Forming a name for a doer of an action in the older Croatian Kajkavian lexicography

Hrvatski jezik i kulturu obogaćuju brojna leksikografska djela. U ovome se radu predstavljaju značajke tvorbene razine iz triju rječnika koji su obilježili stariju kajkavsku leksikografiju. Tvorbenom se analizom utvrđuju značajke naziva za vršitelje radnje u rječnicima Dictionar Jurja Habdelića, Gazophylacium Ivana Belostenca te Lexicon latinum Franje Sušnika i Andrije Jambrešića. Na temelju ekscerpiranih primjera utvrđuju se najčešći sufiksi te se donosi zaključak o tvorbenim obilježjima naziva za vršitelje radnje zabilježenima u trima rječnicima te njihov odnos u višečlanim nazivima i atributnim rečenicama.Many lexicographical works enrich Croatian language and culture. This paper presents characteristics of formative level in three dictionaries, which marked earlier Kajkavian lexicography. Formational analysis determines the characteristics of a name for a doer of an action in dictionaries Dictionar by Juraj Habdelić, Gazophylacium by Ivan Belostenec and Lexicon latinum by Franjo Sušnik and Andrija Jambrešić. Based on the excerpted examples, the most common suffixes are determined and a conclusion is made about formative characteristics of a name for a doer of an action which are marked in the three dictionaries, and their relationship in the multiword expressions and attributive clauses

Tvorba naziva za vršitelje radnje u starijoj hrvatskoj kajkavskoj leksikografiji

Hrvatski jezik i kulturu obogaćuju brojna leksikografska djela. U ovome se radu predstavljaju značajke tvorbene razine iz triju rječnika koji su obilježili stariju kajkavsku leksikografiju. Tvorbenom se analizom utvrđuju značajke naziva za vršitelje radnje u rječnicima Dictionar Jurja Habdelića, Gazophylacium Ivana Belostenca te Lexicon latinum Franje Sušnika i Andrije Jambrešića. Na temelju ekscerpiranih primjera utvrđuju se najčešći sufiksi te se donosi zaključak o tvorbenim obilježjima naziva za vršitelje radnje zabilježenima u trima rječnicima te njihov odnos u višečlanim nazivima i atributnim rečenicama

Long-term effects of copper on the structure of freshwater periphyton communities and their tolerance to copper, zinc, nickel and silver. Aquatic Toxicology 47

Abstract A community adapted to elevated ambient levels of a particular pollutant is expected, compared to a non-exposed community, to display an increased tolerance to that pollutant. The potential of tolerance measurements as a method to detect metal-induced structural impacts at the community level is poorly known. Particularly, the determination of increased tolerance to various metals may confound conclusions related to the causes of the impact. In this study the effects of long-term copper exposure on the community structure of freshwater periphyton, and the short-term community tolerance of photosynthesis to copper, zinc, nickel and silver were determined. Using an outdoor flow-through aquaria system, we carried out long-term exposure of freshwater periphyton communities to copper (0, 0.05, 0.1, 0.5, 1 and 5 mM copper). After 12 weeks we examined how the copper exposure affected the taxonomic composition, photosynthesis rate and tolerance thereof to copper, zinc, nickel and silver. Effects included changes in the distribution of algal classes from a community dominated by Cyanophyceae to one dominated by Chlorophyta. The relative abundance of Oocystis nephrocytioides increased from less than 1% in the control aquaria to 56% in the 5 mM copper treatments. Except at the highest copper exposure, communities did not significantly differ in their photosynthesis rate, although the short-term tolerance of photosynthesis to metals was affected by the copper treatments. Significant increases in tolerance to copper were found in communities previously exposed to ] 0.1 mM copper concentrations. Communities exposed to copper also displayed an increased co-tolerance to zinc, nickel and silver. These observations suggest that copper-induced structural impacts on periphyton communities can be evidenced as an increased tolerance to copper. However, because of the occurrence of co-tolerance, the identification of the metals that have induced the structural and tolerance changes may require metal determinations in organisms

GUIDELINES FOR DIAGNOSIS, THERAPY AND FOLLOW UP OF ANDERSON-FABRY DISEASE

Fabryjeva bolest (Anderson-Fabryjeva bolest) jedna je od najčešćih lizosomskih bolesti nakupljanja (nakon Gaucherove bolesti) uzrokovana smanjenom aktivnošću enzima α-galaktosidaze A (α -Gal A) uz posljedično nakupljanje globotriaozilceramida u različitim stanicama, ponajprije u endotelnim i vaskularnim glatkim mišićnim stanicama uz posljedične multisistemske manifestacije. Pojavnost bolesti u muškaraca je 1:40.000-60.000, dok je u općoj populaciji oko 1 : 117.000. Bol je najčešće prvi simptom bolesti u 60-80 % djece, kao i simptomi probavnog sustava, oftalmološki simptomi, gubitak sluha. Smanjenje bubrežne funkcije, hipertrofična miokardiopatija ili moždani udar mogu se iskazati kao izolirani simptomi bolesti. U bolesnika s Fabryjevom bolešću skraćuje se očekivani životni vijek i to u muškaraca za oko 20 godina, a u žena za 10-15 godina, pa je stoga uvođenje nadomjesne enzimske terapije nužno u svih bolesnika koji zadovoljavaju kriterije za terapiju ove bolesti bez obzira na dob i spol.Fabry disease (Anderson-Fabry disease) is one of the most common lysosomal storage diseases (after Gaucher disease) caused by deficient activity of the α-galactosidase A (α-Gal A) enzyme, which leads to progressive accumulation of globotriaosylceramide in various cells, predominantly in endothelium and vascular smooth muscles, with multisystem clinical manifestations. Estimates of the incidence range from one per 40,000 to 60,000 in males, and 1:117,000 in the general population. Pain is usually the first symptom and is present in 60%-80% of affected children, as well as gastrointestinal disturbances, ophthalmologic abnormalities and hearing loss. Renal failure, hypertrophic cardiomyopathy, or stroke as the presenting symptom may also be found even as isolated symptoms of the disease. Life expectancy is reduced by approximately 20 years in males and 10-15 years in females, therefore enzyme replacement therapy should be introduced in patients of any age and either sex, who meet treatment criteria for Anderson-Fabry disease

Smjernice za dijagnosticiranje, terapiju i praćenje Anderson-Fabryjeve bolesti

Fabry disease (Anderson-Fabry disease) is one of the most common lysosomal storage diseases (after Gaucher disease) caused by deficient activity of the α-galactosidase A (α-Gal A) enzyme, which leads to progressive accumulation of globotriaosylceramide in various cells, predominantly in endothelium and vascular smooth muscles, with multisystem clinical manifestations. Estimates of the incidence range from one per 40,000 to 60,000 in males, and 1:117,000 in the general population. Pain is usually the first symptom and is present in 60%-80% of affected children, as well as gastrointestinal disturbances, ophthalmologic abnormalities and hearing loss. Renal failure, hypertrophic cardiomyopathy, or stroke as the presenting symptom may also be found even as isolated symptoms of the disease. Life expectancy is reduced by approximately 20 years in males and 10-15 years in females, therefore enzyme replacement therapy should be introduced in patients of any age and either sex, who meet treatment criteria for Anderson-Fabry disease.Fabryjeva bolest (Anderson-Fabryjeva bolest) je jedna od najčešćih lizosomskih bolesti nakupljanja (nakon Gaucherove bolesti) uzrokovana smanjenom aktivnošću enzima α-galaktosidaze A (α -Gal A) uz posljedično nakupljanje globotriaozilceramida u različitim stanicama, ponajprije u endotelnim i vaskularnim glatkim mišićnim stanicama uz posljedične multisistemske manifestacije. Pojavnost bolesti u muškaraca je 1:40.000-60.000, dok je u općoj populaciji oko 1:117.000. Bol je najčešće prvi simptom bolesti u 60%-80% djece, kao i simptomi probavnog sustava, oftalmološki simptomi, gubitak sluha. Smanjenje bubrežne funkcije, hipertrofična miokardiopatija ili moždani udar mogu se iskazati kao izolirani simptomi bolesti. Očekivani životni vijek se skraćuje u bolesnika s Fabryjevom bolešću i to u muškaraca za oko 20 godina, a u žena za 10-15 godina, stoga je uvođenje enzimske nadomjesne terapije nužno u svih bolesnika bez obzira na dob i spol koji zadovoljavaju kriterije za terapiju ove bolesti