Sasakian metric as a Ricci soliton and related results

We prove the following results: (i) A Sasakian metric as a non-trivial Ricci soliton is null $\eta$-Einstein, and expanding. Such a characterization permits to identify the Sasakian metric on the Heisenberg group $\mathcal{H}^{2n+1}$ as an explicit example of (non-trivial) Ricci soliton of such type. (ii) If an $\eta$-Einstein contact metric manifold $M$ has a vector field $V$ leaving the structure tensor and the scalar curvature invariant, then either $V$ is an infinitesimal automorphism, or $M$ is $D$-homothetically fixed $K$-contact.Comment: Non

Sasakian Manifolds with Purely Transversal Bach Tensor

We show that a (2n + 1)-dimensional Sasakian manifold (M, g) with a purely transversal Bach tensor has constant scalar curvature ≥2n(2n+1), equality holding if and only if (M, g) is Einstein. For dimension 3, M is locally isometric to the unit sphere S3. For dimension 5, if in addition (M, g) is complete, then it has positive Ricci curvature and is compact with finite fundamental group π1(M)

Bertini theorems revisited

We prove several new Bertini theorems over arbitrary fields and discrete valuation rings.Comment: 37 pages. Substantial revision. Some results were strengthened and new results were adde

Contact Hypersurfaces of a Bochner-Kaehler Manifold

We have studied contact metric hypersurfaces of a Bochner-Kaehler manifold and obtained the following two results: (1) A contact metric constant mean curvature (C M C) hypersurface of a Bochner-Kaehler manifold is a (k, µ)-contact manifold, and (2) If M is a compact contact metric C M C hypersurface of a Bochner-Kaehler manifold with a conformal vector field V that is neither tangential nor normal anywhere, then it is totally umbilical and Sasakian, and under certain conditions on V , is isometric to a unit sphere

Arsenic in Eggs and Excreta of Laying Hens in Bangladesh: A Preliminary Study

The aim of this study was to detect arsenic concentrations in feed, well-water for drinking, eggs, and excreta of laying hens in arsenic-prone areas of Bangladesh and to assess the effect of arsenic-containing feed and well-water on the accumulation of arsenic in eggs and excreta of the same subject. One egg from each laying hen (n=248) and its excreta, feed, and well-water for drinking were collected. Total arsenic concentrations were determined by atomic absorption spectrophotometer, coupled with hydride generator. Effects of arsenic-containing feed and drinking-water on the accumulation of arsenic in eggs and excreta were analyzed by multivariate regression model, using Stata software. Mean arsenic concentrations in drinkingwater, feed (dry weight [DW]), egg (wet weight [WW]), and excreta (DW) of hens were 77.3, 176.6, 19.2, and 1,439.9 ppb respectively. Significant (p<0.01) positive correlations were found between the arsenic contents in eggs and drinking-water (r=0.602), drinking-water and excreta (r=0.716), feed and excreta (r=0.402) as well as between the arsenic content in eggs and the age of the layer (r=0.243). On an average, 55% and 82% of the total variation in arsenic contents of eggs and excreta respectively could be attributed to the variation in the geographic area, age, feed type, and arsenic contents of drinking-water and feed. For each week\u2019s increase in age of hens, arsenic content in eggs increased by 0.94%. For every 1% elevation of arsenic in drinking-water, arsenic in eggs and excreta increased by 0.41% and 0.44% respectively whereas for a 1% rise of arsenic in feed, arsenic in eggs and excreta increased by 0.40% and 0.52% respectively. These results provide evidence that, although high arsenic level prevails in well-water for drinking in Bangladesh, the arsenic shows low biological transmission capability from body to eggs and, thus, the value was below the maximum tolerable limit for humans. However, arsenic in drinking-water and/or feed makes a significant contribution to the arsenic accumulations in eggs and excreta of laying hens

Divergence of transcriptional landscape occurs early in B cell activation

Background: Signaling via B cell receptor (BCR) and Toll-like receptors (TLRs) results in activation of B cells with distinct physiological outcomes, but transcriptional regulatory mechanisms that drive activation and distinguish these pathways remain unknown. Results: Two hours after ligand exposure RNA-seq, ChIP-seq and computational methods reveal that BCR- or TLR-mediated activation of primary resting B cells proceeds via a large set of shared and a smaller subset of distinct signal-selective transcriptional responses. BCR stimulation resulted in increased global recruitment of RNA Pol II to promoters that appear to transit slowly to downstream regions. Conversely, lipopolysaccharide (LPS) stimulation involved an enhanced RNA Pol II transition from initiating to elongating mode accompanied by greater H3K4me3 activation markings compared to BCR stimulation. These rapidly diverging transcriptomic landscapes also show distinct repressing (H3K27me3) histone signatures, mutually exclusive transcription factor binding in promoters, and unique miRNA profiles. Conclusions: Upon examination of genome-wide transcription and regulatory elements, we conclude that the B cell commitment to different activation states occurs much earlier than previously thought and involves a multi-faceted receptor-specific transcriptional landscape. Electronic supplementary material The online version of this article (doi:10.1186/s13072-015-0012-x) contains supplementary material, which is available to authorized users