severe Recurrent Cholestatic Hepatitis C

Recurrent HCV infection following liver transplantation can lead to accelerated allograft injury that is difficult to treat with interferon. The aim of this study is to describe the first ever use of an interferon‐free, all oral regimen in a liver transplant recipient with severe recurrent HCV. A 54‐year‐old male with HCV genotype 1b developed severe cholestatic HCV at 6 months posttransplant with ascites, AST 503 IU/mL, alkaline phosphatase of 298 IU/mL, HCV RNA of 12 000 000 IU/mL, and histological cholestasis with pericellular fibrosis. Sofosbuvir, an HCV polymerase inhibitor (400 mg/day), and daclatasvir, an HCV NS5A replication complex inhibitor (60 mg/day), were co‐administered for 24 weeks. Within 4 weeks of initiating treatment, serum HCV RNA levels became undetectable and liver biochemistries normalized with concomitant resolution of ascites. The patient achieved a sustained virological response with undetectable HCV RNA at 9 months posttreatment. During and following treatment, the daily dose and blood level of tacrolimus remained stable and unchanged. The rapid and sustained suppression of HCV replication in this liver transplant recipient provides great promise for the use of combination oral antiviral regimens in other immunosuppressed and interferon refractory HCV patients. A patient with severe cholestatic hepatitis C virus genotype 1b infection at nine months after liver transplantation was successfully treated with a six‐month course of oral sofosbuvir in combination with daclatasvir and remains HCV RNA negative during posttreatment follow‐up with improved liver biochemistries and health.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/98302/1/ajt12209.pd

A Study of the Correlation between Endoscopic and Histological Diagnoses in Gastroduodenitis

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/72433/1/j.1572-0241.1987.tb01777.x.pd

Campylobacter pylori in Patients with Dyspeptic Symptoms and Endoscopic Evidence of Erosion(s)

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/73797/1/j.1572-0241.1989.tb02609.x.pd

Long-Term Follow-Up of Helicobacter pylori Treatment in Non-Ulcer Dyspepsia Patients

Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/73344/1/j.1572-0241.1995.tb09422.x.pd

Spinal Cord Ischemia Related to Disc Herniation:Case Report and a Review of the Literature

Symptoms of spinal cord ischemia can mimic myelopathy due to spinal cord compression in the acute phase. Thoracic disc herniation with limited spinal cord compression but rapid progression of neurological symptoms causes a clinical dilemma as to whether emergency decompression should be performed. We report a case of acute progressive myelopathy due to spinal cord ischemia related to thoracic disc herniation initially managed by Th8 laminectomy with reduction of the herniated disc. Repeat imaging showed T2-weighted hyperintensity in the posterior cord. The clinical and radiological course supports posterior spinal artery ischemia. This case illustrates and a review of the literature shows that thoracic disc herniation may be complicated by ischemic myelopathy even in the absence of cord compression