201 research outputs found
Metabolism and conidiation in Neurospora crassa
Metabolism and conidiation in Neurospora crass
The hexosemonophosphate shunt as an alternate metabolic pathway for conidial differentiation in Neurospora
The hexosemonophosphate shunt as an alternate metabolic pathway for conidial differentiation in Neurospor
Further studies on the metabolic control of conidiation of N.crassa
Metabolic control of conidiatio
Conidiation antigen and malate dehydrogenase isoenzyme activities
Conidiation antigen and malate dehydrogenase isoenzyme activitie
Improved techniques for study of caratenoid intermediates
Improved techniques for study of caratenoid intermediate
Preliminary studies on pigmentation and ultrastructure of microconidia of Neurospora crassa
Pigmentation and ultrastructure of microconidi
Construction of two Neurospora crassa Lambda ZapII-cDNA libraries
In the presence of the anti-microtubule agent benomyl, N. crassa synthesizes and secretes a major 60 kDa protein in the culture medium. This protein is absent in untreated cells (Rossier et al. 1989. Eur. J. Cell Biol. 50:333-339). To clone the gene coding for the 60 kDa protein and to unravel its function, we have constructed a cDNA library from benomyl-treated cells ( benomyl cDNA library). A second cDNA library was constructed from untreated cells in order to clone genes coding for cytoskeletal proteins ( standard cDNA library)
New mutants of Neurospora crassa highly resistant to the microtubule inhibitor benomyl
Mutants of N. crassa highly resistant to benomyl with respect to hyphal growth were obtained by mutagenizing wild type strain St. Lawrence 74A or mutant Bml 511(r) which is moderately resistant to the fungicide. One of these, strain E1-91 that has a mutation mapping at the Bml (beta-tubulin) locus, showed temporary sensitivity to the benzimidazole
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