1,386 research outputs found

    What does it mean for half of an empty cavity to be full?

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    21 págs.; 8 figs.; 2 app.; PACS numbers: 03.67.Bg, 03.70.+k, 11.10.-z© 2015 American Physical Society. It is well known that the vacuum state of a quantum field is spatially entangled. This is true in both free and confined spaces, for example, in an optical cavity. The obvious consequence of this, however, is surprising and intuitively challenging: namely, that in a mathematical sense, half of an empty cavity is not empty. Formally this is clear, but what does this physically mean in terms of, say, measurements that can actually be made? In this paper we utilize a local quantization procedure along with the tools of Gaussian quantum mechanics to characterize the particle content in the reduced state of a subregion within a cavity and expose the spatial profile of its entanglement with the opposite region. We then go on to discuss a thought experiment in which a mirror is very quickly introduced between the regions. In so doing we expose a simple and physically concrete answer to the above question: the real excitations created by slamming down the mirror are mathematically equivalent to those previously attributed to the reduced states of the subregions. Performing such an experiment in the laboratory may be an excellent method of verifying vacuum entanglement, and we conclude by discussing different possibilities of achieving this aim.This work is supported by Spanish MICINN Projects FIS2011-29287 and CAM research consortium QUITEMAD+ S2013/ICE-2801. E. B. acknowledges support by the Michael Smith Foreign Study Supplements Program, M. del R. was supported by a CSIC JAEPREDOC grant and H. Westman was supported by the JAE-DOC 2011 CSIC & ESF program. A. D. was supported by the National Science Center, Sonata BIS Grant No. 2012/07/E/ST2/01402.Peer Reviewe

    Mass inflation in f(R) gravity: A conjecture on the resolution of the mass inflation singularity

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    We study gravitational collapse of a charged black hole in f(R) gravity using double-null formalism. We require cosmological stability to f(R) models; we used the Starobinsky model and the R + (1/2)cR^2 model. Charged black holes in f(R) gravity can have a new type of singularity due to higher curvature corrections, the so-called f(R)-induced singularity, although it is highly model-dependent. As the advanced time increases, the internal structure will approach the Cauchy horizon, which may not be an inner apparent horizon. There is mass inflation as one approaches the Cauchy horizon and hence the Cauchy horizon may be a curvature singularity with nonzero area. However, the Ricci scalar is finite for an out-going null observer. This can be integrated as follows: Cosmologically stable higher curvature corrections of the Ricci scalar made it bounded even in the presence of mass inflation. Finally, we conjecture that if there is a general action including general higher curvature corrections with cosmological stability, then the corrections can make all curvature components finite even in the presence of mass inflation. This might help us to resolve the problem of inner horizon instability of regular black hole models.Comment: 31 pages, 15 figure

    Parity Mixed Doublets in A = 36 Nuclei

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    The γ\gamma-circular polarizations (PγP_{\gamma}) and asymmetries (AγA_{\gamma}) of the parity forbidden M1 + E2 γ\gamma-decays: 36Cl(Jπ=2;T=1;Ex=1.95^{36}Cl^{\ast} (J^{\pi} = 2^{-}; T = 1; E_{x} = 1.95 MeV) \rightarrow 36Cl(Jπ=2+;T=1;g.s.)^{36}Cl (J^{\pi} = 2^{+}; T = 1; g.s.) and 36Ar(Jπ=2;T=0;Ex=4.97^{36}Ar^{\ast} (J^{\pi} = 2^{-}; T = 0; E_{x} = 4.97 MeV) \rightarrow 36Ar(Jπ=2+;T=0;Ex=1.97^{36}Ar^{\ast} (J^{\pi} = 2^{+}; T = 0; E_{x} = 1.97 MeV) are investigated theoretically. We use the recently proposed Warburton-Becker-Brown shell-model interaction. For the weak forces we discuss comparatively different weak interaction models based on different assumptions for evaluating the weak meson-hadron coupling constants. The results determine a range of PγP_{\gamma} values from which we find the most probable values: PγP_{\gamma} = 1.11041.1 \cdot 10^{-4} for 36Cl^{36}Cl and PγP_{\gamma} = 3.51043.5 \cdot 10^{-4} for 36Ar^{36}Ar.Comment: RevTeX, 17 pages; to appear in Phys. Rev.

    Hyperparathyroidism, platelet intracellular free calcium and hypertension in chronic renal failure

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    Hyperparathyroidism, platelet intracellular free calcium and hypertension in chronic renal failure. To investigate possible relationships between hyperparathyroidism, alterations in intracellular free calcium concentration ([Ca2+]i and hypertension in chronic renal failure, serum concentrations of intact parathyroid hormone (PTH) were measured by two-site immunometric assay, and platelet ([Ca2+]i) was assessed using the fluorescent indicator fura-2. Thirty-six patients with chronic renal failure were studied, 10 with normal serum PTH concentrations (mean 8.0 ± 0.6 pmol/liter), 17 with elevated serum PTH (35.0 ± 7.2 pmol/liter) and 9 patients with elevated PTH (36.2 ±5.9 pmol/liter) who were receiving nifedipine. Platelet [Ca2+]i was increased in patients with elevated PTH, compared with those in whom PTH was normal (138 ± 16 vs. 83 ± 7 nmol/liter, P < 0.01). A linear relation was observed between serum PTH and platelet [Ca2+]i in these patients (r = 0.818, P < 0.001). In contrast, platelet [Ca2+]i was not elevated (84 ± 9 nmol/liter) in the patients with elevated PTH who were receiving nifedipine. A linear relation was also present between both serum PTH (r = 0.616, P < 0.001) and platelet [Ca2+]i (r = 0.576, P < 0.005) and mean blood pressure. Nine patients with hyperparathyroidism were restudied after treatment with the vitamin D analogue alfacalcidol. This resulted in significant decreases in serum PTH (P < 0.01), platelet [Ca2+]i (P < 0.02), and mean blood pressure (P < 0.05). These studies indicate that [Ca2+]i may be increased early in renal failure, and that this increase occurs in association with both hyperparathyroidism and hypertension. Furthermore, treatment of hyperparathyroidism with alfacalcidol may result in reductions in both [Ca2+]i and blood pressure. The lack of elevation in [Ca2+]i in nifedipine-treated patients with hyperparathyroidism suggests that, in uremia, increases in cytosolic calcium induced by PTH or other factors may be mediated in part by dihydropyridine-sensitive mechanisms

    New calculations of the PNC Matrix Element for the JπTJ^{\pi}T 0+1,01^{+}1,0^{-}1 doublet in 14^{14}N

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    A new calculation of the predominantly isoscalar PNC matrix element between the JπTJ^{\pi}T 0+1,010^{+}1,0^{-}1 (Ex_{x} \approx 8.7 MeV) states in 14^{14}N has been carried out in a (0+1+2+3+4)ω\hbar \omega model space with the Warburton-Brown interaction. The magnitude of the PNC matrix element of 0.22 to 0.34 eV obtained with the DDH PNC interaction is substantially suppressed compared with previous calculations in smaller model spaces but shows agreement with the preliminary Seattle experimental data. The calculated sign is opposite to that obtained experimentally, and the implications of this are discussed.Comment: REVTEX, 28 page

    New solutions in 3D gravity

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    We study gravitational theory in 1+2 spacetime dimensions which is determined by the Lagrangian constructed as a sum of the Einstein-Hilbert term plus the two (translational and rotational) gravitational Chern-Simons terms. When the corresponding coupling constants vanish, we are left with the purely Einstein theory of gravity. We obtain new exact solutions for the gravitational field equations with the nontrivial material sources. Special attention is paid to plane-fronted gravitational waves (in case of the Maxwell field source) and to the circularly symmetric as well as the anisotropic cosmological solutions which arise for the ideal fluid matter source.Comment: Revtex, 21 pages, no figure

    Non-Linear Vibrations in Nuclei

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    We have perfomed Time Dependant Hartree-Fock (TDHF) calculations on the non linear response of nuclei. We have shown that quadrupole (and dipole) motion produces monopole (and quadrupole) oscillations in all atomic nuclei. We have shown that these findings can be interpreted as a large coupling between one and two phonon states leading to large anharmonicities.Comment: 4 pages, 3 figure

    Quantum-critical pairing with varying exponents

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    We analyse the onset temperature T_p for the pairing in cuprate superconductors at small doping, when tendency towards antiferromagnetism is strong. We consider the model of Moon and Sachdev (MS), which assumes that electron and hole pockets survive in a paramagnetic phase. Within this model, the pairing between fermions is mediated by a gauge boson, whose propagator remains massless in a paramagnet. We relate the MS model to a generic \gamma-model of quantum-critical pairing with the pairing kernel \lambda (\Omega) \propto 1/\Omega^{\gamma}. We show that, over some range of parameters, the MS model is equivalent to the \gamma-model with \gamma =1/3 (\lambda (\Omega) \propto \Omega^{-1/3}). We find, however, that the parameter range where this analogy works is bounded on both ends. At larger deviations from a magnetic phase, the MS model becomes equivalent to the \gamma-model with varying \gamma >1/3, whose value depends on the distance to a magnetic transition and approaches \gamma =1 deep in a paramagnetic phase. Very near the transition, the MS model becomes equivalent to the \gamma-model with varying \gamma <1/3. Right at the magnetic QCP, the MS model is equivalent to the \gamma-model with \gamma =0+ (\lambda (\Omega) \propto \log \Omega), which is the model for color superconductivity. Using this analogy, we verified the formula for T_c derived for color superconductivity.Comment: 10 pages, 8 figures, submitted to JLTP for a focused issue on Quantum Phase Transition

    Integrin activation - the importance of a positive feedback

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    Integrins mediate cell adhesion and are essential receptors for the development and functioning of multicellular organisms. Integrin activation is known to require both ligand and talin binding and to correlate with cluster formation but the activation mechanism and precise roles of these processes are not yet resolved. Here mathematical modeling, with known experimental parameters, is used to show that the binding of a stabilizing factor, such as talin, is alone insufficient to enable ligand-dependent integrin activation for all observed conditions; an additional positive feedback is required.Comment: in press in Bulletin of Mathematical Biolog
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