Localized Entanglement in one-dimensional Anderson model

The entanglement in one-dimensional Anderson model is studied. We show that the pairwise entanglement measured by the average concurrence has a direct relation to the localization length. The numerical study indicates that the disorder significantly reduces the average entanglement, and entanglement distribution clearly displays the entanglement localization. The maximal pairwise entanglement exhibits a maximum as the disorder strength increases,experiencing a transition from increase to decrease. The entanglement between the center of localization and other site decreases exponentially along the spatial direction. Finally,we study effects of disorder on dynamical properties of entanglement.Comment: 5 pages, 6 figure

Effects of Space Charge, Dopants, and Strain Fields on Surfaces and Grain Boundaries in YBCO Compounds

Statistical thermodynamical and kinetically-limited models are applied to study the origin and evolution of space charges and band-bending effects at low angle [001] tilt grain boundaries in YBa$_2$Cu$_3$O$_7$ and the effects of Ca doping upon them. Atomistic simulations, using shell models of interatomic forces, are used to calculate the energetics of various relevant point defects. The intrinsic space charge profiles at ideal surfaces are calculated for two limits of oxygen contents, i.e. YBa$_2$Cu$_3$O$_6$ and YBa$_2$Cu$_3$O$_7$. At one limit, O$_6$, the system is an insulator, while at O$_7$, a metal. This is analogous to the intrinsic and doping cases of semiconductors. The site selections for doping calcium and creating holes are also investigated by calculating the heat of solution. In a continuum treatment, the volume of formation of doping calcium at Y-sites is computed. It is then applied to study the segregation of calcium ions to grain boundaries in the Y-123 compound. The influences of the segregation of calcium ions on space charge profiles are finally studied to provide one guide for understanding the improvement of transport properties by doping calcium at grain boundaries in Y-123 compound.Comment: 13 pages, 5 figure

Bipartite entanglement and localization of one-particle states

We study bipartite entanglement in a general one-particle state, and find that the linear entropy, quantifying the bipartite entanglement, is directly connected to the paricitpation ratio, charaterizing the state localization. The more extended the state is, the more entangled the state. We apply the general formalism to investigate ground-state and dynamical properties of entanglement in the one-dimensional Harper model.Comment: 4 pages and 3 figures. Version

Open Charm Production at STAR

We present the open charm spectra at mid-rapidity from direct reconstruction of D0, D* and D+/- in d+Au collisions at sqrt(sNN)=200 GeV using the STAR detector at RHIC. The indirect electron/positron measurements via charm semileptonic decays in p+p and d+Au collisions are also reported. The total c\bar(c) cross section per nucleon-nucleon collision is extracted from both direct and indirect measurements and are consistent with each other. By combining the D0 and semileptonic measurements together, the cross section of 1.4+/-0.2+/-0.4 mb is higher than expectations from PYTHIA and other pQCD calculations. The open charm pT distribution from direct measurements covers the pT range up to ~10 GeV/c and follows a power-law distribution.Comment: 7 pages, 4 figures, proceedings for Hot Quark 04 Conference. submitted to J. Phys. G: Nucl. Phy

K∗(892)K^{*}(892) Production in Au+Au and pp Collisions at sNN\sqrt{s_{NN}} = 200GeV at STAR

Mid-rapidity $K^{*0}(892)\to K\pi$ and $K^{*\pm}(892)\to K_S^0\pi^{\pm}$ are measured in Au+Au and pp collisions at $\sqrt{s_{NN}}$=200GeV using the STAR detector at RHIC. The $K^{*0}(892)$ mass is systematically shifted at small transverse momentum for both Au+Au and pp collisions. The $K^{*0}(892)$ transverse mass spectra are measured in Au+Au collisions at different centralities and in pp collisions. The $K^{*0}(892)$ mean transverse momentum as a function of the collision centrality is compared to those of identified $\pi^{-}$, $K^{-}$ and $\bar{p}$. The $K^{*}/K$ and $\phi/K^{*}$ ratios are compared to measurements in A+A, $pp$, $\bar{p}p$, $e^{+}e^{-}$ collisions at various colliding energies. The physics implications of these measurements are also discussed.Comment: 6 pages, 4 figures, proceedings of Strange Quarks in Matter (SQM2003), Atlantic Beach, USA, to be published in J. Phys.

B --> Phi K_S and Supersymmetry

The rare decay B --> Phi K_S is a well-known probe of physics beyond the Standard Model because it arises only through loop effects yet has the same time-dependent CP asymmetry as B --> Psi K_S. Motivated by recent data suggesting new physics in B --> Phi K_S, we look to supersymmetry for possible explanations, including contributions mediated by gluino loops and by Higgs bosons. Chirality-preserving LL and RR gluino contributions are generically small, unless gluinos and squarks masses are close to the current lower bounds. Higgs contributions are also too small to explain a large asymmetry if we impose the current upper limit on B(B_s --> mu mu). On the other hand, chirality-flipping LR and RL gluino contributions can provide sizable effects and while remaining consistent with related results in B --> Psi K_S, Delta M_s, B --> X_s gamma and other processes. We discuss how the LR and RL insertions can be distinguished using other observables, and we provide a string-based model and other estimates to show that the needed sizes of mass insertions are reasonable.Comment: 33 pages, 32 figures, Updated version for PRD. Includes discussions of other recent works on this topic. Added discussions & plots for gluino mass dependence and effects of theoretical uncertaintie

A combinatorial TIR1/AFB–Aux/IAA co-receptor system for differential sensing of auxin

The plant hormone auxin regulates virtually every aspect of plant growth and development. Auxin acts by binding the F-box protein transport inhibitor response 1 (TIR1) and promotes the degradation of the AUXIN/INDOLE-3-ACETIC ACID (Aux/IAA) transcriptional repressors. Here we show that efficient auxin binding requires assembly of an auxin co-receptor complex consisting of TIR1 and an Aux/IAA protein. Heterologous experiments in yeast and quantitative IAA binding assays using purified proteins showed that different combinations of TIR1 and Aux/IAA proteins form co-receptor complexes with a wide range of auxin-binding affinities. Auxin affinity seems to be largely determined by the Aux/IAA. As there are 6 TIR1/AUXIN SIGNALING F-BOX proteins (AFBs) and 29 Aux/IAA proteins in Arabidopsis thaliana, combinatorial interactions may result in many co-receptors with distinct auxin-sensing properties. We also demonstrate that the AFB5–Aux/IAA co-receptor selectively binds the auxinic herbicide picloram. This co-receptor system broadens the effective concentration range of the hormone and may contribute to the complexity of auxin response

Inhibition of Histone Deacetylase Activity in Human Endometrial Stromal Cells Promotes Extracellular Matrix Remodelling and Limits Embryo Invasion

Invasion of the trophoblast into the maternal decidua is regulated by both the trophoectoderm and the endometrial stroma, and entails the action of tissue remodeling enzymes. Trophoblast invasion requires the action of metalloproteinases (MMPs) to degrade extracellular matrix (ECM) proteins and in turn, decidual cells express tissue inhibitors of MMPs (TIMPs). The balance between these promoting and restraining factors is a key event for the successful outcome of pregnancy. Gene expression is post-transcriptionally regulated by histone deacetylases (HDACs) that unpacks condensed chromatin activating gene expression. In this study we analyze the effect of histone acetylation on the expression of tissue remodeling enzymes and activity of human endometrial stromal cells (hESCs) related to trophoblast invasion control. Treatment of hESCs with the HDAC inhibitor trichostatin A (TSA) increased the expression of TIMP-1 and TIMP-3 while decreased MMP-2, MMP-9 and uPA and have an inhibitory effect on trophoblast invasion. Moreover, histone acetylation is detected at the promoters of TIMP-1 and TIMP-3 genes in TSA-treated. In addition, in an in vitro decidualized hESCs model, the increase of TIMP-1 and TIMP-3 expression is associated with histone acetylation at the promoters of these genes. Our results demonstrate that histone acetylation disrupt the balance of ECM modulators provoking a restrain of trophoblast invasion. These findings are important as an epigenetic mechanism that can be used to control trophoblast invasion

Formation Mechanisms of Large‐Scale Folding in Greenland's Ice Sheet

Radio‐echo sounding (RES) shows large‐scale englacial stratigraphic folds are ubiquitous in Greenland's ice sheet. However, there is no consensus yet on how these folds form. Here, we use the full‐Stokescode Underworld2 to simulate ice movements in three‐dimensional convergent flow, mainly considering iceanisotropy due to a crystallographic preferred orientation, vertical viscosity and density gradients in ice layers,and bedrock topography. Our simulated folds show complex patterns and are classified into: large‐scale folds(>100 m amplitude), small‐scale folds (<<100 m) and basal‐shear folds. The amplitudes of large‐scale foldstend to be at their maximum in the middle of the ice column or just below, in accordance with observations inRES data. We conclude that ice anisotropy amplifies the perturbations in ice layers (mainly due to bedrock topography) into large‐scale folds during flow. Density differences between the warm deep ice and cold iceabove may enhance fold amplification