Soft supersymmetry breaking in the nonlinear sigma model

In this work we discuss the dynamical generation of mass in a deformed ${\cal N}=1$ supersymmetric nonlinear sigma model in a two-dimensional ($D=1+1$) space-time. We introduce the deformation by imposing a constraint that softly breaks supersymmetry. Through the tadpole method, we compute the effective potential at leading order in $1/N$ expansion showing that the model exhibit a dynamical generation of mass to the matter fields. Supersymmetry is recovered in the limit of the deformation parameter going to zero.Comment: 9 pages, 2 figures. Revised version. arXiv admin note: text overlap with arXiv:1308.471

Qualidade fisiológica de sementes de arroz armazenadas em diferentes embalagens e temperaturas.

bitstream/item/78791/1/Boletim-163.pd

Agricultores guardiões de sementes e o desenvolvimento in situ de cultivares crioulas.

bitstream/CPACT-2009-09/11716/1/artigo-Bevilaq_sement.pd

Sistema de produção de forragem e de sementes de trevo vesiculoso para a agricultura familiar.

bitstream/item/67324/1/boletim-139-corrigido.pd

Desempenho e qualidade da forragem de trevo-vesiculoso em função da dose de fosfato natural.

bitstream/item/78798/1/Boletim-165-.pd

Kidney-Derived c-Kit(+) Cells Possess Regenerative Potential

Kidney-derived c-Kit(+) cells exhibit progenitor/stem cell properties in vitro (self-renewal capacity, clonogenicity, and multipotentiality). These cells can regenerate epithelial tubular cells following ischemia-reperfusion injury and accelerate foot processes effacement reversal in a model of acute proteinuria in rats. Several mechanisms are involved in kidney regeneration by kidney-derived c-Kit(+ )cells, including cell engraftment and differentiation into renal-like structures, such as tubules, vessels, and podocytes. Moreover, paracrine mechanisms could also account for kidney regeneration, either by stimulating proliferation of surviving cells or modulating autophagy and podocyte cytoskeleton rearrangement through mTOR-Raptor and -Rictor signaling, which ultimately lead to morphological and functional improvement. To gain insights into the functional properties of c-Kit(+) cells during kidney development, homeostasis, and disease, studies on lineage tracing using transgenic mice will unveil their fate. The results obtained from these studies will set the basis for establishing further investigation on the therapeutic potential of c-Kit(+) cells for treatment of kidney disease in preclinical and clinical studies.Conselho Nacional em Pesquisa e Desenvolvimento (CNPq)Fundacao de Amparo a Pesquisa do Estado de Sao Paulo (FAPESP)European Foundation for the Study of Diabetes (EFSD)Univ Sao Paulo, Renal Div, Lab Cellular Genet & Mol Nephrol, Sao Paulo, SP, BrazilUniv Miami, Leonard M Miller Sch Med, Interdisciplinary Stem Cell Inst, Miami, FL USAUniv Miami, Leonard M Miller Sch Med, Dept Mol & Cellular Pharmacol, Miami, FL USAUniv Miami, Div Cardiol, Leonard M Miller Sch Med, Miami, FL USAHosp Israelita Albert Einstein, Inst Israelita Ensino & Pesquisa Albert Einstein, Albert Einstein Ave,627-701 Bldg A, BR-05652900 Sao Paulo, SP, BrazilUniv Fed Sao Paulo, Div Nephrol, Sao Paulo, SP, BrazilUniv Fed Sao Paulo, Div Nephrol, Sao Paulo, SP, BrazilCNPq: 456959/2013-0FAPESP: 13/19560-6Web of Scienc