Keragaman Genetik Dan Pendugaan Jumlah Gen Ketahanan Kacang Panjang (Vigna Sinensis L.) Terhadap Penyakit Kuning

Penyakit kuning pada kacang panjang berdampak pada penurunan produksi. Gejala serangan diawali dari gejala daun keriting serta mengakibatkan polong berwarna kuning. Penelitian ini bertujuan mengetahui nilai heritabilitas dan ragam genetik serta menduga jumlah gen pengendali ketahanan kacang panjang terhadap penyakit kuning. Penelitian dilaksanakan di Kabupaten Kediri pada bulan April sampai Juli 2013. Bahan penelitian adalah populasi UB 715 A (P1), Hitam Putih (P2), populasi F1 dan populasi F2. Berdasarkan hasil penelitian, populasi UB 715 A (P1 ) menunjukkan respon tahan terhadap penyakit kuning, populasi Hitam Putih (P2) menunjukkan respon rentan, dan populasi F1 dan F2 menunjukkan respon sedang. Karakter jumlah polong dan jumlah biji per tanaman memiliki keragaman yang sempit sedangkan karakter panjang polong, bobot segar polong, umur berbunga, dan umur panen memiliki keragaman yang luas. Karakter panjang polong dan jumlah biji per polong memiliki nilai heritabilitas rendah, sedangkan karakter jumlah polong, bobot segar polong, umur berbunga, dan umur panen memiliki nilai heritabilitas tinggi. Rasio sifat ketahanan terhadap penyakit kuning pada populasi F2 adalah 9 tahan : 3 sedang : 4 rentan yang berarti ketahanan terhadap penyakit kuning dikendalikan oleh dua gen dengan aksi gen epistasis resesif

The relationship between the reductions in the average value of PSS and the changes in the E-to-Ea ratio in HOCM patients at half a year after a PTSMA procedure.

<p>The relationship between the reductions in the average value of PSS and the changes in the E-to-Ea ratio in HOCM patients at half a year after a PTSMA procedure.</p

Clinical characteristics of study participants.

<p>HOCM, hypertrophic obstructive cardiomyopathy; HR, heart rate; SBP, systolic blood pressure; DBP, diastolic blood pressure; NYHA, New York Heart Association.</p

Comparison of the number of segments having PSS (left) and the average value of PSS (right) between obstructive hypertrophic cardiomyopathy (HOCM) patients before septal ablation and healthy controls.

<p>PTSMA, percutaneous transluminal septal myocardial ablation.</p

Strain curves from the apical 4-chamber view at baseline (A) and half a year after successful septal ablation (B) in a HOCM patient.

<p>The number of segments having PSS was significantly reduced at half a year after PTSMA. <b><i>Arrows</i></b>, Strain curves showing postsystolic shortening (PSS).</p

Comparison of the number of segments having PSS (left) and the average value of PSS (right) between HOCM patients before and half a year after septal ablation.

<p>Comparison of the number of segments having PSS (left) and the average value of PSS (right) between HOCM patients before and half a year after septal ablation.</p

Strain curves during cardiac cycle with (C) and without (A,B) postsystolic shortening (PSS).

<p>AVO, timing of aortic valve opening; AVC, timing of aortic valve closure.</p

Conventional echocardiographic characteristics of healthy controls and HOCM patients at baseline and half a year after PTSMA.

<p>*, P value of comparing between control subjects and HOCM patients at baseline; &, P value of comparing between HOCM patients at baseline and HOCM patients at half a year after PTSMA; HOCM, hypertrophic obstructive cardiomyopathy; LVPW, left ventricular posterior wall; maxLVT,maximal left ventricular thickness;LAV, left atrial volume; LVEF, left ventricular ejection fraction; LVOT, left ventricular outflow tract; DT, E deceleration time; IVRT, isovolumic relaxation time.</p

Mid-term outcomes of biventricular obstruction and left ventricular outflow tract obstruction after surgery correction in child and adolescent patients with hypertrophic cardiomyopathy - Fig 2

<p>Fig 2a. Preoperative two-dimensional transthoracic echocardiography (tte) parasternal long axis (PLAX) views in a 16-year-old hypertrophic cardiomyopathy patient with BVOTO. (A) PLAX view demonstrating the massive septal hypertrophy and the thickening of the ventricular septum bulging into the LVOT and RVOT resulting in biventricular obstructions (the colour flows). (B) Colour Doppler flow imaging of PLAX view during systole showing high velocity jet flow simultaneously in both LVOT and RVOT. Postoperative PLAX views showing a substantial decrease in the ventricular septum thickness and an increase in the RV and LV cavity sizes during diastole (C) and the LV and RV colour flows showing laminar without evidence of significant residual obstructions during systole (D).RV: right ventricle; RVOT: right ventricular outflow tract; IVS: interventricular septum; LV: left ventricle; LA: left atrium; LVOT: left ventricular outflow tract.AO: aorta. Fig 2b. Preoperational cardiovascular magnetic resonance (CMR) image 3-chamber views during diastole (A) and systole (B) showing remarkable myocardial hypertrophy at the base ventricular level with LVOT and RVOT obstruction. The postoperative CMR images (C, D) showing thinner IVS, wider LVOT and RVOT diameter and larger LV and RV cavity without the projection of septum into RVOT or LVOT after biventricular resection. LA: left atrial; LV: left ventricular.</p

Baseline characteristics.

<p>Baseline characteristics.</p