Graphical Engine for Android Application Development

Import 05/08/2014Bakalářská práce je zaměřena na problematiku vývoje knihovny určené k tvorbě graficky náročných aplikací pro Android platformu. Dále řeší využití hardwarové akcelerace při vykreslování scény. Animace a transformace mesh objektů. Práci s audio soubory, texturami a shadery. Nastavení a ovládání kamery, či možnost využití fyzikálního enginu. Hlavním cílem práce je tedy vytvoření komplexní knihovny poskytující prostředí pro rychlý a snadný vývoj aplikací.Bachelor thesis is focused on issue of development a library to creating high graphical applications for Android platform. Then solves usage of hardware accelelaration. Animation and transformation of mesh objects. Work with audio files, textures and shaders. Configuration and camera controls or the possibility of using physics engine. The main purpose of this thesis is creation of comprehensive library that provides interface for fast and easy development of aplications.460 - Katedra informatikyvýborn

Participant Characteristics.

<p>For age, BMI, food preference, and FRPQ subscales, values are presented as mean (standard deviation). NW  =  normal-weight; OV/OB  =  overweight/obese. BMI indicates body mass index, based on height and weight obtained during testing.</p

Overweight/obese group (n = 21).

<p><i>Top Section</i>, <i>left</i>: plot of correlation between low-calorie vs. nonfood objects contrast during postmeal scan in the right putamen and FRPQ Impairment of Satiety subscale score. <i>Top Section, right</i>: magnitude of average activation for low-calorie foods and nonfood objects in the right putamen (Talairach coordinates x, y, z: 27, 3, −4) at postmeal scan. Beta values reflect BOLD contrast averaged across voxels in region for condition type. <i>Bottom Section</i>, <i>left</i>: plot of correlation between low-calorie vs. nonfood objects contrast during postmeal scan in the right amygdala and FRPQ Impairment of Satiety subscale score. <i>Bottom Section, right</i>: magnitude of average activation for low-calorie foods and nonfood objects in the right amygdala (Talairach coordinates x, y, z: 24, −7, −9) at postmeal scan. Beta values reflect BOLD contrast averaged across voxels in region for condition type.</p

Normal-weight group (n = 14).

<p><i>Left</i>: plot of correlation between high-calorie vs. nonfood objects contrast during premeal scan in the left amygdala and FRPQ Impairment of Satiety subscale score. <i>Right</i> top: activation of left amygdala to high-calorie food vs. nonfood objects (Talairach coordinates x, y, z: −26, −8, −14, t = 4.31, p<.0008, cluster threshold >5 voxels, FDR corrected at p<.05). Right and left are reversed by radiologic convention. <i>Right bottom:</i> magnitude of average activation for high-calorie foods and nonfood objects for right amygdala at premeal. Beta values reflect BOLD contrast averaged across voxels in region for condition type.</p

Additional file 3: of Restriction in lateral bending range of motion, lumbar lordosis, and hamstring flexibility predicts the development of low back pain: a systematic review of prospective cohort studies

Fully annotated forest plots for non-significant meta-analyses. (DOCX 449 kb

Effects of the addition of berberine to 5-ASA on PGE2 production and TNF-α secretion of lymphocytes.

<p>Experimental colitis was induced by giving mice three cycles of DSS. Each cycle consisted of 2% DSS for five days followed by drinking water for 14 days. Control mice received water alone. At the end of treatment, mice were killed and the lymphocyte were isolated from spleens and then cultured for the measurement of PGE2 production and TNF-α secretion without (A and B) or with PMA/ionomycin re-stimulation (C and D). ^a <i>P</i> < 0.05,^c <i>P</i> < 0.001 versus water mice; ^e <i>P</i> < 0.05, ^f <i>P</i> < 0.01 versus vehicle control (<i>n</i> = 3).</p

Potential drug accumulation and toxicity of addition of the berberine to 5-ASA in DSS-induced chronic relapsing colitis mice.

<p>Experimental colitis was induced by giving mice three cycles of DSS. Each cycle consisted of 2% DSS for five days followed by drinking water for 14 days. The first and last day of DSS treatment were designated as days 0 and 43, respectively. From day 13, experimental mice were treated daily with 5-ASA (200 mg/kg) alone or 5-ASA plus berberine hydrochloride (20 mg/kg) through gavage of 0.2 ml of the respective solution during the 30 days of concomitant DSS-colitis induction. Control mice received water alone. At the end of treatment, mice were killed and the concentrations of 5-ASA (A) and Ac-5-ASA (B) in serum, colon and spleen of colitis mice were measured by LC-MS analysis. Paraffin sections of spleen, lung, liver, and brain (C) were stained with hematoxylin and eosin (H&E). <i>n</i> = 4 mice per treatment group. Scale bar = 50 μm. RePu: Red pulp. WhPu: white pulp. Al: alveolus. InSe: interalveolar septum.CV: central vein. Ne: neuron.</p

Chemical structure of 5-ASA and berberine.

<p>Chemical structure of 5-ASA and berberine.</p

Effects of the addition of berberine to 5-ASA on colon tissue injury and mRNA expressions of mucin genes of DSS-induced chronic relapsing colitis mice.

<p>Experimental colitis was induced by giving mice three cycles of DSS. Each cycle consisted of 2% DSS for five days followed by drinking water for 14 days. The first and last day of DSS treatment were designated as days 0 and 43, respectively. From day 13, experimental mice were treated daily with 5-ASA (200 mg/kg) alone or 5-ASA plus berberine hydrochloride (20 mg/kg) through gavage of 0.2 ml of the respective solution during the 30 days of concomitant DSS-colitis induction. Control mice received water alone. At the end of treatment, mice were killed and the colon sections were collected and subjected to paraffin embedding and later hematoxylin and eosin (H&E) staining for light microscopic assessment (A) and colon injury/inflammation scoring (B). Scale bar = 50 or 100 μm. The colonic mRNA expression of MUC1 (C), MUC2 (D), MUC4 (E) and the housekeeping gene Gapdh in colitis mice was determined by RT-qPCR. The cytokine mRNA expression level in the water group was set as 100%, and mRNA expression levels in treated mice were compared with the water group. ^b <i>P</i> < 0.01 versus water; ^e <i>P</i> < 0.05, ^f <i>P</i> < 0.01 versus DSS; ^h <i>P</i> < 0.05 versus 5-ASA alone (n = 4 mice per treatment group).</p

Effects of the addition of berberine to 5-ASA on activated NFκB and JAK/STAT pathways of colon in DSS-induced chronic relapsing colitis mice.

<p>Experimental colitis was induced by giving mice three cycles of DSS. Each cycle consisted of 2% DSS for five days followed by drinking water for 14 days. The first and last day of DSS treatment were designated as days 0 and 43, respectively. From day 13, experimental mice were treated daily with 5-ASA (200 mg/kg) alone or 5-ASA plus berberine hydrochloride (20 mg/kg) through gavage of 0.2 ml of the respective solution during the 30 days of concomitant DSS-colitis induction. Control mice received water alone. At the end of treatment, mice were killed and the colon total cellular lysates were prepared for detecting the amounts of the indicated signals of NFκB (A and B) and JAK/STAT (C and D) pathways by Western blot analysis. Blotting for β-actin was used as a protein loading control. The relative density of the target protein bands on Western blot was compared with the β-actin band in each group. The density ratio in the water group was set as 1, and ratios of other three groups were compared with that of the water group. ^c <i>P</i> < 0.001 and ^d <i>P</i> < 0.0001 versus water; ^f <i>P</i> < 0.01, ^g <i>P</i> < 0.001, ^h <i>P</i> < 0.0001 versus DSS; ⁱ <i>P</i> < 0.05 and ^l <i>P</i> < 0.0001 versus 5-ASA alone (<i>n</i> = 4 mice per treatment group).</p