Ischemic neurons recruit natural killer cells that accelerate brain infarction

Brain ischemia and reperfusion activate the immune system. The abrupt development of brain ischemic lesions suggests that innate immune cells may shape the outcome of stroke. Natural killer (NK) cells are innate lymphocytes that can be swiftly mobilized during the earliest phases of immune responses, but their role during stroke remains unknown. Herein, we found that NK cells infiltrated the ischemic lesions of the human brain. In a mouse model of cerebral ischemia, ischemic neuron-derived fractalkine recruited NK cells, which subsequently determined the size of brain lesions in a T and B cell-independent manner. NK cell-mediated exacerbation of brain infarction occurred rapidly after ischemia via the disruption of NK cell tolerance, augmenting local inflammation and neuronal hyperactivity. Therefore, NK cells catalyzed neuronal death in the ischemic brain

<I>Calculation of Pressure and Density in Solar Power Plant Chimneys</I>

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Performance of the power conversion unit of a solar chimney power plant

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The solar chimney air standard thermodynamic cycle.

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Effect of cross-flow on the performance of air-cooled heat exchangers fans

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Turbogenerator layouts for solar chimney power plants.

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Solar Chimney cycle analysis with system loss and solar collector performance.

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Velocity measurment in a hydrodynamic torque converter.

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High Temperature Thermal Energy Storage Utilizing Metallic Phase Change Materials and Metallic Heat Transfer Fluids

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Performance of the power conversion unit of a solar chimney power plant

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