Foreshock sequence of the 1992 Landers, California, earthquake and its implications for earthquake nucleation

The June 28, 1992, Landers, California, earthquake(Mw=7.3) was preceded for about 7 hours by a foreshock sequence consisting of at least 28 events. In this study we examine the geometry and temporal development of the foreshocks using high-precision locations based on cross correlation of waveforms recorded at nearby stations. By aligning waveforms, rather than trying to obtain travel time picks for each event independently, we are able to improve the timing accuracy greatly and to make very accurate travel time picks even for emergent arrivals. We perform a joint relocation using the improved travel times and reduce the relative location errors to less than 100m horizontally and less than 200m vertically. With the improved locations the geometry of the foreshock sequence becomes clear. The Landers foreshocks occurred at a fight step of about 500m in the mainshock fault plane. The nucleation zone as defined by the foreshock sequence is southeast trending to the south and nearly north trending to the north of the right step. This geometry is confirmed by the focal mechanisms of the foreshock sequence, which are rightlateral and follow the trend as determined by the foreshock locations on the two straight segments of the fault, and are rotated clockwise for foreshocks that occur within the step. The extent of the foreshock sequence is approximately 1 km both vertically and horizontally. Modeling of the Coulomb stress changes due to all previous foreshocks indicates that the foreshocks probably did not trigger each other. This result is particularly clear for the Mw=4.4 immediate foreshock. Since stress transfer in the sequence appears not to have played a significant role in its development, we infer an underlying aseismic nucleation process, probably aseismic creep. Other studies have shown that earthquake nucleation may be controlled by fault zone irregularities. This appears to be true in the case of the Landers earthquake, although the size of the irregularity is so small that it is not detectable by standard location techniques

Foreshock sequence of the 1992 Landers, California, earthquake and its implications for earthquake nucleation

The June 28, 1992, Landers, California, earthquake(Mw=7.3) was preceded for about 7 hours by a foreshock sequence consisting of at least 28 events. In this study we examine the geometry and temporal development of the foreshocks using high-precision locations based on cross correlation of waveforms recorded at nearby stations. By aligning waveforms, rather than trying to obtain travel time picks for each event independently, we are able to improve the timing accuracy greatly and to make very accurate travel time picks even for emergent arrivals. We perform a joint relocation using the improved travel times and reduce the relative location errors to less than 100m horizontally and less than 200m vertically. With the improved locations the geometry of the foreshock sequence becomes clear. The Landers foreshocks occurred at a fight step of about 500m in the mainshock fault plane. The nucleation zone as defined by the foreshock sequence is southeast trending to the south and nearly north trending to the north of the right step. This geometry is confirmed by the focal mechanisms of the foreshock sequence, which are rightlateral and follow the trend as determined by the foreshock locations on the two straight segments of the fault, and are rotated clockwise for foreshocks that occur within the step. The extent of the foreshock sequence is approximately 1 km both vertically and horizontally. Modeling of the Coulomb stress changes due to all previous foreshocks indicates that the foreshocks probably did not trigger each other. This result is particularly clear for the Mw=4.4 immediate foreshock. Since stress transfer in the sequence appears not to have played a significant role in its development, we infer an underlying aseismic nucleation process, probably aseismic creep. Other studies have shown that earthquake nucleation may be controlled by fault zone irregularities. This appears to be true in the case of the Landers earthquake, although the size of the irregularity is so small that it is not detectable by standard location techniques

Observation of a narrow structure in p(gamma,K_s)X via interference with phi-meson production

We report observation of a narrow peak structure at ~1.54 GeV with a Gaussian width sigma=6 MeV in the missing of K_s in the reaction gamma+p = pK_sK_L. The observed structure may be due to the interference between a strange (or anti-strange) baryon resonance in the pK_L system and the phi(K_sK_L) photoproduction leading to the same final state. The statistical significance of the observed excess of events estimated as the log likelihood ratio of the resonant signal+background hypothesis and the phi-production based background only hypothesis corresponds to 5.3 sigma.Comment: Accepted for publication in Physical Review C, 9 pages, 11 figures, 1 table added, revise

Physical Weight Loading Induces Expression of Tryptophan Hydroxylase 2 in the Brain Stem.

Sustaining brain serotonin is essential in mental health. Physical activities can attenuate mental problems by enhancing serotonin signaling. However, such activity is not always possible in disabled individuals or patients with dementia. Knee loading, a form of physical activity, has been found to mimic effects of voluntary exercise. Focusing on serotonergic signaling, we addressed a question: Does local mechanical loading to the skeleton elevate expression of tryptophan hydroxylase 2 (tph2) that is a rate-limiting enzyme for brain serotonin? A 5 min knee loading was applied to mice using 1 N force at 5 Hz for 1,500 cycles. A 5-min treadmill running was used as an exercise (positive) control, and a 90-min tail suspension was used as a stress (negative) control. Expression of tph2 was determined 30 min – 2 h in three brain regions ––frontal cortex (FC), ventromedial hypothalamus (VMH), and brain stem (BS). We demonstrated for the first time that knee loading and treadmill exercise upregulated the mRNA level of tph2 in the BS, while tail suspension downregulated it. The protein level of tph2 in the BS was also upregulated by knee loading and downregulated by tail suspension. Furthermore, the downregulation of tph2 mRNA by tail suspension can be partially suppressed by pre-application of knee loading. The expression of tph2 in the FC and VMH was not significantly altered with knee loading. In this study we provided evidence that peripheral mechanical loading can activate central tph2 expression, suggesting that physical cues may mediate tph2-cathalyzed serotonergic signaling in the brain

Koinonia

Commission Funding, S. Makin Identity: A View from the Garden, W. Kirwan Resident Hall Improvement Without a Budget, B. Ferro Role Playing / RA Training, J. Barnes Career - Planning and Placement, C. Smith Freshman Roommate Selection, S. Raymond Creative Activities, D. Messenger Student Leadership Development, R.E. Dodgehttps://pillars.taylor.edu/acsd_koinonia/1064/thumbnail.jp

Improving temperature‐based predictions of the timing of flowering in cotton

Key management recommendations for cotton (Gossypium hirsutum L.) management require estimates of the timing of crop phenology. Most commonly growing day degree (DD) (thermal time) approaches are used. Currently, across many cotton production regions, there is no consistent approach to predicting first square and flower timing. Day degree approaches vary considerably, with base thresholds different (12.0–15.6 °C) with no consistency using an optimum temperature threshold (i.e., temperature where development ceases to increase). As cotton is grown in variable and changing climates, and cultivars change, there is a need to ensure the accuracy of this approach for predicting timing of flowering for assisting cotton management. In this study new functions to predict first square and first flower were developed and validated using data collected in multiple seasons and regions (Australia and the United States). Earlier controlled environment studies that monitored crop development were used to assess in more detail how temperatures were affecting early cotton development. New DD functions developed predicted first square and first flower better than the existing Australian and U.S. approaches. The best performing functions had base temperatures like those of existing U.S. functions (15.6 °C) and an optimum threshold temperature of 32.0 °C. New universal DD targets for first square (343 DD [°C]) and first flower (584 DD) were developed. Controlled environment studies supported this base temperature outcome; however, it was less clear that 32.0 °C was the optimum threshold temperature from these data. Precise predictions of cotton development will facilitate accurate growth stage assessments and hence better cotton management decisions

Mediating and Moderating Effects of Iron Homeostasis Alterations on Fetal Alcohol-Related Growth and Neurobehavioral Deficits

We have previously demonstrated prenatal alcohol exposure (PAE)-related alterations in maternal and infant iron homeostasis. Given that early iron deficiency and PAE both lead to growth restriction and deficits in recognition memory and processing speed, we hypothesized that PAE-related iron homeostasis alterations may mediate and/or moderate effects of PAE on growth and neurobehavior. We examined this hypothesis in a prenatally recruited, prospective longitudinal birth cohort [87 mother-infant pairs with heavy prenatal alcohol exposure (mean = 7.2 drinks/occasion on 1.4 days/week); 71 controls], with serial growth measures and infant neurobehavioral assessments. PAE was related to growth restriction at 2 weeks and 5 years, and, in infancy, poorer visual recognition memory, slower processing speed, lower complexity of symbolic play, and higher emotionality and shyness on a parental report temperament scale. Lower maternal hemoglobin-to-log(ferritin) ratio, which we have shown to be associated with PAE, appeared to exacerbate PAE-related 2-week head circumference reductions, and elevated maternal ferritin, which we have shown to be associated with PAE, appeared to exacerbate PAE-related visual recognition memory deficits. In causal inference analyses, PAE-related elevations in maternal ferritin and hemoglobin:log(ferritin) appeared to statistically mediate 22.6–82.3% of PAE-related growth restriction. These findings support potential mechanistic roles of iron homeostasis alterations in fetal alcohol spectrum disorders (FASD)