Clostridium perfringens

Sólo visión proyectable

TREPONEMA PALLIDUM

SÓLO VISIÓN PROYECTABLE

Cryptococcus neoformas

Sólo visión proyectable

MYCOPLASMA

SÓLO VISIÓN PROYECTABLE

BORDETELLA PERTUSSIS

SOLO VISION PROYECTABLE

NEISSERIA GONORRHOEAE

SÓLO VISIÓN PROYECTABLE

COMPLEJO PRINCIPAL DE HISTOCOMPATIBILIDAD

SOLO VISION PROYECTABLE

SISTEMA COMPLEMENTO

SOLO VISION PROYECTABLE

Decrease in Sperm Quality due to Infection of Human Papilloma Virus and Chlamydia trachomatis

Male infertility can have different causes, one of which may be the presence of etiologic agents that cause sexually transmitted infections. Among the most important sexually transmitted infections are human papillomavirus and Chlamydia trachomatis, which are as‐ sociated with infertility in females – whether they cause infertility in men is controversial. The purpose of the chapter is to review the effect of these two pathogens on male fertility, the evidence suggests that the most important infertility effect is linked to the condition of the sperm. However, it is noteworthy that there are few studies with respect to infertil‐ ity in regard to both pathogens, so it is important to further research this to elucidate the mechanisms by which these pathogens act on male infertility.UNIVERSIDAD AUTÓNOMA DEL ESTADO DE MÉXIC

Envolvimento da imunidade inata na infecção do Papilomavírus Humano

Innate immunity is the first defense in the host and is crucial to prevent Human Papillomavirus (HPV) infection and subse - quent development to neoplasia. HPV is involved in the squamous intraepithelial lesions and cervical cancer development. Fortunately, not everyone infected with HPV develop cervical cancer as the immune system can control the virus infection. Innate immunity is the first defense in the host and is crucial to prevent HPV infection and subsequent development to neoplasia. This type of immunity has different antiviral defense pathways, among which are: physical barriers (skin and mucous membranes), keratinocytes and immune cells such as Langerhans cells, dendritic cells, and natural killer cells, which may secrete interferons and cytokines that activate signaling cascades that may prevent HPV infection. However, HPV has also developed various strategies to evade this immune response, which could allow the squamous intraepithelial lesions and cervical cancer development. The aim of this review was to describe the innate immunity involvement in HPV infection