3 research outputs found
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Special Education and Massachusetts Charter Schools: Examining Issues and Practices Associated with Enrollment, Placement and Compliance
Over the last ten years, charter schools have become a viable choice for parent’s seeking an alternative to traditional public schools. However, research shows this is not the case for students with disabilities. The purpose of this dissertation was to examine special education in charter schools and traditional public schools located in Massachusetts. I examined three key areas specific to special education; enrollment, placement and compliance, to determine if differences exist between charter schools and traditional public schools located in the state. Logistic regression analyses were conducted to examine differences between Massachusetts charter schools and public schools with respect to enrollment for each disability category. Multiple regression was used to examine differences in placement for special education students. Lastly, descriptive statistics were used to analyze the Massachusetts Department of Elementary and Secondary Education Coordinated Program Review (CPR) reports to determine if charter schools are compliant with state and federal regulations under the Individuals with Disabilities Education Act (IDEA). Findings revealed charter schools disproportionately under-enrolled students with disabilities, specifically severe disabilities, and placed students in full inclusion placements more frequently than public schools throughout the state. Charter schools also struggled to comply with state and federal regulations under the IDEA, specifically in areas related to direct services and supports to students with disabilities
Regulation of autism-relevant behaviors by cerebellar-prefrontal cortical circuits
Cerebellar dysfunction has been demonstrated in autism spectrum disorders (ASDs); however, the circuits underlying cerebellar contributions to ASD-relevant behaviors remain unknown. In this study, we demonstrated functional connectivity between the cerebellum and the medial prefrontal cortex (mPFC) in mice; showed that the mPFC mediates cerebellum-regulated social and repetitive/inflexible behaviors; and showed disruptions in connectivity between these regions in multiple mouse models of ASD-linked genes and in individuals with ASD. We delineated a circuit from cerebellar cortical areas Right crus 1 (Rcrus1) and posterior vermis through the cerebellar nuclei and ventromedial thalamus and culminating in the mPFC. Modulation of this circuit induced social deficits and repetitive behaviors, whereas activation of Purkinje cells (PCs) in Rcrus1 and posterior vermis improved social preference impairments and repetitive/inflexible behaviors, respectively, in male PC-Tsc1 mutant mice. These data raise the possibility that these circuits might provide neuromodulatory targets for the treatment of ASD