17 research outputs found
Antimuslimischer Rassismus - ein neues "PhÀnomen"?
Dieser Beitrag geht der Frage nach, ob es sich bei antimuslimischem Rassismus um ein neues PhĂ€nomen handelt und ob sich Konstrukte die sich in der Rassismusforschung bewĂ€hrt haben dazu eignen auch diesen zu erklĂ€ren. Vor dem Hintergrund einer langen Traditionslinie antimuslimischer Ressentiments zeigt sich, dass diese keinesfalls neu sind. Der aktuelle Rassismus gegen Menschen (vermeintlich) muslimischen Glaubens greift tradierte Islambilder auf, indem er diese von jenen der westlichen Welt abgrenzt. Dies wird auch als Neo-Rassismus bezeichnet, welcher sich als Rassismus ohne Rassen konstituiert und die Unvereinbarkeit der Kulturen behauptet. Auch dieser arbeitet mit Stigmatisierungen. Es verschiebt sich demnach der AnknĂŒpfungspunkt des Rassismus. Im Zuge dieser konzeptionellen Verschiebung stellt sich die Frage, inwieweit die in der empirischen Rassismusforschung bewĂ€hrten erklĂ€renden Variablen fĂŒr biologistischen Rassismus sich auch zur ErklĂ€rung eines antimuslimischen Rassismus im Sinne eines Neo-Rassismus eignen. Dies wird mithilfe von multiplen Regressionen auf Daten des ALLBUS 2012 getestet. Dabei bewĂ€hren sich die aus der Empirie entnommenen ErklĂ€rungen nahezu vollstĂ€ndig bei der ErklĂ€rung des antimuslimischen Rassismus, wenngleich sich geringe Abweichungen zeigen und die VarianzaufklĂ€rung geringer ausfĂ€llt. Zwei Strukturgleichungsmodelle bestĂ€tigen dies. Dennoch lassen sich Unterschiede zwischen beiden Rassismen finden - zum Beispiel hinsichtlich der weniger starken Rolle des Autoritarismus und der stĂ€rkeren Rolle des Alters im Modell fĂŒr den antimuslimischen Rassismus.This paper focuses on the question whether antimuslim racism is a new phenomenon and whether empirically confirmed explanations for racism in general are able to explain antimuslim racism, too. Against the background of a long tradition of antimuslim resentments it becomes clear, that these are not new at all. The present racism against Muslims is actually just using traditional resentments to separate Islam from the western world. This can be called a neo-racism, which is supranational and does not make use of biological races like the classic racism, but asserts an incompatibility of cultures. Both racisms have in common, that they are working with stigmatization, but conceptually change the content of stigmatization. We therefore ask whether confirmed explanations of racism are also able to explain antimuslim racism. This is tested by the use of multiple regressions on data of the ALLBUS 2012, which show, that most explanations confirmed by empirical studies on racism work for antimuslim racism, There are a few small deviations and the ratio of explained variance is smaller than for classic racism. Two structural equation models support these findings but also show small differences between classical and antimuslim racism. For example, authoritarianism is less important for the explanation of antimuslim racism, while age gets more important
Colour Passing Revisited: Lifted Model Construction with Commutative Factors
Lifted probabilistic inference exploits symmetries in a probabilistic model
to allow for tractable probabilistic inference with respect to domain sizes. To
apply lifted inference, a lifted representation has to be obtained, and to do
so, the so-called colour passing algorithm is the state of the art. The colour
passing algorithm, however, is bound to a specific inference algorithm and we
found that it ignores commutativity of factors while constructing a lifted
representation. We contribute a modified version of the colour passing
algorithm that uses logical variables to construct a lifted representation
independent of a specific inference algorithm while at the same time exploiting
commutativity of factors during an offline-step. Our proposed algorithm
efficiently detects more symmetries than the state of the art and thereby
drastically increases compression, yielding significantly faster online query
times for probabilistic inference when the resulting model is applied
Basics of collaborative research data management: Requirements for a Schleswig-Holstein state initiative on research data management
Das Papier "Grundlagen eines partnerschaftlichen Forschungsdatenmanagements - Anforderungen an eine schleswig-holsteinische Landesinitiative zum Forschungsdatenmanagement" umreiĂt die Anforderungen fĂŒr eine schleswig-holsteinische Landesinitiative zum Forschungsdatenmanagement (FDM-SH). HierfĂŒr wird zunĂ€chst das Umfeld, in dem eine solche Initiative entstehen und agieren soll, beschrieben. So beeinflussen sowohl die Eigenheiten der regionalen Forschungslandschaft wie auch die Entwicklungen im Bereich der Nationalen Forschungsdateninfrastruktur (NFDI) die AusprĂ€gungen von Landesinitiativen. Die speziellen Anforderungen werden durch den Vergleich mit anderen Landesinitiativen, die Analyse von spezifischen Umfrageergebnissen aus Schleswig-Holstein sowie die BerĂŒcksichtigung der Anforderungen der NFDI gesammelt. Der Ansatz des partnerschaftlichen Forschungsdatenmanagements (FDM) spiegelt das Anliegen Schleswig-Holsteins wider, die Herausforderungen fĂŒr ein zeitgemĂ€Ăes FDM vor Ort gemeinsam zu bewĂ€ltigen und dabei sowohl Know-how zu teilen als auch Ressourcen zu schonen
Recommended from our members
Pathogenic LRRK2 Mutations Do Not Alter Gene Expression in Cell Model Systems or Human Brain Tissue
Point mutations in LRRK2 cause autosomal dominant Parkinson's disease. Despite extensive efforts to determine the mechanism of cell death in patients with LRRK2 mutations, the aetiology of LRRK2 PD is not well understood. To examine possible alterations in gene expression linked to the presence of LRRK2 mutations, we carried out a case versus control analysis of global gene expression in three systems: fibroblasts isolated from LRRK2 mutation carriers and healthy, non-mutation carrying controls; brain tissue from G2019S mutation carriers and controls; and HEK293 inducible LRRK2 wild type and mutant cell lines. No significant alteration in gene expression was found in these systems following correction for multiple testing. These data suggest that any alterations in basal gene expression in fibroblasts or cell lines containing mutations in LRRK2 are likely to be quantitatively small. This work suggests that LRRK2 is unlikely to play a direct role in modulation of gene expression, although it remains possible that this protein can influence mRNA expression under pathogenic cicumstances
Complete congruences on topologies and down-set lattices
From the work of Simmons about nuclei in frames it follows that a topological space X is scattered if and only if each congruence Î on the frame of open sets is induced by a unique subspace A so that Î = {(U, V) | U â© A = V â© A}, and that the same holds without the uniqueness requirement iff X is weakly scattered (corrupt). We prove a seemingly similar but substantially different result about quasidiscrete topologies (in which arbitrary intersections of open sets are open): each complete congruence on such a topology is induced by a subspace if and only if the corresponding poset is (order) scattered, i.e. contains no dense chain. More questions concerning relations between frame, complete, spatial, induced and open congruences are discussed as well