DESCRIPTIVE ANALYSIS OF HIP AND KNEE JOINT LOADING DURING REVERSE ROUNDHOUSE KICK (HOOK) KARATE KICK PERFORMED IN TRAINING AND COMPETITION MODES

The purpose of this study was to examine hip and knee joint loading during the performance of the common reverse roundhouse or hook kick (Ura Mawash geri. Twenty eight black belt karate players performed hook kicks in two different ways, training kick and competition kick. Motion and force data were collected with a VICON motion analysis system and two Kistler force plates. 3D joint motions and joint moments about the hip and knee of both the support leg and kicking leg for all kicks were calculated in Visual3D. The maximum moments were more varied between kick types for the kicking leg but the joint angles were similar in most cases. Joint loading higher than in the literature for cutting actions were found, with cutting actions considered a risky action

HIP AND KNEE LOADING OF KARATE PLAYERS PERFORMING TRAINING AND COMPETITION STYLE VERSIONS OF A ROUNDHOUSE KICK

The main purpose of this study was to examine hip and knee joint loading during the performance of the common roundhouse kicking technique in both a training mode and in a competition mode. 15 black belt karate players performed roundhouse kicks in two different ways, basic kick and competition kick. Motion and force data were collected with a VlCON motion analysis system and two Kistler force plates. 3D joint motions and joint moments about the hip and knee of both the support leg and kicking leg for all kicks were calculated. The maximum moments were more varied between kick types for the kicking leg but the joint angles were similar in most cases. Joint loading comparable to the literature was found for joint previously examined but several high joint moments at extremes of motion were found in the supporting leg

A critical review of Optimization MANET routing protocols

The main challenges in routing protocols are node mobility, resource constraints, error-prone channel states, and problems with hidden and unprotected terminals. This article provides an overview of the main protocols, their problems, and ways to improve them. The cell phone allows communication between two moving units via mobile stations (MS), mobile units, and landing units. The term "mobile ad hoc network" (MANET) refers to a group of devices connecting and communicating. The military, law enforcement, and emergency services have quickly become interested in MANETS because they provide a high quality of service. Path loss (PL), one of the biggest problems in wireless communications, can be caused by multiple reflections from an obstacle or by the source of the signal being far from the destination on the network. Multipath propagation, path loss, and interference reduce the network's quality of service (QoS). The best route is added to a router's routing table using a dynamic routing protocol, and an alternate path is chosen when the primary route is unavailable

Distinct genetic architectures for syndromic and nonsyndromic congenital heart defects identified by exome sequencing.

Congenital heart defects (CHDs) have a neonatal incidence of 0.8-1% (refs. 1,2). Despite abundant examples of monogenic CHD in humans and mice, CHD has a low absolute sibling recurrence risk (∼2.7%), suggesting a considerable role for de novo mutations (DNMs) and/or incomplete penetrance. De novo protein-truncating variants (PTVs) have been shown to be enriched among the 10% of 'syndromic' patients with extra-cardiac manifestations. We exome sequenced 1,891 probands, including both syndromic CHD (S-CHD, n = 610) and nonsyndromic CHD (NS-CHD, n = 1,281). In S-CHD, we confirmed a significant enrichment of de novo PTVs but not inherited PTVs in known CHD-associated genes, consistent with recent findings. Conversely, in NS-CHD we observed significant enrichment of PTVs inherited from unaffected parents in CHD-associated genes. We identified three genome-wide significant S-CHD disorders caused by DNMs in CHD4, CDK13 and PRKD1. Our study finds evidence for distinct genetic architectures underlying the low sibling recurrence risk in S-CHD and NS-CHD

Shared Situational Awareness within the Hospital Emergency Context: A Scoping Review

Background. Shared Situation Awareness (SSA) has been applied in many fields such as sport, the military and aviation with promising outcomes on team performance. The application of SSA within the hospital emergency healthcare context has not been explored yet. The aim of this scoping review is to explore and map literature related to shared situational awareness within the hospital emergency healthcare context. Methods. The Arksey and O’Malley (2005) framework was used in which three electronic databases were searched for evidence investigating SSA within a hospital emergency healthcare context. Results. A review of the literature showed a clear lack of evidence that directly investigates SSA within the context of hospital emergency care. In the emergency medical field, the term SSA is seldom used and ‘team situation awareness’ is the most frequently used term. The most common framework was the three-level framework. Two techniques were reported in the selected studies to investigate SSA (1) freeze probe technique and (2) observer-based rating technique. The freeze probe technique mandates a simulation or artificial environment, while the observer-based rating technique could be applied in an ecological as well as an artificial environment. There is no standardized technique to calculate the score of the SSA. Finally, there was a significant impact of SSA on clinical team performance as well as some related skills such as leadership, task management, mindfulness and task prioritization. Conclusions. Reviewing the literature revealed a lack of studies investigating the use of SSA within the context of hospital emergency care. There is also a lack of agreement on how a SSA score should be calculated. Further studies are required to overcome these issues

Distinct genetic architectures for syndromic and nonsyndromic congenital heart defects identified by exome sequencing

Congenital Heart Defects (CHD) have a neonatal incidence of 0.8-1%1,2. Despite abundant examples of monogenic CHD in humans and mice, CHD has a low absolute sibling recurrence risk (~2.7%)3, suggesting a considerable role for de novo mutations (DNM), and/or incomplete penetrance4,5. De novo protein-truncating variants (PTVs) have been shown to be enriched among the 10% of ‘syndromic’ patients with extra-cardiac manifestations6,7. We exome sequenced 1,891 probands, including both syndromic (S-CHD, n=610) and non-syndromic cases (NS-CHD, n=1,281). In S-CHD, we confirmed a significant enrichment of de novo PTVs, but not inherited PTVs, in known CHD-associated genes, consistent with recent findings8. Conversely, in NS-CHD we observed significant enrichment of PTVs inherited from unaffected parents in CHD-associated genes. We identified three novel genome-wide significant S-CHD disorders caused by DNMs in CHD4, CDK13 and PRKD1. Our study reveals distinct genetic architectures underlying the low sibling recurrence risk in S-CHD and NS-CHD