The interference between virtual photon and 1-- charmonium in e+e-experiment

e+e- Experiments producing charmonium are reviewed. It is found that the contribution of the continuum amplitude via virtual photon was neglected in almost all the experiments and the channels analyzed. It is shown that the contribution of the continuum part may affect the final results significantly in psi(2S) and psi(3770) decays, while the interference between continuum and resonance amplitudes may even affect the J/psi decays as well as the psi(2S) and psi(3770). This should be considered in analyzing the "rho-pi puzzle" between J/psi and psi(2s) decays, and the difference between inclusive hadron and DDbar cross sections in psi(3770) decays.Comment: 6 pages, 2 figure

Relative phase between strong and electromagnetic amplitudes in psi(2S) to 0- 0- decays

With the known branching ratios of psi(2S) to pi+pi- and psi(2S) to K+K-, the branching ratio of psi(2S) to K0short K0long is calculated as a function of the relative phase between the strong and the electromagnetic amplitudes of the psi(2S) decays. The study shows that the branching ratio of psi(2S) to K0short K0long is sensitive to the relative phase and a measurement of the K0short K0long branching ratio will shed light on the relative phase determination in psi(2S) to 0- 0- decays.Comment: 5 pages, 1 figur

Semi-inclusive B Decays and Direct CP Violation in QCD Factorization

We have systematically investigated the semi-inclusive B decays B->MX, which are manifestations of the quark decay b->Mq, within the framework of QCD-improved factorization. These decays are theoretically clean and have distinctive experimental signatures. We focus on a class of these that do not require any form factor information and therefore may be especially suitable for extracting information on the angles $\alpha$ and $\gamma$ of the unitarity triangle. The nonfactorizable effects, such as vertex-type and penguin-type corrections to the two-body b decay and hard spectator corrections to the 3-body decay are calculable in the heavy quark limit. QCD factorization is applicable when the emitted meson is a light meson or a charmonium. We discuss the issue of the CPT constraint on partial rate asymmetries. The strong phase coming from final-state rescattering due to hard gluon exchange between the final states can induce large rate asymmetries for tree-dominated color-suppressed modes $(\pi^0,\rho^0,\omega)X_{\bar s}$. The nonfactorizable hard spectator interactions in the 3-body decay, though phase-space suppressed, are extremely important for the tree-dominated modes $(\pi^0,\rho^0,\omega)X_{\bar s}, \phi X$, $J X_s,J X$ and the penguin-dominated mode $\omega X_{s\bar s}$. In fact, they are dominated by the hard spectator corrections. Our result for ${\cal B} (B\to J/\psi X_s)$ is in agreement with experiment. The semi-inclusive decay modes: $B^0_s\to (\pi^0,\rho^0,\omega)X_{\bar s}$, $\rho^0X_{s\bar s}$, $B^0\to(K^-X,K^{*-}X)$ and $B^-\to (K^0X_s,K^{*0}X_s)$ are the most promising ones in searching for direct CP violation. In fact, they have branching ratios of order $10^{-6}-10^{-4}$ and CP rate asymmetries of order $(10-40)$.Comment: 28 page

Influence of cigarette smoking on rate of reopening of the infarct-related coronary artery after myocardial infarction: A multivariate analysis

AbstractObjectives. This study sought to determine whether the reopening of the infarct-related vessel is related to clinical characteristics or cardiovascular risk factors, or both.Background. In acute myocardial infarction, thrombolytic therapy reduces mortality by restoring the patency of the infarct-related vessel. However, despite the use of thrombolytic agents, the infarct-related vessel remains occluded in up to 40% of patients.Methods. We studied 295 consecutive patients with an acute myocardial infarction who underwent coronary angiography within 15 days (mean [±SD] 6.7 ± 3.2 days) of the onset of symptoms. Infarct-related artery patency was defined by Thrombolysis in Myocardial Infarction trial flow grade ≥ 2. Four cardiovascular risk factors—smoking, hypertension, hypercholesterolemin and diabetes mellitus—and eight different variables—age, gender, in-hospital death, history of previous myocardial infarction, location of current myocardial infarction, use of thrombolytic agents, time interval between onset of symptoms, thrombolytic therapy and coronary angiography—were recorded in all patients.Results. Thrombolysis in current smokers and anterior infarct location on admission were the three independent factors highly correlated with the patency of the infarct-related vessel (odds ratios 3.2, 3.0 and 1.9, respectively). In smokers, thrombolytic therapy was associated with a higher reopening rate of the infarct vessel, from 35% to 77% (p < 0.001). Nonsmokers did not benefit from thrombolytic therapy, regardless of infarct location.Conclusions. These observational data, if replicated, suggest that in patients with acute myocardial infarction, thrombolytic therapy may be most effective in current smokers, whereas non-smokers and ex-smokers may require other management strategies, such as emergency percutaneous transluminal coronary angioplasty

Eta-eta' masses and mixing: a large N_c reappraisal

We reconcile the 1/N_c expansion with the observed eta-eta' mass spectrum. The chiral corrections introduced for that purpose are natural and consistent with the octet-singlet mixing angle theta=-(22\pm 1)^{\circ} extracted from phenomenology in the large N_c limit.Comment: 9 pages, 1 figure. Version 2: three new equations added, reference update

Ratio of Hadronic Decay Rates of J\psi and \psi(2S) and the \rho\pi Puzzle

The so-called \rho\pi puzzle of J\psi and \psi(2S) decays is examined using the experimental data available to date. Two different approaches were taken to estimate the ratio of J\psi and \psi(2S) hadronic decay rates. While one of the estimates could not yield the exact ratio of \psi(2S) to J\psi inclusive hadronic decay rates, the other, based on a computation of the inclusive ggg decay rate for \psi(2S) (J\psi) by subtracting other decay rates from the total decay rate, differs by two standard deviations from the naive prediction of perturbative QCD, even though its central value is nearly twice as large as what was naively expected. A comparison between this ratio, upon making corrections for specific exclusive two-body decay modes, and the corresponding experimental data confirms the puzzles in J\psi and \psi(2S) decays. We find from our analysis that the exclusively reconstructed hadronic decays of the \psi(2S) account for only a small fraction of its total decays, and a ratio exceeding the above estimate should be expected to occur for a considerable number of the remaining decay channels. We also show that the recent new results from the BES experiment provide crucial tests of various theoretical models proposed to explain the puzzle.Comment: 8 pages, no figure, 4 table

Charmless hadronic decays B→PP,PV,VVB \to PP, PV, VV and new physics effects in the general two-Higgs doublet models

Based on the low-energy effective Hamiltonian with the generalized factorization, we calculate the new physics contributions to the branching ratios of the two-body charmless hadronic decays of $B_u$ and $B_d$ mesons induced by the new gluonic and electroweak charged-Higgs penguin diagrams in the general two-Higgs doublet models (models I, II and III). Within the considered parameter space, we find that: (a) the new physics effects from new gluonic penguin diagrams strongly dominate over those from the new $\gamma$- and $Z^0$- penguin diagrams; (b) in models I and II, new physics contributions to most studied B meson decay channels are rather small in size: from -15% to 20%; (c) in model III, however, the new physics enhancements to the penguin-dominated decay modes can be significant, $\sim (30 -200)$, and therefore are measurable in forthcoming high precision B experiments; (d) the new physics enhancements to ratios {\cal B}(B \to K \etap) are significant in model III, $\sim (35 -70)$, and hence provide a simple and plausible new physics interpretation for the observed unexpectedly large B \to K \etap decay rates; (e) the theoretical predictions for ${\cal B}(B \to K^+ \pi)$ and ${\cal B}(B \to K^0 \pi^+)$ in model III are still consistent with the data within $2\sigma$ errors; (f) the significant new physics enhancements to the branching ratios of $B \to K^0 \pi^0, K^* \eta, K^{*+} \pi^-, K^+ \phi, K^{*0} \omega, K^{*+} \phi$ and $K^{*0} \phi$ decays are helpful to improve the agreement between the data and the theoretical predictions; (g) the theoretical predictions of ${\cal B}(B \to PP, PV, VV)$ in the 2HDM's are generally consistent with experimental measurements and upper limits ($90$)Comment: 55 pages, Latex file, 17 PS and EPS figures. With minor corrections, final version to be published in Phys.Rev. D. Repot-no: PKU-TH-2000-4

The use of tumour necrosis factor alpha-blockers in daily routine. An Austrian consensus project

To define relevant disease parameters and their respective limits indicating the initiation of TNF-α-blockers in individual patients. Subsequently, to analyze retrospectively patients with rheumatoid arthritis (RA), psoriatic arthritis (PsA) or ankylosing spondylitis (AS), who started TNF-α inhibition in 2006. Points to consider, regarded relevant for individual treatment decisions as well as their assessment methods, were ascertained by experts’ consensus applying the Delphi technique. Subsequently, these parameters’ thresholds with respect to the initiation of a TNF-α-blocker were identified. Thereafter, the rheumatologists representing 12 centres all over Austria agreed to retrospectively analyze their patients started on a TNF-α-blocker in 2006. Experts’ opinion regarding disease parameters relevant to initiate TNF-α-blockers in RA patients only slightly differed from those applied in clinical trials, but the parameters’ threshold values were considerably lower. For PsA patients, some differences and for AS patients, considerable differences between experts’ opinion and clinical studies appeared, which held also true for decisive parameters’ means and thresholds. Six hundred and fifty patients, started on TNF-blockers in 2006, could be analyzed retrospectively, 408 RA patients (53.3 years mean, 340 females), 93 PsA patients (48.9 years mean, 59 males) and 149 AS patients AS (42.2 years mean, 108 males), representing approximately 25% of all Austrian patients initiated on a TNF-blocker in this respective year. Far more individualized, patient-oriented treatment approaches, at least in part, are applied in daily routine compared with those derived from clinical trials or recommendations from investigative rheumatologists