Effects of Nitrogen and Phosphorus Input on Lake Louise, Dallas, Luzerne County, PA

The runoff of nitrogen and phosphorus can be detrimental to a lakes health and ability to support life (Baker, et al. 2007). Eutrophication is a product of large amounts of nitrogen and phosphorous in lakes (Rice and Horgan, 2017). The purpose of this study is to examine if large amounts of nitrogen and phosphorus are present in Lake Louise. The proposed research question is: Are there high concentrations of nitrogen and phosphorus in and around Lake Louise? The null hypothesis of this study is that there will be no significant difference in nitrogen and phosphorus concentrations in areas in and around Lake Louise. The data gathered allowed us to reject the null that there will be no significant difference in nitrogen and phosphorus concentrations in areas in and around Lake Louise. The data shows a trend that the fluctuations in pH, DO, NO3, and NH4 are affected by the time of year, rather than the site. More sampling and gathering of data must be done to properly conclude why Lake Louise is having algae and sediment issues.https://digitalcommons.misericordia.edu/research_posters2021/1056/thumbnail.jp

A reversible light- and genotype-dependent acquired thermotolerance response protects the potato plant from damage due to excessive temperature

A powerful acquired thermotolerance response in potato was demonstrated and characterised in detail, showing the time course required for tolerance, the reversibility of the process and requirement for light. Potato is particularly vulnerable to increased temperature, considered to be the most important uncontrollable factor affecting growth and yield of this globally significant crop. Here, we describe an acquired thermotolerance response in potato, whereby treatment at a mildly elevated temperature primes the plant for more severe heat stress. We define the time course for acquiring thermotolerance and demonstrate that light is essential for the process. In all four commercial tetraploid cultivars that were tested, acquisition of thermotolerance by priming was required for tolerance at elevated temperature. Accessions from several wild-type species and diploid genotypes did not require priming for heat tolerance under the test conditions employed, suggesting that useful variation for this trait exists. Physiological, transcriptomic and metabolomic approaches were employed to elucidate potential mechanisms that underpin the acquisition of heat tolerance. This analysis indicated a role for cell wall modification, auxin and ethylene signalling, and chromatin remodelling in acclimatory priming resulting in reduced metabolic perturbation and delayed stress responses in acclimated plants following transfer to 40 °C

Ozone-induced oxidative burst in the ozone biomonitor plant, tobacco Bel W3

Localized cell death is a common feature of ozone phytotoxicity and is generally thought to be initiated by the strong oxidant ozone itself as well as by ozone-derived reactive oxygen intermediates (ROIs). Here we report that ozone (150 nl l(-1), 5 h) elicits cellular ROI production in the ozone-sensitive tobacco cv. Bel W3, but not in the tolerant cv. Bel B. Both cultivars exhibited a transient first maximum of apoplastic ROI accumulation followed by a comparable induction of glutathione peroxidase transcript levels. During postcultivation in pollutant-free air, a second and sustained peak of apoplastic ROI accumulation was detected only in cv. Bel W3. Histochemical staining revealed a spot-like accumulation of H2O2 and, to a lesser extent, of superoxide anion radicals in this cultivar. The H2O2 spots ('burst initiation sites') occurred mainly in the vicinity of leaf veins and correlated in number and distribution with discrete sites of local cell death and with visible symptoms that evolved between 15 and 72 h. The results indicate that ozone effects are amplified in the sensitive tobacco cv. Bel W3 by an oxidative burst which participates in the generation of hypersensitive cell deathlike lesions