3-D object mesh geometry compression with vector quantization

IEEE 12th Signal Processing and Communications Applications Conference -- APR 28-30, 2004 -- Kusadasi, TURKEYWOS: 000225861200076In this study, the objective is to develop a new combined method for efficient compression of classical 3-D object mesh representation. This can be realized in two primary steps: Mesh connectivity coding and data (geometry) compression. For realizing the first step, the algorithm of Isenburg [1] has been employed. For the second step, vector quantization methods have been used to compress the vertex coordinate. The difference between our study and the others is that our study uses ECVQ method for vertex coordinate compression to improve the results.IEEE, Tubitak, Istanbul Teknik Univ, Aselsan, Profile Telre, TURCom, Sgi, Datacore, Divi

Deoxycorticosterone acetate-salt mice exhibit blood pressure-independent sexual dimorphism.

We tested the hypothesis that female and male mice differ in terms of cardiac hypertrophy after deoxycorticosterone acetate (DOCA)+salt hypertension (uninephrectomy and 1% saline in drinking water) and focused on calcineurin signaling. We excluded confounding effects of blood pressure elevation or sex-related blood pressure differences by treating DOCA-salt mice with hydralazine (250 mg/L in drinking water). We found that directly measured mean arterial blood pressure was lowered to control values with hydralazine and corroborated this finding in separate mouse groups with radiotelemetry. Male mice were more responsive to DOCA-salt-related effects. They developed more left ventricular hypertrophy and more renal hypertrophy after 6 weeks of DOCA-salt+hydralazine compared with female mice. In hearts, transcripts for calcineurin Abeta and for myocyte-enriched calcineurin interacting protein 1 were upregulated in male but not in female mice. Enhanced activity of calcineurin Abeta, as indicated by diminished phosphorylation of NFATc2 in male mice, accounted for this sex-specific difference. Stretch-related, inflammatory, and profibrotic responses were also accentuated in male mice, as shown by higher transcript levels of atrial natriuretic peptide, monocyte chemoattractant protein-1, and transforming growth factor-beta. Our results support sex-specific regulation of the calcineurin pathway in response to largely blood pressure-independent mineralocorticoid action. We suggest that sex-specific calcineurin activation determines the maladaptive cardiac and renal hypertrophic responses and accompanying organ injury in male mice

Predictors of hepatic steatosis in living liver donors

38th Annual Meeting of the European-Association-for-the-Study-of-the-Liver -- MAR 29-APR 01, 2003 -- ISTANBUL, TURKEYWOS: 000182174500505European Assoc Study Live