Tropospheric ozone and aerosols measured by airborne lidar during the 1988 Arctic boundary layer experiment

Ozone (O3) and aerosol distributions were measured from an aircraft using a differential absorption lidar (DIAL) system as part of the 1988 NASA Global Tropospheric Experiment - Arctic Boundary Layer Experiment (ABLE-3A) to study the sources and sinks of gases and aerosols over the tundra regions of Alaska during the summer. The tropospheric O3 budget over the Arctic was found to be strongly influenced by stratospheric intrusions. Regions of low aerosol scattering and enhanced O3 mixing ratios were usually correlated with descending air from the upper troposphere or lower stratosphere. Several cases of continental polar air masses were examined during the experiment. The aerosol scattering associated with these air masses was very low, and the atmospheric distribution of aerosols was quite homogeneous for those air masses that had been transported over the ice for greater than or = 3 days. The transition in O3 and aerosol distributions from tundra to marine conditions was examined several times. The aerosol data clearly show an abrupt change in aerosol scattering properties within the mixed layer from lower values over the tundra to generally higher values over the water. The distinct differences in the heights of the mixed layers in the two regions was also readily apparent. Several cases of enhanced O3 were observed during ABLE-3 in conjunction with enhanced aerosol scattering in layers in the free atmosphere. Examples are presented of the large scale variations of O3 and aerosols observed with the airborne lidar system from near the surface to above the tropopause over the Arctic during ABLE-3

Thermal control characteristics of a diffuse bladed specular base louver system Final report

Diffuse bladed specular base louver system for spacecraft temperature contro

Comment on "Valence QCD: Connecting QCD to the Quark Model"

I criticize certain conclusions about the physics of hadrons drawn from a "valence QCD" approximation to QCD.Comment: 12 pages, 8 figures; some minor improvements made to the tex

A MERLIN Observation of PSR B1951+32 and its associated Plerion

In an investigative 16 hour L band observation using the MERLIN radio interferometric array, we have resolved both the pulsar PSR B1951+32 and structure within the flat spectral radio continuum region, believed to be the synchrotron nebula associated with the interaction of the pulsar and its `host' supernova remnant CTB 80. The extended structure we see, significant at $\sim$ 4.5 $\sigma$, is of dimensions 2.5" $\times$ 0.75", and suggests a sharp bow shaped arc of shocked emission, which is correlated with similar structure observed in lower resolution radio maps and X-ray images. Using this MERLIN data as a new astrometric reference for other multiwavelength data we can place the pulsar at one edge of the HST reported optical synchrotron knot, ruling out previous suggested optical counterparts, and allowing an elementary analysis of the optical synchrotron emission which appears to trail the pulsar. The latter is possibly a consequence of pulsar wind replenishment, and we suggest that the knot is a result of magnetohydrodynamic (MHD) instabilities. These being so, it suggests a dynamical nature to the optical knot, which will require high resolution optical observations to confirm.Comment: 12 pages, 2 figures. Accepted for publication in ApJ

Not All Antibodies Are Created Equal: Factors That Influence Antibody Mediated Rejection.

Consistent with Dr. Paul Terasaki's "humoral theory of rejection" numerous studies have shown that HLA antibodies can cause acute and chronic antibody mediated rejection (AMR) and decreased graft survival. New evidence also supports a role for antibodies to non-HLA antigens in AMR and allograft injury. Despite the remarkable efforts by leaders in the field who pioneered single antigen bead technology for detection of donor specific antibodies, a considerable amount of work is still needed to better define the antibody attributes that are associated with AMR pathology. This review highlights what is currently known about the clinical context of pre and posttransplant antibodies, antibody characteristics that influence AMR, and the paths after donor specific antibody production (no rejection, subclinical rejection, and clinical dysfunction with AMR)