Regions Based Semi-fragile Watermarking Scheme

In this paper, we propose a new semi-fragile watermarking scheme in the frequency domain for surveillance videos authentication. Our system starts operating by generating a binary watermark based on a novel watermark construction process. This latter combines Speeded Up Robust Features (SURF) and Maximally Stable Extremal Regions (MSER) detectors to extract frames relevant features that can resist common attacks while being fragile to intentional manipulations. Furthermore, the watermark security is improved using torus automorphism mapping. For the embedding process, Regions of Interest (ROI) are detected and then used as watermark holders. These regions are decomposed into different frequency sub-bands using Singular Value Decomposition (SVD) as well as Discrete Wavelet Transform (DWT). Then, the watermark is embedded in selected bands following an additive method. A blind detection is conducted to extract the hidden signature from the watermarked video. Evaluation results show that the proposed scheme is suitable for authentication purpose since it efficiently discriminates malicious manipulations from non-malicious ones. Besides, it preserves a high level of perceptual quality

Cytogenetic and molecular diagnosis of Fanconi anemia revealed two hidden phenotypes: Disorder of sex development and cerebro‐oculo‐facio‐skeletal syndrome

International audienceBackground: Several studies have shown a high rate of consanguinity and endogamy in North African populations. As a result, the frequency of autosomal recessive diseases is relatively high in the region with the co-occurrence of two or more diseases.Methods: We report here on a consanguineous Libyan family whose child was initially diagnosed as presenting Fanconi anemia (FA) with uncommon skeletal deformities. The chromosome breakage test has been performed using mitomycin C (MMC) while molecular analysis was performed by a combined approach of linkage analysis and whole exome sequencing.Results: Cytogenetic analyses showed that the karyotype of the female patient is 46,XY suggesting the diagnosis of a disorder of sex development (DSD). By looking at the genetic etiology of FA and DSD, we have identified p.[Arg798*];[Arg798*] mutation in FANCJ (OMIM #605882) gene responsible for FA and p.[Arg108*];[Arg1497Trp] in EFCAB6 (Gene #64800) gene responsible for DSD. In addition, we have incidentally discovered a novel mutation p.[Gly1372Arg];[Gly1372Arg] in the ERCC6 (CSB) (OMIM #609413) gene responsible for COFS that might explain the atypical severe skeletal deformities.Conclusion: The co-occurrence of clinical and overlapping genetic heterogeneous entities should be taken into consideration for better molecular and genetic counselin