A Survey of Lepton Number Violation Via Effective Operators

We survey 129 lepton number violating effective operators, consistent with the minimal Standard Model gauge group and particle content, of mass dimension up to and including eleven. Upon requiring that each one radiatively generates the observed neutrino masses, we extract an associated characteristic cutoff energy scale which we use to calculate other observable manifestations of these operators for a number of current and future experimental probes, concentrating on lepton number violating phenomena. These include searches for neutrinoless double-beta decay and rare meson, lepton, and gauge boson decays. We also consider searches at hadron/lepton collider facilities in anticipation of the LHC and the future ILC. We find that some operators are already disfavored by current data, while more are ripe to be probed by next-generation experiments. We also find that our current understanding of lepton mixing disfavors a subset of higher dimensional operators. While neutrinoless double-beta decay is the most promising signature of lepton number violation for the majority of operators, a handful is best probed by other means. We argue that a combination of constraints from various independent experimental sources will help to pinpoint the ``correct'' model of neutrino mass, or at least aid in narrowing down the set of possibilities.Comment: 34 pages, 10 eps figures, 1 tabl

Proposal of a new Hcal geometry avoiding cracks in the calorimeter

The classical geometry of a calorimeter consists most of the time in several modules, whose edges are pointing on the beam axis. Thus, detection discontinuities between two consecutive modules induce cracks in the calorimeter, and consequently a loss of precious information. This paper describes two new possible Hcal geometries avoiding such cracks in the detection. Then it deals with the internal layout and assembly procedure

Detection of myocardial injury during transvenous implantation of automatic cardioverter-defibrillators

AbstractOBJECTIVESThe present study was designed to assess the extent of myocardial injury in patients undergoing transvenous implantation of an automatic implantable cardioverter-defibrillator (ICD) using cardiac troponin I (cTNI), which is a highly specific marker of structural cardiac injury.BACKGROUNDDuring ICD implantation, repetitive induction and termination of ventricular fibrillation (VF) via endocardial direct current shocks is required to demonstrate the correct function of the device. Transthoracic electrical shocks can cause myocardial cell injury.METHODSMeasurements of total creatine kinase (CK), CK-MB, myoglobin, cardiac troponin T (cTNT) and cTNI were obtained before and after ICD implantation in 49 consecutive patients. Blood samples were drawn before and 2, 4, 8, and 24 h after implantation.RESULTSElevations of CK, CK-MB, myoglobin, cTNT and cTNI above cut-off level were found in 25%, 6%, 76%, 37% and 14% of patients, respectively, with peak cTNI concentrations ranging from 1.7 to 5.5 ng/ml. Cumulative defibrillation energy (DFE), mean DFE, cumulative VF time, number of shocks as well as prior myocardial infarction (MI) were found to be significantly related to a rise of cTNI. Mean DFE ≥ 18 J and a recent MI were identified as strong risk factors for cTNI rise.CONCLUSIONSDuring transvenous ICD implantation myocardial injury as assessed by cTNI rise occurs in about 14% of the patients. Peak cTNI concentrations are only minimally elevated reflecting subtle myocardial cell damage. Patients with a recent MI and a mean DFE ≥ 18 J seem to be prone to cTNI rise

Students' mental prototypes for functions and graphs

This research study investigates the concept of function developed by students studying English A-level mathematics. It shows that, while students may be able to use functions in their practical mathematics, their grasp of the theoretical nature of the function concept may be tenuous and inconsistent. The hypothesis is that students develop prototypes for the function concept in much the same way as they develop prototypes for concepts in everyday life. The definition of the function concept, though given in the curriculum, is not stressed and proves to be inoperative, with their understanding of the concept reliant on properties of familiar prototype examples: those having regular shaped graphs, such as x2 or sin*, those often encountered (possibly erroneously), such as a circle, those in which y is defined as an explicit formula in x, and so on. Investigations reveal significant misconceptions. For example, threequarters of a sample of students starting a university mathematics course considered that a constant function was not a function in either its graphical or algebraic forms, and threequarters thought that a circle is a function. This reveals a wide gulf between the concepts as perceived to be taught and as actually learned by the students

SLC-2000: A luminosity upgrade for the SLC

The authors discuss a possible upgrade to the Stanford Linear Collider (SLC), whose objective is to increase the SLC luminosity by at least a factor 7, to an average Z production rate of more than 35,000 per week. The centerpiece of the upgrade is the installation of a new superconducting final doublet with a field gradient of 240 T/m, which will be placed at a distance of only 70 cm from the interaction point. In addition, several bending magnet in each final focus will be lengthened and two octupole correctors are added. A complementary upgrade of damping rings and bunch compressors will allow optimum use of the modified final focus and can deliver, or exceed, the targeted luminosity. The proposed upgrade will place the SLC physics program in a very competitive position, and will also enable it to pursue its pioneering role as the first and only linear collider

The Two Roads to "Intrinsic Charm" in B Decays

We describe two complementary ways to show the presence of higher order effects in the 1/m_Q expansion for inclusive B decays that have been dubbed "Intrinsic Charm". Apart from the lessons they can teach us about QCD's nonperturbative dynamics their consideration is relevant for precise extractions of |V_{cb}|: for they complement the estimate of the potential impact of 1/m_Q^4 contributions. We draw semiquantitative conclusions for the expected scale of Weak Annihilation in semileptonic B decays, both for its valence and non-valence components.Comment: 17 pages, 3 figure

Development of Readout Interconnections for the Si-W Calorimeter of SiD

The SiD collaboration is developing a Si-W sampling electromagnetic calorimeter, with anticipated application for the International Linear Collider. Assembling the modules for such a detector will involve special bonding technologies for the interconnections, especially for attaching a silicon detector wafer to a flex cable readout bus. We review the interconnect technologies involved, including oxidation removal processes, pad surface preparation, solder ball selection and placement, and bond quality assurance. Our results show that solder ball bonding is a promising technique for the Si-W ECAL, and unresolved issues are being addressed.Comment: 8 pages + title, 6 figure

Human C-peptide Dose Dependently Prevents Early Neuropathy in the BB/Wor-rat

In order to explore the neuroprotective and crossspecies activities of.C-peptide on type 1 diabetic neuropathy, spontaneously diabetic BB/W-rats were given increasing doses of human recombinant Cpeptide (hrC-peptide). Diabetic rats received 10, 100, 500, or 1000 μg of hrC-peptide/kg body weight/ day from onset of diabetes. After 2 months of hrC-peptide administration, 100 μg and greater doses completely prevented the nerve conduction defect, which was associated with a significant but incomplete prevention of neural Na+/K+-ATPase activity in diabetic rats with 500 μg or greater C-peptide replacement. Increasing doses of hrC-peptide showed increasing prevention of early structural abnormalities such as paranodal swelling and axonal degeneration and an increasing frequency of regenerating sural nerve fibers. We conclude that hrC-peptide exerts a dose dependent protection on type 1 diabetic neuropathy in rats and that this effect is probably mediated by the partially conserved sequence of the active C-terminal pentapeptid