39 research outputs found

    Stage length, spline function and lactate minimum swimming speed

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    Aim. Lactate minimum test (LMT) has become popular in running evaluation. This study analysed the influence of different stage lengths and determination methods on lactate minimum swimming speed (LMS) and its validity for maximal lactate steady-state speed (MLSS-S) assessment. Methods. Twelve male swimmers (19.7+/-1.6 years, 70.3+/-8.7 kg, 181.4+/-7.9 cm) randomly underwent 4-5 evaluations in a 2-week period. LMS was accessed by simple visualisation (SV) and spline function (SF) methods during 200 and 300 in stages LMT (LMT200 and LMT300, respectively), and MLSS-S was determined during constant speed 2000 m efforts. Results. Respectively, SV and SF provided LMS during LMT200 (1.31+/-0.12 m.s(-1) and 1.32+/-0.10 m.sec(-1)) and LMT300 (1.28+/-0.11 m.sec(-1) and 1.28+/-0.10 m.sec(-1)) which were not significantly different (p>0.05) from each other. However, LMS accessed during LMT200 were significantly greater (p0.05) from those found during LMT200 (5.4+/-2.2 and 5.5+/-2.2 mM vs 6.8+/-2.5 and 7.0+/-2.6 mM, respectively for SV and SF). Conclusions. Our results suggest that LMS is not affected by different stage lengths and determination methods. However, LMT300 results seems to provide a more accurate MLSS-S prediction, being adequate for swimmers evaluation.43331231

    State length, spline function and lactate minimus swimming speed

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    Le test de lactate minimum est devenu une méthode populaire d'évaluation en course à pied. Cependant la vitesse à lactate minimum peut être affectée par différentes manipulations de protocole. Dans le cadre d'une étude sur 12 nageurs brésiliens de niveau national, les auteurs examinent l'influence de sessions de longueur différente (200 ou 300m) et de méthodes de détermination (mathématique ou non) sur la vitesse à lactate minimum ainsi que la validité du test de lactate minimum sur l'évaluation de l'état stable maximal de lactatémie en natation. Il en résulte que la vitesse à lactate minimum n'est pas affectée par la longueur des sessions ni la méthode de détermination mais que le test de 300m semble fournir une prévision plus précise de l'état stable maximal

    Effect of an acute β-adrenergic blockade on exercise intensity corresponding to the lactate minimum

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    β-Adrenoreceptor blockade is reported to impair endurance, power output and work capacity in healthy subjects and patients with hypertension. The purpose of this study was to investigate the effect in eighth athletic males of an acute β-adrenergic blockade with propranolol on their individual power output corresponding to a defined lactate minimum (LM). Eight fit males (cyclist or triathlete) performed a protocol to determine the power output corresponding to their individual LM (defined from an incremental exercise test after a rapidly induced exercise lactic acidosis). This protocol was performed twice in a double-blind randomized order by each athlete first ingesting propranolol (80mg) and in a second trial a placebo, 120 minutes respectively prior to the test sequence. The blood lactate concentration obtained 7 minutes after anaerobic exercise (a Wingate test) was significantly lower after acute β-adrenergic blockade (8.6 ± 1.6mM) than under the placebo condition (11.7 ± 1.6mM). The work rate at the LM was lowered from 215.0 ± 18.6 to 184.0 ± 18.6 watts and heart rate at the LM was reduced from 165 ± 1.5 to 132 ± 2.2 beats/minute as a result of the blockade. There was a non-significant correlation (r = 0.29) between the power output at the LM with and without acute β-adrenergic blockade. In conclusion, since the intensity corresponding to the LM is related to aerobic performance, the results of the present study, are able to explain in part, the reduction in aerobic power output produced during β-adrenergic blockade

    The significance of the residual mediastinal mass in treated Hodgkin's disease.

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    The Effects Of Physical Fitness And Body Composition On Oxygen Consumption And Heart Rate Recovery After High-intensity Exercise

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    The aim of this study was to investigate the potential relationship between excess post-exercise oxygen consumption (EPOC), heart rate recovery (HRR) and their respective time constants (tvo 2 and t HR) and body composition and aerobic fitness (VO 2max) variables after an anaerobic effort. 14 professional cyclists (age=28.4±4.8 years, height=176.0±6.7 cm, body mass=74.4±8.1 kg, VO 2max=66. 8±7.6 mL·kg 1·min 1) were recruited. Each athlete made 3 visits to the laboratory with 24 h between each visit. During the first visit, a total and segmental body composition assessment was carried out. During the second, the athletes undertook an incremental test to determine VO 2max. In the final visit, EPOC (15-min) and HRR were measured after an all-out 30 s Wingate test. The results showed that EPOC is positively associated with % body fat (r=0.64), total body fat (r=0.73), fat-free mass (r=0.61) and lower limb fat-free mass (r=0.55) and negatively associated with HRR (r= 0.53, p<0.05 for all). HRR had a significant negative correlation with total body fat and % body fat (r= 0.62, r= 0.56 respectively, p<0.05 for all). These findings indicate that VO 2max does not influence HRR or EPOC after high-intensity exercise. Even in short-term exercise, the major metabolic disturbance due to higher muscle mass and total muscle mass may increase EPOC. 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