The Impact of Hunting Package Attributes on Hunting Package Prices in Mississippi

Potential economic impacts of hunting activities suggested opportunities for non-industrial private landowners in Mississippi to capitalize on apparent market demand for fee-access hunting. Data were collected from outfitting individuals/firms operating within Mississippi to analyze the impact of hunting package attributes on package prices. Generally, package prices were directly related to the length of the package in days, with the increase in price decreasing with each additional day. Provision of other amenities such as lodging as well as joint activities such as fishing also increased package prices. Finally, there were differences in package prices depending on species being hunted. These results provide landowners with added information about the potential values of hunting package prices, which, when combined with costs of providing the packages, can assist in making optimal enterprise management decisions.Resource /Energy Economics and Policy,

The Value of Hunting Package Attributes

Economic impacts of hunting activities reveals opportunities for landowners to capitalize on apparent market demand for fee-access hunting. This paper discusses the marginal values of hunting package attributes. The results will provide landowners the information needed to make optimal management decision.Resource /Energy Economics and Policy,

How many stars form in galaxy mergers?

We forward model the difference in stellar age between post-coalescence mergers and a control sample with the same stellar mass, environmental density, and redshift. In particular, we use a pure sample of 445 post-coalescence mergers from the recent visually-confirmed post-coalescence merger sample identified by Bickley et al. and find that post-coalescence mergers are on average younger than control galaxies for $10<\log (M_\star/\mathrm{M}_\odot)<11$. The difference in age from matched controls is up to 1.5 Gyr, highest for lower stellar mass galaxies. We forward model this difference using parametric star formation histories, accounting for the pre-coalescence inspiral phase of enhanced star formation using close pair data, and a final additive burst of star formation at coalescence. We find a best-fitting stellar mass burst fraction of $f_\mathrm{burst}=\Delta M_\star/M_{\star,\mathrm{merger}}=0.18 \pm 0.02$ for $10<\log (M_\star/\mathrm{M}_\odot)<11$ galaxies, with no evidence of a trend in stellar mass. The modeled burst fraction is robust to choice of parametric star formation history, as well as differences in burst duration. The result appears consistent with some prior observationally-derived values, but is significantly higher than that found in hydrodynamical simulations. Using published Luminous InfraRed Galaxy (LIRG) star formation rates, we find a burst duration increasing with stellar mass, from $120-250$ Myr. A comparison to published cold gas measurements indicates there is enough molecular gas available in very close pairs to fuel the burst. Additionally, given our stellar mass burst estimate, the predicted cold gas fraction remaining after the burst is consistent with observed post-coalescence mergers.Comment: Accepted for publication in MNRAS; 12 pages, 9 figure

A subset of dynamic actin rearrangements in Drosophila requires the Arp2/3 complex

The Arp2/3 complex has been shown to dramatically increase the slow spontaneous rate of actin filament nucleation in vitro, and it is known to be important for remodeling the actin cytoskeleton in vivo. We isolated and characterized loss of function mutations in genes encoding two subunits of the Drosophila Arp2/3 complex: Arpc1, which encodes the homologue of the p40 subunit, and Arp3, encoding one of the two actin-related proteins. We used these mutations to study how the Arp2/3 complex contributes to well-characterized actin structures in the ovary and the pupal epithelium. We found that the Arp2/3 complex is required for ring canal expansion during oogenesis but not for the formation of parallel actin bundles in nurse cell cytoplasm and bristle shaft cells. The requirement for Arp2/3 in ring canals indicates that the polymerization of actin filaments at the ring canal plasma membrane is important for driving ring canal growth

Constraining quenching timescales in galaxy clusters by forward-modelling stellar ages and quiescent fractions in projected phase space

We forward-model mass-weighted stellar ages (MWAs) and quiescent fractions in projected phase space (PPS), using data from the Sloan Digital Sky Survey, to jointly constrain an infall quenching model for galaxies in $\log(M_{\mathrm{vir}}/\mathrm{M}_{\odot})>14$ galaxy clusters at $z\sim 0$. We find the average deviation in MWA from the MWA-$M_\star$ relation depends on position in PPS, with a maximum difference between the inner cluster and infalling interloper galaxies of $\sim 1$ Gyr. Our model employs infall information from N-body simulations and stochastic star-formation histories from the UniverseMachine model. We find total quenching times of $t_\mathrm{Q}=3.7\pm 0.4$ Gyr and $t_\mathrm{Q}=4.0\pm 0.2$ Gyr after first pericentre, for $9<\log(M_{\star}/\mathrm{M}_{\odot})<10$ and $10<\log(M_{\star}/\mathrm{M}_{\odot})<10.5$ galaxies, respectively. By using MWAs, we break the degeneracy in time of quenching onset and timescale of star formation rate (SFR) decline. We find that time of quenching onset relative to pericentre is $t_{\mathrm{delay}}=3.5^{+0.6}_{-0.9}$ Gyr and $t_{\mathrm{delay}}=-0.3^{+0.8}_{-1.0}$ Gyr for our lower and higher stellar mass bins, respectively, and exponential SFR suppression timescales are $\tau_{\mathrm{env}}\leq 1.0$ Gyr and $\tau_{\mathrm{env}}\sim 2.3$ Gyr for our lower and higher stellar mass bins, respectively. Stochastic star formation histories remove the need for rapid infall quenching to maintain the bimodality in the SFR of cluster galaxies; the depth of the green valley prefers quenching onsets close to first pericentre and a longer quenching envelope, in slight tension with the MWA-driven results. Taken together these results suggest that quenching begins close to, or just after pericentre, but the timescale for quenching to be fully complete is much longer and therefore ram-pressure stripping is not complete on first pericentric passage.Comment: 21 pages, 13 figures, submitted to MNRA

NuSTAR hard X-ray observation of a sub-A class solar flare

We report a NuSTAR observation of a solar microflare, SOL2015-09-01T04. Although it was too faint to be observed by the GOES X-ray Sensor, we estimate the event to be an A0.1 class flare in brightness. This microflare, with only 5 counts per second per detector observed by RHESSI, is fainter than any hard X-ray (HXR) flare in the existing literature. The microflare occurred during a solar pointing by the highly sensitive NuSTAR astrophysical observatory, which used its direct focusing optics to produce detailed HXR microflare spectra and images. The microflare exhibits HXR properties commonly observed in larger flares, including a fast rise and more gradual decay, earlier peak time with higher energy, spatial dimensions similar to the RHESSI microflares, and a high-energy excess beyond an isothermal spectral component during the impulsive phase. The microflare is small in emission measure, temperature, and energy, though not in physical size; observations are consistent with an origin via the interaction of at least two magnetic loops. We estimate the increase in thermal energy at the time of the microflare to be 2.4x10^27 ergs. The observation suggests that flares do indeed scale down to extremely small energies and retain what we customarily think of as "flarelike" properties.Comment: Status: Accepted by the Astrophysical Journal, 2017 July 1

Drosophila Kelch regulates actin organization via Src64-dependent tyrosine phosphorylation

The Drosophila kelch gene encodes a member of a protein superfamily defined by the presence of kelch repeats. In Drosophila, Kelch is required to maintain actin organization in ovarian ring canals. We set out to study the actin cross-linking activity of Kelch and how Kelch function is regulated. Biochemical studies using purified, recombinant Kelch protein showed that full-length Kelch bundles actin filaments, and kelch repeat 5 contains the actin binding site. Two-dimensional electrophoresis demonstrated that Kelch is tyrosine phosphorylated in a src64-dependent pathway. Site-directed mutagenesis determined that tyrosine residue 627 is phosphorylated. A Kelch mutant with tyrosine 627 changed to alanine (KelY627A) rescued the actin disorganization phenotype of kelch mutant ring canals, but failed to produce wild-type ring canals. Electron microscopy demonstrated that phosphorylation of Kelch is critical for the proper morphogenesis of actin during ring canal growth, and presence of the nonphosphorylatable KelY627A protein phenocopied src64 ring canals. KelY627A protein in ring canals also dramatically reduced the rate of actin monomer exchange. The phenotypes caused by src64 mutants and KelY627A expression suggest that a major function of Src64 signaling in the ring canal is the negative regulation of actin cross-linking by Kelch

Distinct phosphorylation clusters determines the signalling outcome of the free fatty acid receptor FFA4/GPR120

It is established that long-chain free fatty acids including ω-3 fatty acids mediate an array of biological responses through members of the free fatty acid receptor family, which includes FFA4. However, the signalling mechanisms and modes of regulation of this receptor class remain unclear. Here we employ mass spectrometry to determine that phosphorylation of mouse (m)FFAR4 occurs at five serine and threonine residues clustered in two separable regions of the C terminal tail, designated cluster 1 (Thr347, Thr349 and Ser350) and cluster 2 (Ser357 and Ser361). Mutation of these phospho-acceptor sites to alanine completely prevented phosphorylation of mFFA4 but did not limit receptor coupling to ERK1/2 activation. Rather an inhibitor of Gq/11 proteins completely prevented receptor signalling to ERK1/2. In contrast, the recruitment of arrestin 3, receptor internalization and activation of Akt were regulated by mFFA4 phosphorylation. The analysis of mFFA4 phosphorylation-dependent signalling was extended further by selective mutations of the phospho-acceptor sites. Mutations within cluster 2 did not affect agonist activation of Akt but instead significantly compromised receptor internalization and arrestin 3 recruitment. Distinctly, mutation of the phospho-acceptor sites within cluster 1 had no effect on receptor internalization and a less extensive effect on arrestin 3 recruitment, but significantly uncoupled the receptor from Akt activation. These unique observations define differential effects on signalling mediated by phosphorylation at distinct locations. This hallmark feature supports the possibility that the signalling outcome of mFFA4 activation can be determined by the pattern of phosphorylation (phosphorylation barcode) at the C-terminus of the receptor

Impact of Hunting and Fishing on Mississippi Counties

Mississippi is largely agricultural with access to many watersheds, the combination of which makes the state attractive to hunters and fishermen. This paper investigates the impact of hunting/fishing on the sales tax revenue in the counties of Mississippi. Our results indicate that a 1% increase in the non-resident hunting/fishing license sold increases tax revenue by $14,535.65 per county on average.Resource /Energy Economics and Policy,

Fine root dynamics and trace gas fluxes in two lowland tropical forest soils

Fine root dynamics have the potential to contribute significantly to ecosystem-scale biogeochemical cycling, including the production and emission of greenhouse gases. This is particularly true in tropical forests which are often characterized as having large fine root biomass and rapid rates of root production and decomposition. We examined patterns in fine root dynamics on two soil types in a lowland moist Amazonian forest, and determined the effect of root decay on rates of C and N trace gas fluxes. Root production averaged 229 ( 35) and 153 ( 27) gm 2 yr 1 for years 1 and 2 of the study, respectively, and did not vary significantly with soil texture. Root decay was sensitive to soil texture with faster rates in the clay soil (k5 0.96 year 1) than in the sandy loam soil (k5 0.61 year 1),leading to greater standing stocks of dead roots in the sandy loam. Rates of nitrous oxide (N2O) emissions were significantly greater in the clay soil (13 1ngNcm 2 h 1) than in the sandy loam (1.4 0.2 ngNcm 2 h 1). Root mortality and decay following trenching doubled rates of N2O emissions in the clay and tripled them in sandy loam over a 1-year period. Trenching also increased nitric oxide fluxes, which were greater in the sandy loam than in the clay. We used trenching (clay only) and a mass balance approach to estimate the root contribution to soil respiration. In clay soil root respiration was 264–380 gCm 2 yr 1, accounting for 24% to 35% of the total soil CO2 efflux. Estimates were similar using both approaches. In sandy loam, root respiration rates were slightly higher and more variable (521 206 gCm2 yr 1) and contributed 35% of the total soil respiration. Our results show that soil heterotrophs strongly dominate soil respiration in this forest, regardless of soil texture. Our results also suggest that fine root mortality and decomposition associated with disturbance and land-use change can contribute significantly to increased rates of nitrogen trace gas emissions