21 research outputs found
Complete genome sequence of the Clostridium difficile laboratory strain 630Δerm reveals differences from strain 630, including translocation of the mobile element CTn5
- Author
- Publication venue
- Springer Nature
- Publication date
- 01/01/2015
- Field of study
Molecular Technology and Informatics for Personalised Medicine and Healt
Search for the standard model Higgs boson at LEP
- Author
- Abbaneo D
- Abbaneo D
- Abbiendi G
- Abbiendi G
- Abdallah J
- Abreu P
- Achard P
- Adam W
- Adriani O
- Adzic P
- Affholderbach K
- Aguilar-Benitez M
- Ainsley C
- Akesson PF
- Albrecht T
- Alcaraz J
- Alderweireld T
- Alemanni G
- Alemany-Fernandez R
- Alexander G
- Allaby J
- Allison J
- Allmendinger T
- Allport PP
- Aloisio A
- Alviggi MG
- Amaldi U
- Amapane N
- Amaral P
- Amato S
- Anagnostou G
- Anashkin E
- Anderhub H
- Anderson KJ
- Andreazza A
- Andreev VP
- Andringa S
- Anjos N
- Anselmo F
- Antilogus P
- Antonelli A
- Antonelli M
- Apel WD
- Arcelli S
- Arefiev A
- Armstrong SR
- Arnoud Y
- Asai S
- Ask S
- Asman B
- Augustin JE
- Augustinus A
- Awunor O
- Axen D
- Azemoon T
- Aziz T
- Azuelos G
- Azzurri P
- Baarmand M
- Badaud F
- Bagliesi G
- Bagnaia P
- Bailey I
- Baillon P
- Bajo A
- Baksay G
- Baksay L
- Baldew SV
- Ballestrero A
- Bambade P
- Banerjee S
- Banerjee S
- Barate R
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- Barberio E
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- Bardin D
- Barillere R
- Barker G
- Barklow T
- Barlow RJ
- Baroncelli A
- Bartalini P
- Basile M
- Batalova N
- Batley RJ
- Battaglia M
- Battiston R
- Baubillier M
- Bay A
- Becattini F
- Bechtle P
- Becker U
- Becks KH
- Begalli M
- Behner F
- Behnke T
- Behrmann A
- Bell KW
- Bell PJ
- Bella G
- Bellerive A
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- Ben-Haim E
- Bencivenni G
- Benekos N
- Benelli G
- Benvenuti A
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- Beuselinck R
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- Vogel H
- Vogt H
- Vollmer CF
- von Krogh J
- von Wimmersperg-Toeller JH
- Vorobiev I
- Vorobyov AA
- Voss H
- Vossebeld J
- Vrba V
- Waananen A
- Wachsmuth H
- Wadhwa M
- Wahlen H
- Waller D
- Wallraff W
- Wang M
- Wang T
- Wang XL
- Wang ZM
- Ward CP
- Ward DR
- Ward JJ
- Washbrook AJ
- Wasserbacch S
- Watkins PM
- Watson AT
- Watson NK
- Weber M
- Weiser C
- Well PS
- Wengler T
- Wermes N
- Werner S
- Wetterling D
- White R
- Wicke D
- Wickens J
- Wiedenmann W
- Wienemann P
- Wilkens H
- Wilkinson G
- Wilson GW
- Wilson JA
- Winter M
- Witek M
- Wolf G
- Wolf G
- Wu J
- Wu SL
- Wu SX
- Wu X
- Wunsch M
- Wyatt TR
- Wynhoff S
- Xia L
- Xie Y
- Xu R
- Xu ZZ
- Xue S
- Yamamoto J
- Yamashita S
- Yang BZ
- Yang CG
- Yang HJ
- Yang M
- Yeh SC
- Yuan C
- Yushchenko O
- Zachariadou K
- Zalewska A
- Zalewski P
- Zalite A
- Zalite Y
- Zavrtanik D
- Zeitnitz C
- Zer-Zion D
- Zhang J
- Zhang L
- Zhang ZP
- Zhao J
- Zhao W
- Zhu GY
- Zhu RY
- Zhuang HL
- Zhuravlov V
- Zichichi A
- Ziegler T
- Zilizi G
- Zimin NI
- Zimmermann B
- Zintchenko A
- Zito G
- Zivkovic L
- Zobernig G
- Zoller M
- Zupan M
- Publication venue
- ELSEVIER SCIENCE BV
- Publication date
- 17/07/2003
- Field of study
False negatives in GBA1 sequencing due to polymerase dependent allelic imbalance
- Author
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 08/01/2021
- Field of study
A variant in the GBA1 gene is one of the most common genetic risk factors to develop Parkinson’s disease (PD). Here the serendipitous finding is reported of a polymerase dependent allelic imbalance when using next generation sequencing, potentially resulting in false-negative results when the allele frequency falls below the variant calling threshold (by default commonly at 30%). The full GBA1 gene was sequenced using next generation sequencing on saliva derived DNA from PD patients. Four polymerase chain reaction conditions were varied in twelve samples, to investigate the effect on allelic imbalance: (1) the primers (n = 4); (2) the polymerase enzymes (n = 2); (3) the primer annealing temperature (Ta) specified for the used polymerase; and (4) the amount of DNA input. Initially, 1295 samples were sequenced using Q5 High-Fidelity DNA Polymerase. 112 samples (8.6%) had an exonic variant and an additional 104 samples (8.0%) had an exonic variant that did not pass the variant frequency calling threshold of 30%. After changing the polymerase to TaKaRa LA Taq DNA Polymerase Hot-Start Version: RR042B, all samples had an allele frequency passing the calling threshold. Allele frequency was unaffected by a change in primer, annealing temperature or amount of DNA input. Sequencing of the GBA1 gene using next generation sequencing might be susceptible to a polymerase specific allelic imbalance, which can result in a large amount of flase-negative results. This was resolved in our case by changing the polymerase. Regions displaying low variant calling frequencie
The complete genome sequence of the murine pathobiont Helicobacter typhlonius
- Author
- Publication venue
- 'Frontiers Media SA'
- Publication date
- 01/01/2016
- Field of study
BackgroundImmuno-compromised mice infected with Helicobacter typhlonius are used to model microbially inducted inflammatory bowel disease (IBD). The specific mechanism through which H. typhlonius induces and promotes IBD is not fully understood. Access to the genome sequence is essential to examine emergent properties of this organism, such as its pathogenicity. To this end, we present the complete genome sequence of H. typhlonius MIT 97-6810, obtained through single-molecule real-time sequencing.ResultsThe genome was assembled into a single circularized contig measuring 1.92 Mbp with an average GC content of 38.8%. In total 2,117 protein-encoding genes and 43 RNA genes were identified. Numerous pathogenic features were found, including a putative pathogenicity island containing components of type IV secretion system, virulence-associated proteins and cag pathogenicity island protein. We compared the genome of H. typhlonius to those of the murine pathobiont Helicobacter hepaticus and human pathobiont Helicobacter pylori. H. typhlonius resembles H. hepaticus most with 1,594 (75.3%) of its genes being orthologous to genes in H. hepaticus. Determination of the global methylation state revealed eight distinct recognition motifs for adenine and cytosine methylation. H. typhlonius shares four of its recognition motifs with H. pylori. ConclusionsThe complete genome sequence of H. typhlonius MIT 97-6810 enabled us to identify many pathogenic features suggesting that H. typhlonius can act as a pathogen. Follow-up studies are necessary to evaluate the true nature of its pathogenic capabilities. We found many methylated sites and a plethora of restriction-modification systems. The genome, together with the methylome, will provide an essential resource for future studies investigating gene regulation, host interaction and pathogenicity of H. typhlonius. In turn, this work can contribute to unraveling the role of Helicobacter in enteric disease
A Large-Scale Full GBA1 Gene Screening in Parkinson's Disease in the Netherlands.
- Author
- Baas Frank
- Berendse Henk W.
- Boertien Jeffrey M.
- Bonifati Vincenzo
- Boon Agnita J. W.
- Contarino M. Fiorella
- Cullen Valerie C.
- de Bie Rob M. A.
- den Heijer Jonas M.
- Groeneveld Geert J.
- Hilt Dana C.
- Hoff Jorrit I.
- Lansbury Peter
- Majoor-Krakauer Danielle
- Munts Alex G.
- Ponsen Mirthe M.
- Quadri Marialuisa
- Schmitz Arnoud
- van de Berg Wilma D. J.
- van der Plas Anne A.
- van Hilten Jacobus J.
- van Laar Teus
- van Mierlo Tom
- Publication venue
- 'Wiley'
- Publication date
- 01/09/2020
- Field of study
Background: The most common genetic risk factor for Parkinson's disease known is a damaging variant in the GBA1 gene. The entire GBA1 gene has rarely been studied in a large cohort from a single population. The objective of this study was to assess the entire GBA1 gene in Parkinson's disease from a single large population. Methods: The GBA1 gene was assessed in 3402 Dutch Parkinson's disease patients using next-generation sequencing. Frequencies were compared with Dutch controls (n = 655). Family history of Parkinson's disease was compared in carriers and noncarriers. Results: Fifteen percent of patients had a GBA1 nonsynonymous variant (including missense, frameshift, and recombinant alleles), compared with 6.4% of controls (OR, 2.6; P < 0.001). Eighteen novel variants were detected. Variants previously associated with Gaucher's disease were identified in 5.0% of patients compared with 1.5% of controls (OR, 3.4; P < 0.001). The rarely reported complex allele p.D140H + p.E326K appears to likely be a Dutch founder variant, found in 2.4% of patients and 0.9% of controls (OR, 2.7; P = 0.012). The number of first-degree relatives (excluding children) with Parkinson's disease was higher in p.D140H + p.E326K carriers (5.6%, 21 of 376) compared with p.E326K carriers (2.9%, 29 of 1014); OR, 2.0; P = 0.022, suggestive of a dose effect for different GBA1 variants. Conclusions: Dutch Parkinson's disease patients display one of the largest frequencies of GBA1 variants reported so far, consisting in large part of the mild p.E326K variant and the more severe Dutch p.D140H + p.E326K founder allele
Post-crisis banking regulation: evolution of economic thinking as it happened on Vox
- Author
- Atkinson Paul E.
- Bair Sheila
- Blundell-Wignall Adrian
- Boot Arnoud
- Borio Claudio
- Bremus Franziska
- Buch Claudia M.
- Cabellero Ricardo
- Calomiris Charles W.
- Claessens Stijn
- Danielsson Jon
- Gersbach Hans
- Goodhart Charles
- Hagendorff Jens
- Hoshi Takeo
- Igan Deniz
- Laeven Luc
- Levine Ross
- Mishra Prachi
- Nier Erlend W.
- Perotti Enrico
- Persaud Avinash
- Pozsar Zoltan
- Ratnovski Lev
- Russ Katheryn
- Schmitz Stefan W.
- Schnitzer Monika
- Shin Hyun Song
- Singh Manmohan
- Spagnolo Giancarlo
- Tressel Thierry
- Vallascas Francesco
- Zamil Raihan
- Čihák Martin
- Publication venue
- The Centre for Economic Policy Research
- Publication date
- 02/03/2015
- Field of study
This eBook collects some of the best Vox columns on financial regulations, starting with the fundamentals of financial regulations, moving on to bank capital and the Basel regulations, and finishing with the wider considerations of the regulatory agenda and the political dimension. Collecting columns from over the past six years, this eBook maps the evolution of leading thought on banking regulation
Measurement of the Mass of the Z-Boson and the Energy Calibration of Lep
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- Abbaneo D.
- Abreu P.
- Acton P. D.
- Adam W.
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- Adriani O.
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- Ahlen S.
- Ajaltouni Z.
- Alcaraz J.
- Aleksan R.
- Alekseev G. D.
- Alemany R.
- ALEPH Collaboration
- Alexander G.
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- Allison J.
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- Xie Y.
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- Xu D.
- Xu R.
- Xu Y. D.
- Xu Z. Z.
- Xue Z. L.
- Yaari R.
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- Zaitsev A.
- Zalewska A.
- Zanini L.
- Zavrtanik D.
- Zemp P.
- Zeng M.
- Zeng Y.
- Zeuner W.
- Zevgolatakos E.
- Zhang D. H.
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- Zichichi A.
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- Zito G.
- Zito M.
- Zobernig G.
- Zontar D.
- Zorn G. T.
- Zuberi R.
- Zumerle G.
- Zuniga J.
- Publication venue
- Elsevier science bv
- Publication date
- 01/01/1993
- Field of study
Contains fulltext :
26847___.PDF (publisher's version ) (Open Access
Small Firm Credit Market Discrimination, SBA-Guaranteed Lending, and Local Market Economic Performance
- Author
- A N Berger
- A N Berger
- Allen N Berger
- Allen N Berger
- Allen N Berger
- Arnoud W A Boot
- Ben R Craig
- Ben R Craig
- Ben R Craig
- Ben R. Craig
- Cole
- D G Blanchflower
- E M
- Edward J Kane
- Elisabeth H Rhymes
- F Bergstr�m
- Frank Klieberger
- G S Becker
- James A Schmitz
- James B. Thomson
- Jith Jayartne
- Joan Robinson
- Joseph E Stiglitz
- Joseph Schumpeter
- K S Calvalluzzo
- Lawrence White
- Luigi Guiso
- M Kauffman Foundation
- M Petersen
- Marc Cowling
- Mitchell A Petersen
- Mitchell Petersen
- Mitchell Petersen
- P S Calem
- Raghuram Rajan
- Robert G King
- Ross Levine
- Sander Wennekers
- Sba
- Sherrill Shaffer
- Sherrill Shaffer
- Steven Bond
- Steven J Davis
- W E Jackson
- W E Jackson
- W W Lang
- Walker Todd
- William A Brock
- William E. Jackson
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2006
- Field of study
Does SBA Guaranteed Lending Improve Economic Performance in Low-Income Areas?
- Author
- _______________
- A A Dick
- A N Berger
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- Arnoud W A Boot
- B R Craig
- Ben R Craig
- Ben R Craig
- Ben R. Craig
- Cole
- D G Blanchflower
- E M Kauffman Foundation
- Edward J Kane
- Elisabeth H Rhymes
- F Bergstr�m
- Frank Klieberger
- G S Becker
- James A Schmitz
- James B. Thomson
- Jith Jayartne
- Joan Robinson
- Joseph E Stiglitz
- Joseph Schumpeter
- K S Calvalluzzo
- Lawrence J White
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- M Kauffman Foundation
- M Petersen
- Marc Cowling
- Mitchell A Petersen
- Mitchell Petersen
- Mitchell Petersen
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- Robert G King
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- S Shaffer
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- Sander Wennekers
- Steven Bond
- Steven J Davis
- W E Jackson
- W E Jackson
- Walker F Todd
- William A Brock
- William E. Jackson
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2006
- Field of study
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- Buchhalter Ivo
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- Bullman Susan
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- Burns Kathleen H.
- Busanovich John
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- Butte Atul J.
- Byrne Niall J.
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- Choi Wan
- Chomienne Christine
- Chong Zechen
- Choo Su Pin
- Chou Angela
- Christ Angelika N.
- Christie Elizabeth L.
- Chuah Eric
- Cibulskis Carrie
- Cibulskis Kristian
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- Corbo Vincenzo
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- Zhang Yan
- Zhang Zemin
- Zhao Zhongming
- Zheng Liangtao
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- Zhou Wanding
- Zhou Yong
- Zhu Bin
- Zhu Hongtu
- Zhu Jingchun
- Zhu Shida
- Zou Lihua
- Zou Xueqing
- Publication venue
- Nature Communications
- Publication date
- 28/08/2020
- Field of study
Funder: Canadian Network for Research and Innovation in Machining Technology, Natural Sciences and Engineering Research Council of Canada (NSERC Canadian Network for Research and Innovation in Machining Technology); doi: https://doi.org/10.13039/501100002790Funder: Genome Canada (Génome Canada); doi: https://doi.org/10.13039/100008762Funder: Canada Foundation for Innovation (Fondation canadienne pour l'innovation); doi: https://doi.org/10.13039/501100000196Funder: Terry Fox Research Institute (Institut de Recherche Terry Fox); doi: https://doi.org/10.13039/501100004376Abstract: Sex differences have been observed in multiple facets of cancer epidemiology, treatment and biology, and in most cancers outside the sex organs. Efforts to link these clinical differences to specific molecular features have focused on somatic mutations within the coding regions of the genome. Here we report a pan-cancer analysis of sex differences in whole genomes of 1983 tumours of 28 subtypes as part of the ICGC/TCGA Pan-Cancer Analysis of Whole Genomes (PCAWG) Consortium. We both confirm the results of exome studies, and also uncover previously undescribed sex differences. These include sex-biases in coding and non-coding cancer drivers, mutation prevalence and strikingly, in mutational signatures related to underlying mutational processes. These results underline the pervasiveness of molecular sex differences and strengthen the call for increased consideration of sex in molecular cancer research