Learning to Believe in Papua New Guinea

This chapter examines how witchcraft and sorcery beliefs are reproduced among the educated working and middle classes in Papua New Guinea. In a context where tertiary schooling is accessible only to a tiny segment of the population, many educated people in PNG feel anxious about their social position and worry that their upward mobility will provoke envy and resentment in the less fortunate. This anxiety is projected most strongly onto the “ples lain” or rural population, who are thought to maintain many traditional practices, including witchcraft and sorcery. Drawing on ethnographic research among nursing students in the Eastern Highlands, I examine the ways that class identity and Pentecostal social forms coalesce, giving students resources for narrating, understanding, and resisting the dangers they face as social outsiders and (future) employees of a neglectful state. Looking specifically at events during a nursing practicum in rural Eastern Highlands Province, I describe how students and their teachers collapsed different forms of invisible violence—both traditional and contemporary—into a generic evil to be discerned and resisted. Following Robbins (2009) I argue that witchcraft talk is exceptionally socially productive—in this case, productive of a distinctly Christian, professional class identity in which the problems created by “the villagers” and “pasin tumbuna” (ancestral practices) are objects of profound concern.falseAccepte

L'anthropologie psychanalytique. Un paradigme marginal

Gillison Gillian. L'anthropologie psychanalytique. Un paradigme marginal. In: L'Homme, 1999, tome 39 n°149. Anthropologie psychanalytique. pp. 43-52

Le Pénis géant Le frère de la mère dans les Hautes Terres de Nouvelle-Guinée

Gillison Gillian. Le Pénis géant Le frère de la mère dans les Hautes Terres de Nouvelle-Guinée. In: L'Homme, 1986, tome 26 n°99. pp. 41-69

Germline Mutation in ATR in Autosomal- Dominant Oropharyngeal Cancer Syndrome

ATR (ataxia telangiectasia and Rad3 related) is an essential regulator of genome integrity. It controls and coordinates DNA-replication origin firing, replication-fork stability, cell-cycle checkpoints, and DNA repair. Previously, autosomal-recessive loss-of-function mutations in ATR have been demonstrated in Seckel syndrome, a developmental disorder. Here, however, we report on a different kind of genetic disorder that is due to functionally compromised ATR activity, which translates into an autosomal-dominant inherited disease. The condition affects 24 individuals in a five-generation pedigree and comprises oropharyngeal cancer, skin telangiectases, and mild developmental anomalies of the hair, teeth, and nails. We mapped the disorder to a ∼16.8 cM interval in chromosomal region 3q22–24, and by sequencing candidate genes, we found that ATR contained a heterozygous missense mutation (c.6431A>G [p.Gln2144Arg]) that segregated with the disease. The mutation occurs within the FAT (FRAP, ATM, and TRRAP) domain—which can activate p53—of ATR. The mutation did not lead to a reduction in ATR expression, but cultured fibroblasts showed lower p53 levels after activation of ATR with hydroxyurea than did normal control fibroblasts. Moreover, loss of heterozygosity for the ATR locus was noted in oropharyngeal-tumor tissue. Collectively, the clinicopathological and molecular findings point to a cancer syndrome and provide evidence implicating a germline mutation in ATR and susceptibility to malignancy in humans