Teaching Cultural Studies; Teaching Stuart Hall

I belong to a generation of cultural studies researchers for whom Stuart Hall was not the primary voice defining the field as I first encountered it. He was not even among the first wave of writers that I read or heard discussed as doing ‘cultural studies’. Instead, I came to Hall’s work from a distance defined by the history of cultural studies as a discipline; first by the diffusion of some of its most important interventions through other fields, so that it was not only people who said they were doing cultural studies who were taken up as key to the field, and second by the institutionalisation of a cultural studies canon in which Hall became only one voice, however influential. Nevertheless, by the time Hall died I had come not only to an enthusiastic appreciation of his work but to strongly feel my own indebtedness to it. I want to reflect here on how teaching cultural studies brought me to a close engagement with Hall’s work, and how teaching keeps Hall’s work and ideas alive when the exigencies of academic publishing might relegate him to citational history

Don, Betty and Jackie Kennedy: On Mad Men and Periodisation

Why is it that we watch Mad Men and think it represents a period? Flashes of patterned wallpaper, whiskey neat, babies born that are never mentioned, contact lining for kitchen drawers, Ayn Rand, polaroids, skinny ties, Hilton hotels, Walter Cronkite, and a time when Don Draper can ask ‘What do women want?’ and dry old Roger Sterling can reply ‘Who Cares?’ This essay explores the embrace of period detail in Mad Men finding it to be both loving and fetishistic, and belonging, like all period film, to the politics of the present

1864-04-12 Catherine Driscoll requests husband\u27s enlistment and muster papers

https://digitalmaine.com/cw_me_1st_heavy_corr/1227/thumbnail.jp

Dossier on Govan Young: Exploring children’s historical consciousness through film and archaeology

Govan Young (2017) is a 30-minute documentary in which schoolchildren from Glasgow learn of the area’s important but largely unknown medieval history. This dossier brings together four essays that reflect on the film from various academic perspectives – film studies, archaeology and education – to explore how schoolchildren might learn about the past, and develop a historical consciousness, by participating in film-making projects. The dossier also reflects on how educators can learn from those whom they are supposedly teaching, thereby highlighting that experimental pedagogical projects often bring unexpected learning outcomes into being. Consequently, educators must resist the pressures to predict the outcomes of projects, and must strive to keep the future open-ended

Meaghan

Editorial by Chris Healy, Katrina Schlunke, Prudence Black, Stephen Muecke, and Catherine Driscoll

Urban colonization through multiple genetic lenses: The city‐fox phenomenon revisited

Urbanization is driving environmental change on a global scale, creating novel environments for wildlife to colonize. Through a combination of stochastic and selective processes, urbanization is also driving evolutionary change. For instance, difficulty in traversing human‐modified landscapes may isolate newly established populations from rural sources, while novel selective pressures, such as altered disease risk, toxicant exposure, and light pollution, may further diverge populations through local adaptation. Assessing the evolutionary consequences of urban colonization and the processes underlying them is a principle aim of urban evolutionary ecology. In the present study, we revisited the genetic effects of urbanization on red foxes (Vulpes vulpes) that colonized Zurich, Switzerland. Through use of genome‐wide single nucleotide polymorphisms and microsatellite markers linked to the major histocompatibility complex (MHC), we expanded upon a previous neutral microsatellite study to assess population structure, characterize patterns of genetic diversity, and detect outliers associated with urbanization. Our results indicated the presence of one large evolutionary cluster, with substructure evident between geographic sampling areas. In urban foxes, we observed patterns of neutral and functional diversity consistent with founder events and reported increased differentiation between populations separated by natural and anthropogenic barriers. We additionally reported evidence of selection acting on MHC‐linked markers and identified outlier loci with putative gene functions related to energy metabolism, behavior, and immunity. We concluded that demographic processes primarily drove patterns of diversity, with outlier tests providing preliminary evidence of possible urban adaptation. This study contributes to our overall understanding of urban colonization ecology and emphasizes the value of combining datasets when examining evolutionary change in an increasingly urban world

Sources to Seafood: Mercury Pollution in the Marine Environment

In 2010, the Toxic Metals Superfund Research Program at Dartmouth College brought together a group of 50 scientists and policy stakeholders to form C-MERC, the Coastal and Marine Mercury Ecosystem Research Collaborative. The goal was to review current knowledge—and knowledge gaps—relating to a global environmental health problem, mercury contamination of the world’s marine fish. C-MERC participants attended two workshops over a two-year period, and in 2012 C-MERC authors published a series of peer-reviewed papers in the journals Environmental Health Perspectives and Environmental Research that elucidated key processes related to the inputs, cycling, and uptake of mercury in marine ecosystems, effects on human health, and policy implications. This report synthesizes the knowledge from these papers in an effort to summarize the science relevant to policies being considered at regional, national, and global levels. The Dartmouth Toxic Metals Superfund Research Program uses an interdisciplinary approach to investigate the ways that arsenic and mercury in the environment affect ecosystems and human health. Arsenic and mercury are commonly found in Superfund sites around the U.S. as well as other areas that result in exposures to certain communities. The Research Translation Core of the program communicates program science to government partners, non-governmental organizations, health care providers and associations, universities and the lay community, and facilitates the use of its research for the protection of public health. The Research Translation Core organized the C-MERC effort. The Superfund Research Program of the National Institute of Environmental Health Sciences supports a network of university programs that investigate the complex health and environmental issues associated with contaminants found at the nation’s hazardous waste sites. The Program coordinates with the Environmental Protection Agency and the Agency for Toxic Substances and Disease Registry of the Centers for Disease Control and Prevention, federal entities charged with management of environmental and human health hazards associated with toxic substances

Candidate Sequence Variants and Fetal Hemoglobin in Children with Sickle Cell Disease Treated with Hydroxyurea

Fetal hemoglobin level is a heritable complex trait that strongly correlates with the clinical severity of sickle cell disease. Only few genetic loci have been identified as robustly associated with fetal hemoglobin in patients with sickle cell disease, primarily adults. The sole approved pharmacologic therapy for this disease is hydroxyurea, with effects largely attributable to induction of fetal hemoglobin. In a multi-site observational analysis of children with sickle cell disease, candidate single nucleotide polymorphisms associated with baseline fetal hemoglobin levels in adult sickle cell disease were examined in children at baseline and induced by hydroxyurea therapy. For baseline levels, single marker analysis demonstrated significant association with BCL11A and the beta and epsilon globin loci (HBB and HBE, respectively), with an additive attributable variance from these loci of 23%. Among a subset of children on hydroxyurea, baseline fetal hemoglobin levels explained 33% of the variance in induced levels. The variant in HBE accounted for an additional 13% of the variance in induced levels, while variants in the HBB and BCL11A loci did not contribute beyond baseline levels. These findings clarify the overlap between baseline and hydroxyurea-induced fetal hemoglobin levels in pediatric disease. Studies assessing influences of specific sequence variants in these and other genetic loci in larger populations and in unusual hydroxyurea responders are needed to further understand the maintenance and therapeutic induction of fetal hemoglobin in pediatric sickle cell disease