More poor kids in more poor places: children increasingly live where poverty persists

More poor kids in more poor places: children increasingly live where poverty persist

The Increasing Diversity of America\u27s Youth

This brief documents how unfolding demographic forces have placed today’s children and youth at the forefront of America’s new racial and ethnic diversity. Authors Kenneth M. Johnson, Andrew Schaefer, Daniel T. Lichter, and Luke T. Rogers discuss how the rapidly changing racial and ethnic composition of the youth population has important implications for intergroup relations, ethnic identities, and electoral politics. They report that diversity is increasing among America’s youth because there are more minority children and fewer non-Hispanic white children. Minority births exceeded non-Hispanic white births for the first time in U.S. history in 2011 according to Census Bureau estimates. Both the declining number of non-Hispanic white women of prime child-bearing and growing numbers of minority women contributed to this change as did differential fertility rates. The largest gains in child diversity between 2000 and 2012 were in suburban and smaller metropolitan areas. Yet, child diversity is geographically uneven, with minimal diversity in some areas of the country and significant diversity in other areas. They conclude that natural population increase—particularly fertility rates—will continue to reshape the racial and ethnic mix of the country, and this change will be reflected first among the nation’s youngest residents

Child Poverty Higher and More Persistent in Rural America

The negative consequences of growing up in a poor family are well known. Poor children are less likely to have timely immunizations, have lower academic achievement, are generally less engaged in school activities, and face higher delinquency rates in adolescent years. Each of these has adverse impacts on their health, earnings, and family status in adulthood. Less understood is how the experience of poverty can differ depending on the community context. Being poor in a relatively well-off community with good infrastructure and schools is different from being poor in a place where poverty rates have been high for generations, where economic investment in schools and infrastructure is negligible, and where pathways to success are few. The hurdles are even higher in rural areas, where low population density, physical isolation, and the broad spatial distribution of the poor make service delivery and exposure to innovative programs more challenging

Charge Neutrality of the Color-Flavor Locked Phase from the Low Energy Effective Theory

We investigate the issue of charge neutrality of the CFL$K^0$ phase of dense quark matter using the low energy effective theory of high density QCD. We show that the local electric and color charge neutrality of the ground state in a homogeneous color superconducting medium follows from its dynamics. We also consider the situation of a spatially inhomogeneous medium, such as may be found in a neutron star core. We find that spatial inhomogeneity results in the generation of electric fields, and positrons/electrons may be present in the ground state. We estimate the concentration of charged leptons in the ground state to be $n_{e}\sim{10^2}{cm}^{-3}$ and consider their influence on the opacity of the medium with respect to the modified photons.Comment: typos corrected, this version to appear in PR

Abelson kinase acts as a robust, multifunctional scaffold in regulating embryonic morphogenesis

Abelson family kinases (Abl) are key regulators of cell behavior and the cytoskeleton during development and in leukemia. Abl's SH3, SH2, and tyrosine kinase domains are joined via a linker to an F-actin-binding domain (FABD). Research on Abl's roles in cell culture led to several hypotheses for its mechanism of action: 1) Abl phosphorylates other proteins, modulating their activity. 2) Abl directly regulates the cytoskeleton via its cytoskeletal interaction domains, and/or 3) Abl is a scaffold for a signaling complex. The importance of these roles during normal development remains untested. We tested these mechanistic hypotheses during Drosophila morphogenesis using a series of mutants to examine Abl's many cell biological roles. Strikingly, Abl lacking the FABD fully rescued morphogenesis, cell shape change, actin regulation, and viability, while kinase dead Abl, though reduced in function, retained substantial rescuing ability in some but not all Abl functions. We also tested the function of four conserved motifs in the linker region, revealing a key role for a conserved PXXP motif known to bind Crk and Abi. We propose Abl acts as a robust multi-domain scaffold with different protein motifs and activities contributing differentially to diverse cellular behaviors