Tolerância do tamanho da semente de soja (Glycine max (L.) Merr.) ao metribuzin Tolerance of soybeans (Glycine max (L.) merr.) seeds of different sizes to metribuzin

Este experimento foi instalado em casa-de-vegetação com o objetivo de avaliar a tolerância de três tamanhos de sementes de soja (Glycine max (L.) Merr.), cv. UFV-1 classificadas nas peneiras de 10/64" x 3/4", 13/64" x 3/4" e 15/64 x 3/4", a diferentes doses de metribuzin (4-amino-6-tertbutil-3-(metiltio-as-triazina-5(4H) ona ). As doses de 0, 300, 600, 900 e 1.200g do i.a./ha foram aplicadas nos materiais do solo de Ponta Nova-MG classificado como franco-argilo-arenoso, e as doses de 0, 50, 100, 150, 200, 250, 300, 350 e 600g do i.a./ha em areia lavada. Nos materiais de solo de Ponte Nova, a inibição de 50% do crescimento (I50) das plantas de soja correspondeu à aplicação de 600, 1050 e 1150 g/ha de metribuzin, enquanto em areia lavada os I-,,, foram de 150, 210 e 265g/ha de metribuzin para as sementes retidas nas peneiras 10/64" x 3/4", 13/64" x 3/4" e 15/64" x 3/4", respectivamente. O aumento do tamanho da semente aumentou a toleráncia da soja ao metribuzin.<br>The tolerance of soybeans (Glycine max (L.) Merr.) seeds of three different sizes to the herbicide metribuzin (4-amino-6-tert-butil-3-(metiltio)-astriazina-5(4H)ona) was examined in greenhouse. Seeds were separated on 10/64" x 3/4", 13/64" x x 3/4" and 15/64" x 3/4" sieves. Herbicide doses equivalent to 0, 300, 600, 900 and 1.200g a.i./ha were applied to a sandy loam Ponte Nova (M.G.) soil and 0, 50, 100, 150, 200, 250, 300, 350 to 600g a.i./ha to washed sand. The growth of seeds of size 10, 13 and 15 were 50% inhibited (I50) at levels of 600, 1050 and 1150g of metribuzin in the soil and 150, 210 and 265g/ha in the sand. Thus larger seeds showed greater tolerance

Operation of the repeating pneumatic injector on TFTR and design of an 8-shot deuterium pellet injector

The repeating pneumatic hydrogen pellet injector, which was developed at the Oak Ridge National Laboratory (ORNL), has been installed and operated on the Tokamak Fusion Test Reactor (TFTR). The injector combines high-speed extruder and pneumatic acceleration technologies to propel frozen hydrogen isotope pellets repetitively at high speeds. The pellets are transported to the plasma in an injection line that also serves to minimize the gas loading on the torus; the injection line incorporates a fast shutter valve and two stages of guide tubes with intermediate vacuum pumping stations. A remote, stand-alone control and data acquisition system is used for injector and vacuum system operation. In early pellet fueling experiments on TFTR, the injector has been used to deliver deuterium pellets at speeds ranging from 1.0 to 1.5 km/s into plasma discharges. First, single large (nominal 4-mm-dia) pellets provided high densities in TFTR (1.8 x 10/sup 14/ cm/sup -3/ on axis); after conversion to smaller (nominal 2.7-mm-dia) pellets, up to five pellets were injected at 0.25-s intervals into a plasma discharge, giving a line-averaged density of 1 x 10/sup 14/ cm/sup -3/. Operating characteristics and performance of the injector in initial tests on TFTR are presented

D3 Dopamine and Kappa Opioid Receptor Alterations in Human Brain of Cocaine-overdose Victims

Cocaine is thought to be addictive because chronic use leads to molecular adaptations within the mesolimbic dopamine (DA) circuitry, which affects motivated behavior and emotion. Although the reinforcing effects of cocaine are mediated primarily by blockade of DA uptake, reciprocal signaling between DA and endogenous opioids has important implications for understanding cocaine dependence. We have used in vitro autoradiography and ligand binding to map D3 DA and kappa opioid receptors in the human brains of cocaine‐overdose victims. The number of D3 binding sites was increased one‐ to threefold over the nucleus accumbens and ventromedial sectors of the caudate and putamen from cocaine‐overdose victims, as compared to age‐matched and drug‐free control subjects. D3 receptor/cyclophilin mRNA ratios in the nucleus accumbens were increased sixfold in cocaine‐overdose victims over control values, suggesting that cocaine exposure also affects the expression of D3 receptor mRNA. The number of kappa opioid receptors in the nucleus accumbens and other corticolimbic areas from cocaine fatalities was increased twofold as compared to control values. Cocaine‐overdose victims exhibiting preterminal excited delirium had a selective upregulation of kappa receptors measured also in the amygdala. Understanding the complex regulatory profiles of DA and opioid synaptic markers that occur with chronic misuse of cocaine may suggest multitarget strategies for treating cocaine dependence