26 research outputs found
Chronic Small Vessel Disease Affects Clinical Outcome in Patients with Acute Striatocapsular Stroke
Tribocorrosion behavior and ions release of CoCrMo alloy coated with a TiAlVCN/CNx multilayer in simulated body fluid plus bovine serum albumin
Reversible pathological gambling under treatment with pramipexole [Reversible glückspielsucht unter pramipexol]
In the recent past, pathological gambling has been increasingly reported to be related with dopamine agonist application in patients with Parkinson's disease. We present the case of a female patient whose gambling behavior occurred shortly after reaching the final dose of a combined levodopa and pramipexole therapy and resolved completely after stopping pramipexole. Pathological gambling seems to be a rare and serious but reversible adverse reaction to treatment with dopamine agonists of which clinicians should be aware in patients with Parkinson's disease. © Springer Medizin Verlag 2006
Chronische tiefe Hirnstimulation bei Dystonie: Wiederauftreten dystoner Symptome im Zeitverlauf nach Unterbrechung der Stimulation
Differierende Profile somatosensorisch evozierter Potentiale (Nervus medianus) bei Ableitungen aus dem Pallidum und dem Thalamus bei Patienten mit Dystonie
Rekonstruktion des Felsenbeins zur Behandlung eines kinetischen Tremors nach zerebellärer Herniation und Torsion der efferenten zerebellären Bahnen
Die tiefe Hirnstimulation ist eine effektive Behandlungsalternative bei der segmentalen Dystonie: eine prospektive Serie von 15 Patienten
Patterns of reoccurrence of segmental dystonia after discontinuation of deep brain stimulation
The pattern of reoccurrence of symptoms after discontinuation of deep brain stimulation (DBS) has not been systematically studied in dystonia. Eight patients (mean age (SD) 53.8 (14.4) years) with segmental dystonia at a mean follow‐up of 11.3 (4.2) months were studied after implantation of bilateral DBS electrodes in the internal globus pallidus using a standard video protocol and clinical rating scales, immediately and at 2 and 4 h after switching off DBS. Dystonic signs returned sequentially, with a rapid worsening of phasic and a slower worsening of tonic dystonic components. In all patients, phasic dystonic features appeared within a few minutes, whereas the tonic elements of dystonia reoccurred with a more variable delay. Differential clinical effects when withdrawing DBS might reflect its influence on different pathophysiological mechanisms in dystonia