18 research outputs found
Grape Resveratrol Increases Serum Adiponectin and Downregulates Inflammatory Genes in Peripheral Blood Mononuclear Cells: A Triple-Blind, Placebo-Controlled, One-Year Clinical Trial in Patients with Stable Coronary Artery Disease
Interaction of Tertiary Phosphines with Acetylenic Compouxds. Alkyl Migration Accompanied by C-P Bond Cleavage and Fragmentation During the Interaction of Trialkyphosphines with Phenylacetylene in the Presence of Proton Donors
Identification of peptides from autolysates 0f Saccharomyces cerevisiae that exhibit glucose tolerance factor activity in a yeast assay
Reduced meal-related gastrointestinal hormone response to adrenocorticotropic hormone stimulation test in female athletes
Histological changes in insulin-immunoreactive pancreatic ?-cells, and suppression of insulin secretion and somatotrope activity in brook trout (Salvelinus fontinalis) maintained on reduced food intake or exposed to acidic environment
Disposition of a Mixed Meal by Conscious Dogs after Seven Days of Treatment with Cyclosporine A and Prednisone
Epicardial Adipose Tissue in Cardiovascular Disease
Cardiovascular disease (CVD) is the maincause of morbidity and mortality in industrialized countries, despite the evolution of treatments and revascularization strategies.Obesity, also accompanied by a chronicinflammatory process, is an independent riskfactor for CVD. Abdominal adipose tissue is acomplex, metabolically very active organcapable of producing different adipokines andhormones, responsible for endocrinemetabolic comorbidities. The epicardial adipose tissue (EAT) has not been as extensivelystudied as the abdominal or subcutaneous adipose tissue. However, recent evidence associates it with an increased cardiometabolic riskdue to its apposition with the heart. EAT storestriglycerides to provide energy to the myocardium and is characterized by its greater ability to release and capture free fatty acids. EATstrategic localization allows a singular crosstalk with cardiomyocytes and vascular wallcells. The fact that EAT produces proinflammatory adipokines as well as metalloproteinases and pro-oxidant substances,highlights its possible direct impact on plaquevulnerability and heart failure, being still necessary further studies of EAT behavior inCVD.Fil: Berg, Gabriela Alicia. Universidad de Buenos Aires. Facultad de Farmacia y BioquĂmica. Departamento de BioquĂmica ClĂnica; Argentina. Consejo Nacional de Investigaciones CientĂficas y TĂ©cnicas. Oficina de CoordinaciĂłn Administrativa Houssay; ArgentinaFil: Miksztowicz, VerĂłnica Julieta. Universidad de Buenos Aires. Facultad de Farmacia y BioquĂmica. Departamento de BioquĂmica ClĂnica; Argentina. Consejo Nacional de Investigaciones CientĂficas y TĂ©cnicas. Oficina de CoordinaciĂłn Administrativa Houssay; ArgentinaFil: Morales, Celina. Universidad de Buenos Aires. Facultad de Medicina; ArgentinaFil: Barchuk, MagalĂ. Universidad de Buenos Aires. Facultad de Farmacia y BioquĂmica. Departamento de BioquĂmica ClĂnica; Argentina. Consejo Nacional de Investigaciones CientĂficas y TĂ©cnicas. Oficina de CoordinaciĂłn Administrativa Houssay; Argentin