10 research outputs found

    RAGE does not contribute to renal injury and damage upon ischemia/reperfusion-induced injury.

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    Item does not contain fulltextThe receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury

    Compounds of Order I

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    Eicosanoids and Smooth Muscle Function

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    Interactions among Cytoplasm, Endomembranes, and the Cell Surface

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    Method parameters’ impact on mortality and variability in mouse stroke experiments: a meta-analysis

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