5 research outputs found

    Les cançons en els contes de tradició oral. Metanàlisi del repertori tradicional català

    Get PDF
    Aquest article pretĂ©n analitzar les cançons presents en contes tradicionals catalans recollides a les antologies mĂ©s importants de cançó i conte a Catalunya. Inicialment es fa un repĂ s de la importĂ ncia del conte en el desenvolupament de l'infant i del simbolisme associat, per passar a fer un resum dels principals trets diferencials de la cançó tradicional i en concret de la cançó tradicional catalana. A continuaciĂł es procedeix a una metanĂ lisi dels continguts musicals presents a les divuit cançons que figuren dins dels contes, en els aspectes melĂČdics, rĂ­tmics, textuals, formals i mĂštrics. Per acabar es presenten els resultats obtinguts de l'anĂ lisi efectuada, i una bibliografia d'aprofundiment del tema

    Visual Event-Related Potentials in Mild Cognitive Impairment and Alzheimer’s Disease: A Literature Review

    No full text

    Alzheimer's disease and symbiotic microbiota: an evolutionary medicine perspective

    No full text
    Microorganisms resident in our bodies participate in a variety of regulatory and pathogenic processes. Here, we describe how etiological pathways implicated in Alzheimer’s disease (AD) may be regulated or disturbed by symbiotic microbial activity. Furthermore, the composition of symbiotic microbes has changed dramatically across human history alongside the rise of agriculturalism, industrialization, and globalization. We postulate that each of these lifestyle transitions engendered progressive depletion of microbial diversity and enhancement of virulence, thereby enhancing AD risk pathways. It is likely that the human life span extended into the eighth decade tens of thousands of years ago, yet little is known about premodern geriatric epidemiology. We propose that microbiota of the gut, oral cavity, nasal cavity, and brain may modulate AD pathogenesis, and that changes in the microbial composition of these body regions across history suggest escalation of AD risk. Dysbiosis may promote immunoregulatory dysfunction due to inadequate education of the immune system, chronic inflammation, and epithelial barrier permeability. Subsequently, proinflammatory agents—and occasionally microbes—may infiltrate the brain and promote AD pathogenic processes. APOE genotypes appear to moderate the effect of dysbiosis on AD risk. Elucidating the effect of symbiotic microbiota on AD pathogenesis could contribute to basic and translational research
    corecore