5 research outputs found
Anticoagulant selection in relation to the SAMe-TT2R2 score in patients with atrial fibrillation: The GLORIA-AF registry
- Author
- Abban D.W.
- Abdul N.
- Abud A.M.
- Adams F.
- Addala S.
- Adragao P.
- Ageno W.
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- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2021
- Field of study
No full textAim: The SAMe-TT2R2 score helps identify patients with atrial fibrillation (AF) likely to have poor anticoagulation control during anticoagulation with vitamin K antagonists (VKA) and those with scores >2 might be better managed with a target-specific oral anticoagulant (NOAC). We hypothesized that in clinical practice, VKAs may be prescribed less frequently to patients with AF and SAMe-TT2R2 scores >2 than to patients with lower scores. Methods and results: We analyzed the Phase III dataset of the Global Registry on Long-Term Oral Antithrombotic Treatment in Patients with Atrial Fibrillation (GLORIA-AF), a large, global, prospective global registry of patients with newly diagnosed AF and 651 stroke risk factor. We compared baseline clinical characteristics and antithrombotic prescriptions to determine the probability of the VKA prescription among anticoagulated patients with the baseline SAMe-TT2R2 score >2 and 64 2. Among 17,465 anticoagulated patients with AF, 4,828 (27.6%) patients were prescribed VKA and 12,637 (72.4%) patients an NOAC: 11,884 (68.0%) patients had SAMe-TT2R2 scores 0-2 and 5,581 (32.0%) patients had scores >2. The proportion of patients prescribed VKA was 28.0% among patients with SAMe-TT2R2 scores >2 and 27.5% in those with scores 642. Conclusions: The lack of a clear association between the SAMe-TT2R2 score and anticoagulant selection may be attributed to the relative efficacy and safety profiles between NOACs and VKAs as well as to the absence of trial evidence that an SAMe-TT2R2-guided strategy for the selection of the type of anticoagulation in NVAF patients has an impact on clinical outcomes of efficacy and safety. The latter hypothesis is currently being tested in a randomized controlled trial. Clinical trial registration: URL: https://www.clinicaltrials.gov//Unique identifier: NCT01937377, NCT01468701, and NCT01671007
Anticoagulant selection in relation to the SAMe-TT2R2 score in patients with atrial fibrillation: The GLORIA-AF registry
- Publication venue
- 'Elsevier BV'
- Publication date
- 01/01/2021
- Field of study
No full textAim: The SAMe-TT2R2 score helps identify patients with atrial fibrillation (AF) likely to have poor anticoagulation control during anticoagulation with vitamin K antagonists (VKA) and those with scores >2 might be better managed with a target-specific oral anticoagulant (NOAC). We hypothesized that in clinical practice, VKAs may be prescribed less frequently to patients with AF and SAMe-TT2R2 scores >2 than to patients with lower scores. Methods and results: We analyzed the Phase III dataset of the Global Registry on Long-Term Oral Antithrombotic Treatment in Patients with Atrial Fibrillation (GLORIA-AF), a large, global, prospective global registry of patients with newly diagnosed AF and ≥1 stroke risk factor. We compared baseline clinical characteristics and antithrombotic prescriptions to determine the probability of the VKA prescription among anticoagulated patients with the baseline SAMe-TT2R2 score >2 and ≤ 2. Among 17,465 anticoagulated patients with AF, 4,828 (27.6%) patients were prescribed VKA and 12,637 (72.4%) patients an NOAC: 11,884 (68.0%) patients had SAMe-TT2R2 scores 0-2 and 5,581 (32.0%) patients had scores >2. The proportion of patients prescribed VKA was 28.0% among patients with SAMe-TT2R2 scores >2 and 27.5% in those with scores ≤2. Conclusions: The lack of a clear association between the SAMe-TT2R2 score and anticoagulant selection may be attributed to the relative efficacy and safety profiles between NOACs and VKAs as well as to the absence of trial evidence that an SAMe-TT2R2-guided strategy for the selection of the type of anticoagulation in NVAF patients has an impact on clinical outcomes of efficacy and safety. The latter hypothesis is currently being tested in a randomized controlled trial. Clinical trial registration: URL: https://www.clinicaltrials.gov//Unique identifier: NCT01937377, NCT01468701, and NCT01671007. © 2020 Hellenic Society of Cardiolog
Anticoagulant selection in relation to the SAMe-TT2R2 score in patients with atrial fibrillation: The GLORIA-AF registry
- Author
- Abban Dzifa Wosornu
- Abdul Nasser
- Abud Atilio Marcelo
- Adams Fran
- Addala Srinivas
- Adragão Pedro
- Ageno Walter
- Aggarwal Rajesh
- Agosti Sergio
- Agostoni Piergiuseppe
- Aguilar Francisco
- Aguinaga Luis
- Ahmed Jameel
- Aiello Allessandro
- Ainsworth Paul
- Aiub Jorge Roberto
- Al-Dallow Raed
- Alderson Lisa
- Aldrete Velasco Jorge Antonio
- Alexopoulos Dimitrios
- Aliyar Pareed
- Almeida Luísa Fonseca
- Alonso David
- Alves da Costa Fernando Augusto
- Amado José
- Amara Walid
- Amelot Mathieu
- Amjadi Nima
- Ammirati Fabrizio
- Andrade Marianna
- Andrawis Nabil
- Angela Koh Su Mei
- Annoni Giorgio
- Ansalone Gerardo
- Ariani M.Kevin
- Arias Juan Carlos
- Armero Sébastien
- Arora Chander
- Asensi Joaquín Osca
- Aslam Muhammad Shakil
- Asselman M.
- Audouin Philippe
- Augenbraun Charles
- Aydin S.
- Ayryanova Ivaneta
- Aziz Emad
- Backes Luciano Marcelo
- Badings E.
- Bagni Ermentina
- Baker Seth
- Bala Richard
- Baldi Antonio
- Bando Shigenobu
- Banerjee Subhash
- Bank Alan
- Barr Craig
- Bartlett Maria
- Baula Giovanni
- Behrens Steffen
- Bell Alan
- Benedetti Raffaella
- Benhalima Bouziane
- Berg J.M. Ten
- Bergler-Klein Jutta
- Berneau Jean-Baptiste
- Bernstein Richard
- Berrospi Percy
- Berti Sergio
- Berz Andrea
- Best Elizabeth
- Bettencourt Paulo
- Betzu Robert
- Bhagwat Ravi
- Bhatta Luna
- Biscione Francesco
- Bisignani Giovanni
- Black Toby
- Bloch Michael
- Bloom Stephen
- Blumberg Edwin
- Bo Mario
- Bodes Rafael Salguero
- Bollmann Andreas
- Bongiorni Maria Grazia
- Boriani Giuseppe
- Boswijk D.J.
- Bott Jochen
- Bottacchi Edo
- Bradman Drew
- Brautigam Donald
- Breton Nicolas
- Briones Ignacio Rodriguez
- Brkljacic Diana Delic
- Brouwers P.J.A.M.
- Browne Kevin
- Bruni A.
- Brunschwig Claude
- Bryan Yan Ping Yen
- Buathier Hervé
- Buenostro Gabriel Contreras
- Buhl Aurélie
- Bullinga John
- Bøhmer Ellen
- Cabrera Jose Walter
- Caccavo Alberto
- Cai Shanglang
- Caine Sarah
- Calvi Valeria
- Calò Leonardo
- Campos Rodrigo Noronha
- Candeias Rui
- Capuano Vincenzo
- Capucci Alessandro
- Caputo Ronald
- Cardona Francisco
- Carlos da Costa Darrieux Francisco
- Carolei Antonio
- Carreño Susana
- Carvalho Paula
- Cary Susanna
- Casu Gavino
- Cavallini Claudio
- Cayla Guillaume
- Celentano Aldo
- Cha Kwang Soo
- Cha Tae-Joon
- Chae Jei Keon
- Chalamidas Kathrine
- Challappa Krishnan
- Chand Sunil Prakash
- Chandrashekar Harinath
- Chartier Ludovic
- Chatterjee Kausik
- Chavez Ayala Carlos Antero
- Cheema Aamir
- Cheema Amjad
- Chen Jyh Hong
- Chen Lin
- Chen Shih-Ann
- Chiang Fu-Tien
- Chiarella Francesco
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- Szavits-Nossan Janko
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- Takagi Yuichiro
- Tamm Angelika
- Tanaka Katsumi
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- Tang Aylmer
- Tang Sherman
- Tarsi Giovanni
- Tassinari Tiziana
- Tayal Ashis
- Tayebjee Muzahir
- Tcurner Wayne
- Tesloianu Dan
- Teutsch Christine
- The Salem H.K.
- Thomas Dierk
- Thompson Milla
- Timsit Serge
- Tobaru Tetsuya
- Tomasik Andrzej
- Topete Gilberto de La Pena
- Torosoff Mikhail
- Torres Torres Ivett Guadalupe
- Touze Emmanuel
- Trendafilova Elina
- Tsai W. Kevin
- Tse Hung Fat
- Tsutsui Hiroshi
- Tu Tian Ming
- Tuininga Ype
- Turakhia Minang
- Turk Samir
- Tveit Arnljot
- Tytus Richard
- Valadão C.
- Vallejo Gregorio Sanchez
- van Bergen P.F.M.M.
- van de Borne Philippe
- van den Berg B.J.
- van Der Zwaan C.
- van Eck M.
- Vanacker Peter
- Vasilev Dimo
- Vasilikos Vasileios
- Vasilyev Maxim
- Veerareddy Srikar
- Venkataraman Asok
- Vera Tomas Ripolil
- Verdecchia Paolo
- Versaci Francesco
- Vester Ernst Günter
- Vial Hubert
- Vicenzo de Paola Angelo Amato
- Victory Jason
- Villamil Alejandro
- Vincent Marc
- Vlastaris Anthony
- Vora Kishor
- Vranian Robert
- Wakefield Paul
- Wang Feng
- Wang Mingsheng
- Wang Ningfu
- Wang Tian
- Wang Xinhua
- Warner Alberta
- Watanabe Kouki
- Wei Jeanne
- Weimar Christian
- Weiner Stanislav
- Weinrich Renate
- Wen Ming-Shien
- Wiemer Marcus
- Wiggers Preben
- Wilke Andreas
- Williams David
- Williams Marcus
- Witzenbichler Bernhard
- Wong Brian
- Wozakowska-Kaplon Beata
- Wu Richard
- Wu Shulin
- Wunderlich Silke
- Wyatt Nell
- Wylie John (jack)
- Xu Xiangdong
- Xu Yong
- Yamanoue Hiroki
- Yamashita Takeshi
- Yang Tianlun
- Yao Jing
- Yeh Kuo-Ho
- Yin Wei Hsian
- Yotov Yoto
- Zahn Ralf
- Zanetti Fernando Lanas
- Publication venue
- Elsevier
- Publication date
- 01/03/2021
- Field of study
No full textAim: The SAMe-TT2R2 score helps identify patients with atrial fibrillation (AF) likely to have poor anticoagulation control during anticoagulation with vitamin K antagonists (VKA) and those with scores >2 might be better managed with a target-specific oral anticoagulant (NOAC). We hypothesized that in clinical practice, VKAs may be prescribed less frequently to patients with AF and SAMe-TT2R2 scores >2 than to patients with lower scores. Methods and results: We analyzed the Phase III dataset of the Global Registry on Long-Term Oral Antithrombotic Treatment in Patients with Atrial Fibrillation (GLORIA-AF), a large, global, prospective global registry of patients with newly diagnosed AF and ≥1 stroke risk factor. We compared baseline clinical characteristics and antithrombotic prescriptions to determine the probability of the VKA prescription among anticoagulated patients with the baseline SAMe-TT2R2 score >2 and ≤ 2. Among 17,465 anticoagulated patients with AF, 4,828 (27.6%) patients were prescribed VKA and 12,637 (72.4%) patients an NOAC: 11,884 (68.0%) patients had SAMe-TT2R2 scores 0-2 and 5,581 (32.0%) patients had scores >2. The proportion of patients prescribed VKA was 28.0% among patients with SAMe-TT2R2 scores >2 and 27.5% in those with scores ≤2. Conclusions: The lack of a clear association between the SAMe-TT2R2 score and anticoagulant selection may be attributed to the relative efficacy and safety profiles between NOACs and VKAs as well as to the absence of trial evidence that an SAMe-TT2R2-guided strategy for the selection of the type of anticoagulation in NVAF patients has an impact on clinical outcomes of efficacy and safety. The latter hypothesis is currently being tested in a randomized controlled trial. Clinical trial registration: URL: https://www.clinicaltrials.gov//Unique identifier: NCT01937377, NCT01468701, and NCT01671007
Anticoagulant selection in relation to the SAMe-TT2R2 score in patients with atrial fibrillation: The GLORIA-AF registry
- Author
- A. Bruni
- A. de Veer
- A. Duygun
- A. Huizinga
- A. Shekhar Pandey
- Aamir Cheema
- Abraham Salacata
- Achille Gaspardone
- Achim Küppers
- Adalberto Menezes Lorga Filho
- Adam Sokal
- Adnan El Jabali
- Adrien Salem
- AJIT Singh Khaira
- Akber Mohammed
- Alan Bank
- Alan Bell
- Alberta L. Warner
- Alberto Caccavo
- Alberto Conti
- Alberto Giniger
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- Steven Zizzo
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- Stuart Zarich
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- Sébastien Armero
- T. Lenderink
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- Tamotsu Sakamoto
- Tanyanan Tanawuttiwat
- Tatiana Cárdenas Rizo
- Tatiana Novikova
- Taya V. Glotzer
- Teresa Schnabel
- Teresiano De Franceschi
- Terri K. Peters
- Terry McCormack
- Tetsuo Hisadome
- Tetsuya Haruna
- Tetsuya Tobaru
- Thomas J. Mulhearn
- Thomas Kümler
- Thuraia Nageh
- Tian Ming Tu
- Tian Wang
- Tianlun Yang
- Tillmann Maurer
- Tim A. Simmers
- Tina Cullen
- Tiziana Tassinari
- Tiziano Moccetti
- Toby Black
- Tomas Joen-Jakobsen
- Tomas Ripolil Vera
- Torben Larsen
- Toru Nakayama
- Tristan Mirault
- Ursula Rauch-Kroehnert
- Uwe Gremmler
- Valeria Calvi
- Vanja Basic Kes
- Vasileios Vasilikos
- Venkat Gottipaty
- Venkat Iyer
- Venkatesh K. Gurusamy
- Vicente Bertomeu Gonzalez
- Vicente Davalos
- Vicente Montagud
- Vicky Maqueda
- Victoria Padilla Rios
- Vijay Kumar Sharma
- Viktor Persic
- Vinay Shah
- Vincenzo Capuano
- Vittorio Pengo
- Víctor Manuel González López
- W. Kevin Tsai
- Waldemar Krysiak
- Walid Amara
- Walter Ageno
- Wanda Sudnik
- Wayne Tcurner
- Wei Hsian Yin
- Weihua Li
- Weimar de Souza
- Wen Ter Lai
- Wenxia Zong
- Werner Jung
- Werner Rieker
- William Eaves
- William French
- William J. Kostis
- William McGarity
- William Pentz
- Witold Streb
- Wladmir Faustino Saporito
- Wlodzimierz Musial
- Xavier Ducrocq
- Xi Su
- Xiang Gu
- Xiangdong Xu
- Xiaodong Li
- Xiaofei Sun
- Xiaoming Li
- Xingwei Zhang
- Xinhua Wang
- Yan Carlos Duarte Vera
- Yang Zheng
- Yannie Soo Oi Yan
- Yat Yin Lam
- Yinglong Hou
- Yong Keun Cho
- Yong Seog Oh
- Yong Xu
- Yorihiko Higashino
- Yoto Yotov
- Young Hoon Kim
- Ype Tuininga
- Yuichiro Nakamura
- Yuichiro Takagi
- Yujie Zhao
- Yutaka Furukawa
- Yves Cottin
- Z. Kromhout
- Zdravka Poljakovic
- Zhanquan Li
- Zhaohui Qiu
- Zhichen Zhao
- Publication venue
- Elsevier BV
- Publication date
- 01/03/2021
- Field of study
No full textAntiinflammatory therapy with canakinumab for atherosclerotic disease
- Author
- Abaci F.
- Abbate A.
- Abdullakutty J.
- Abhaichand R.
- Abhyankar A.
- Abib E.Jr.
- Aboufakher R.
- Abrantes J.
- Abreu M.
- Abulencia K.
- Acampora D.
- Acampora M.
- Accini Mendoza JL.
- Aceto L.
- Acevedo B.
- Acevedo L.
- Acheatel R.
- Actis M.V.
- Adams A.
- Adams M.
- Adjic N.C.
- Affaki G.S.
- Agafina A.
- Agarwal D.K.
- Aggarwal R.K.
- Agosta G.F.
- Aguilar M.
- Ahire N.
- Ahmad I.
- Ahmed S.H.
- Ahn Y.
- Aish B.
- Aiub F.
- Aiub J.
- Ajax K.
- Ajioka M.
- Akai Y.
- Akatova E.
- Akrap B.
- Akyea-Djamson A.
- Al Joundi T.
- Al-Khalili F.
- Alan A.
- Albisu J.
- Alcalde J.M.
- Alcide P.
- Alfieri A.
- Alfonso T.
- Alfrey A.
- Allen J.
- Alllison D.C.
- Almaliky T.
- Alvarisqueta A.
- Amerena J.
- Amos A.
- Anadiotis A.
- Andersen K.
- Andor M.
- Angelova I.
- Angiolillo D.
- Anita S.
- Anker S.D.
- Anneveldt A.
- Antalik L.
- Antolick A.
- Antonicelli R.
- Aparicio C.V.
- Apel T.
- Apostolovic S.
- Ara M.
- Aragorn L.
- Arango J.L.
- Araujo Fonseca M.
- Ardissino D.
- Arenas Leon JL.
- Arevalo S.
- Argiolas G.
- Arif I.
- Armas E.
- Aron G.
- Arora S.
- Arthur A.
- Asafu-Adjaye N.
- Ashcom T.
- Ashford M.
- Ashino K.
- Asim Oktay AO.
- Aslam A.
- Assarsson E.
- Ata B.
- Ather N.
- Atieh M.
- Aull L.
- Avalos V.
- Avdonina N.
- Awasthi A.K.
- Axthelm C.
- Ayala M.
- Ayasse D.
- Ayasse M.
- Azizad M.
- Azize G.M.
- Baar M.
- Babu R.
- Backer T.
- Badea C.
- Baden M.
- Baehl S.
- Baek C.
- Baeumer A.
- Bagi E.S.
- Bai A.
- Bailey K.
- Bailey S.
- Bair S.
- Baker A.
- Baker C.
- Bakhai A.
- Bakker H.
- Baksi A.
- Baldin M.G.
- Bali H.K.
- Ballantyne C.
- Ballmajo M.
- Balode A.
- Balukova E.
- Bang S.A.
- Banker D.
- Banker K.
- Baquero A.
- Barbarash O.L.
- Barbato E.
- Barbosa E.
- Barnett S.
- Baron S.
- Barreto M.
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No full textBACKGROUND: Experimental and clinical data suggest that reducing inflammation without affecting lipid levels may reduce the risk of cardiovascular disease. Yet, the inflammatory hypothesis of atherothrombosis has remained unproved. METHODS: We conducted a randomized, double-blind trial of canakinumab, a therapeutic monoclonal antibody targeting interleukin-1β, involving 10,061 patients with previous myocardial infarction and a high-sensitivity C-reactive protein level of 2 mg or more per liter. The trial compared three doses of canakinumab (50 mg, 150 mg, and 300 mg, administered subcutaneously every 3 months) with placebo. The primary efficacy end point was nonfatal myocardial infarction, nonfatal stroke, or cardiovascular death. RESULTS: At 48 months, the median reduction from baseline in the high-sensitivity C-reactive protein level was 26 percentage points greater in the group that received the 50-mg dose of canakinumab, 37 percentage points greater in the 150-mg group, and 41 percentage points greater in the 300-mg group than in the placebo group. Canakinumab did not reduce lipid levels from baseline. At a median follow-up of 3.7 years, the incidence rate for the primary end point was 4.50 events per 100 person-years in the placebo group, 4.11 events per 100 person-years in the 50-mg group, 3.86 events per 100 person-years in the 150-mg group, and 3.90 events per 100 person-years in the 300-mg group. The hazard ratios as compared with placebo were as follows: in the 50-mg group, 0.93 (95% confidence interval [CI], 0.80 to 1.07; P=0.30); in the 150-mg group, 0.85 (95% CI, 0.74 to 0.98; P=0.021); and in the 300-mg group, 0.86 (95% CI, 0.75 to 0.99; P=0.031). The 150-mg dose, but not the other doses, met the prespecified multiplicity-adjusted threshold for statistical significance for the primary end point and the secondary end point that additionally included hospitalization for unstable angina that led to urgent revascularization (hazard ratio vs. placebo, 0.83; 95% CI, 0.73 to 0.95; P=0.005). Canakinumab was associated with a higher incidence of fatal infection than was placebo. There was no significant difference in all-cause mortality (hazard ratio for all canakinumab doses vs. placebo, 0.94; 95% CI, 0.83 to 1.06; P=0.31). CONCLUSIONS: Antiinflammatory therapy targeting the interleukin-1β innate immunity pathway with canakinumab at a dose of 150 mg every 3 months led to a significantly lower rate of recurrent cardiovascular events than placebo, independent of lipid-level lowering. Copyright © 2017 Massachusetts Medical Society
