625 research outputs found

    Dissipationless BCS Dynamics with Large Branch Imbalance

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    In many situations a BCS-type superconductor will develop an imbalance between the populations of the holelike and electronlike spectral branches. This imbalance suppresses the gap. It has been noted by Gal'perin et al. [Sov. Phys. JETP 54, 1126 (1981)] that at large imbalance, when the gap is substantially suppressed, an instability develops. The analytic treatment of the system beyond the instability point is complicated by the fact that the Boltzmann approach breaks down. We study the short-time behavior following the instability, in the collisionless regime, using methods developed by Yuzbashyan et al. [J. Phys. A 38, 7831 (2005); Phys. Rev. B 72, 220503(R) (2005)].Comment: 12 pages, 3 figure

    Quantum Hall transitions: An exact theory based on conformal restriction

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    We revisit the problem of the plateau transition in the integer quantum Hall effect. Here we develop an analytical approach for this transition, based on the theory of conformal restriction. This is a mathematical theory that was recently developed within the context of the Schramm-Loewner evolution which describes the stochastic geometry of fractal curves and other stochastic geometrical fractal objects in 2D space. Observables elucidating the connection with the plateau transition include the so-called point-contact conductances (PCCs) between points on the boundary of the sample, described within the language of the Chalker-Coddington network model. We show that the disorder-averaged PCCs are characterized by classical probabilities for certain geometric objects in the plane (pictures), occurring with positive statistical weights, that satisfy the crucial restriction property with respect to changes in the shape of the sample with absorbing boundaries. Upon combining this restriction property with the expected conformal invariance at the transition point, we employ the mathematical theory of conformal restriction measures to relate the disorder-averaged PCCs to correlation functions of primary operators in a conformal field theory (of central charge c=0c=0). We show how this can be used to calculate these functions in a number of geometries with various boundary conditions. Since our results employ only the conformal restriction property, they are equally applicable to a number of other critical disordered electronic systems in 2D. For most of these systems, we also predict exact values of critical exponents related to the spatial behavior of various disorder-averaged PCCs.Comment: Published versio

    On harmonic measure of critical curves

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    Fractal geometry of critical curves appearing in 2D critical systems is characterized by their harmonic measure. For systems described by conformal field theories with central charge c⩽1c\leqslant 1, scaling exponents of harmonic measure have been computed by B. Duplantier [Phys. Rev. Lett. {\bf 84}, 1363 (2000)] by relating the problem to boundary two-dimensional gravity. We present a simple argument that allows us to connect harmonic measure of critical curves to operators obtained by fusion of primary fields, and compute characteristics of fractal geometry by means of regular methods of conformal field theory. The method is not limited to theories with c⩽1c\leqslant 1.Comment: Some more correction

    Critical curves in conformally invariant statistical systems

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    We consider critical curves -- conformally invariant curves that appear at critical points of two-dimensional statistical mechanical systems. We show how to describe these curves in terms of the Coulomb gas formalism of conformal field theory (CFT). We also provide links between this description and the stochastic (Schramm-) Loewner evolution (SLE). The connection appears in the long-time limit of stochastic evolution of various SLE observables related to CFT primary fields. We show how the multifractal spectrum of harmonic measure and other fractal characteristics of critical curves can be obtained.Comment: Published versio

    Generic critical points of normal matrix ensembles

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    The evolution of the degenerate complex curve associated with the ensemble at a generic critical point is related to the finite time singularities of Laplacian Growth. It is shown that the scaling behavior at a critical point of singular geometry x3∼y2x^3 \sim y^2 is described by the first Painlev\'e transcendent. The regularization of the curve resulting from discretization is discussed.Comment: Based on a talk given at the conference on Random Matrices, Random Processes and Integrable Systems, CRM Montreal, June 200

    Distribution of Class 1 Integrons with IS26-Mediated Deletions in Their 3′-Conserved Segments in Escherichia coli of Human and Animal Origin

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    Class 1 integrons play a role in the emergence of multi-resistant bacteria by facilitating the recruitment of gene cassettes encoding antibiotic resistance genes. 512 E. coli strains sourced from humans (n = 202), animals (n = 304) and the environment (n = 6) were screened for the presence of the intI1 gene. In 31/79 integron positive E. coli strains, the gene cassette regions could not be PCR amplified using standard primers. DNA sequence analysis of 6 serologically diverse strains revealed atypical integrons harboured the dfrA5 cassette gene and only 24 bp of the integron 3′-conserved segment (CS) remained, due to the insertion of IS26. PCR targeting intI1 and IS26 followed by restriction fragment length polymorphism (RFLP) analysis identified the integron-dfrA5-IS26 element in 27 E. coli strains of bovine origin and 4 strains of human origin. Southern hybridization and transformation studies revealed the integron-dfrA5-IS26 gene arrangement was either chromosomally located or plasmid borne. Plasmid location in 4/9 E. coli strains and PCR linkage of Tn21 transposition genes with the intI1 gene in 20/31 strains, suggests this element is readily disseminated by horizontal transfer

    Adaptation of Autocatalytic Fluctuations to Diffusive Noise

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    Evolution of a system of diffusing and proliferating mortal reactants is analyzed in the presence of randomly moving catalysts. While the continuum description of the problem predicts reactant extinction as the average growth rate becomes negative, growth rate fluctuations induced by the discrete nature of the agents are shown to allow for an active phase, where reactants proliferate as their spatial configuration adapts to the fluctuations of the catalysts density. The model is explored by employing field theoretical techniques, numerical simulations and strong coupling analysis. For d<=2, the system is shown to exhibits an active phase at any growth rate, while for d>2 a kinetic phase transition is predicted. The applicability of this model as a prototype for a host of phenomena which exhibit self organization is discussed.Comment: 6 pages 6 figur

    Molecular Characterization of a 21.4 Kilobase Antibiotic Resistance Plasmid from an α-Hemolytic Escherichia coli O108:H- Human Clinical Isolate

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    This study characterizes the 21.4 kilobase plasmid pECTm80 isolated from Escherichia coli strain 80, an α hemolytic human clinical diarrhoeal isolate (serotype O108:H-). DNA sequence analysis of pECTm80 revealed it belonged to incompatibility group X1, and contained plasmid partition and toxin-antitoxin systems, an R6K-like triple origin (ori) replication system, genes required for replication regulation, insertion sequences IS1R, ISEc37 and a truncated transposase gene (Tn3-like ΔtnpA) of the Tn3 family, and carried a class 2 integron. The class 2 integron of pECTm80 contains an intact cassette array dfrA1-sat2, encoding resistance to trimethoprim and streptothricin, and an aadA1 gene cassette truncated by the insertion of IS1R. The complex plasmid replication system includes α, β and γ origins of replication. Pairwise BLASTn comparison of pECTm80 with plasmid pE001 reveals a conserved plasmid backbone suggestive of a common ancestral lineage. Plasmid pECTm80 is of potential clinical importance, as it carries multiple genes to ensure its stable maintenance through successive bacterial cell divisions and multiple antibiotic resistance genes
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