Another factor to consider in choosing a child support guideline: Errors in child support calculations

In an effort to standardize the calculation of monthly child support awards, the federal government requires states to use preestablished formulas to determine the amount of awards. However, because of human error, differences in the experience and training of the officials making the calculations, and the extent to which computers are used to calculate the awards, the formulas do not always yield the same result. In fact, the discrepancy between the amount calculated by an individual child support official and the approved amount as calculated by the state in which that official works can be quite large, on the order of several hundred dollars. Adopting simpler formulas will reduce errors; this should be a priority even if child support officials use computers to calculate award amounts (computers can reduce errors but will not eliminate them, particularly in the case of complex formulas). Efforts to further the training and education of personnel who calculate awards would also help, and child support offices should revise their formulas to cover high-income cases.

Child support enforcement for teenage fathers: Problems and prospects

The NLSY data indicate that about 7.3 percent of teenage males become fathers and that very few of these fathers live with their children. Father absence and the concurrent increase in female-headed households are closely associated with the impoverishment of children. Most absent teen fathers never come into contact with the child support enforcement program, and the extent to which they financially support their children informally is not well understood. While the income of absent teen fathers is low in the teen years, it increases over time, as does the potential for collecting child support. Nevertheless, men who were absent teen fathers earn less in early adulthood than men who deferred parenting until age twenty or later and teen fathers who lived with their children. Early establishment of paternity and greater standardization in the treatment of adolescent fathers by the child support enforcement program are recommended. Further, the substantial and persistent income deficit experienced by adolescent fathers who live apart from their children raises an interesting dilemma. While children may benefit financially and psychosocially from living with two parents, the lower income of men who were absent teenage fathers may make them poor marital prospects. This raises doubts about the recent recommendations of some scholars that we should bring back the shotgun wedding.

Self-gravitating magnetised tori around black holes in general relativity

We investigate stationary, self-gravitating, magnetised disks (or tori) around black holes. The models are obtained by numerically solving the coupled system of the Einstein equations and the equations of ideal general-relativistic magnetohydrodynamics. The mathematical formulation and numerical aspects of our approach are similar to those reported in previous works modeling stationary self-gravitating perfect-fluid tori, but the inclusion of magnetic fields represents a new ingredient. Following previous studies of purely hydrodynamical configurations, we construct our models assuming Keplerian rotation in the disks and both spinning and spinless black holes. We focus on the case of a toroidal distribution of the magnetic field and build a large set of models corresponding to a wide range of values of the magnetisation parameter, starting with weakly magnetised disks and ending at configurations in which the magnetic pressure dominates over the thermal one. In all our models, the magnetic field affects the equilibrium structure of the torus mainly due to the magnetic pressure. In particular, an increasing contribution of the magnetic field shifts the location of the maximum of the rest-mass density towards inner regions of the disk. The total mass of the system and the angular momentum are affected by the magnetic field in a complex way, that depends on the black hole spin and the location of the inner radius of the disk. The non-linear dynamical stability of the solutions presented in this paper will be reported elsewhere.Comment: 17 pages, 5 figures, 1 tabl

Using Networks To Build Collaborative And Equitable Food Systems

Local food systems are widely regarded as go-to examples for fostering rural-urban and farmer-consumer connections, but they require significant investments in stakeholder networks and the institutional infrastructure necessary to sustain and expand them. In our latest research brief, authors Rich Pirog and Marcus A. Coleman focus on local food systems as vehicles for collaboration and racial equity among multiple stakeholders and networks. They highlight examples of statewide, regional, and national initiatives to illustrate the power of networks to foster deeper levels of collaboration and reciprocity. They also explore the vital roles of Minority Serving Institutions (MSI) and Cooperative Extension

Skeletal dysplasias associated with mild myopathy a clinical and molecular review

Musculoskeletal system is a complex assembly of tissues which acts as scaffold for the body and enables locomotion. It is often overlooked that different components of this system may biomechanically interact and affect each other. Skeletal dysplasias are diseases predominantly affecting the development of the osseous skeleton. However, in some cases skeletal dysplasia patients are referred to neuromuscular clinics prior to the correct skeletal diagnosis. The muscular complications seen in these cases are usually mild and may stem directly from the muscle defect and/or from the altered interactions between the individual components of the musculoskeletal system. A correct early diagnosis may enable better management of the patients and a better quality of life. This paper attempts to summarise the different components of the musculoskeletal system which are affected in skeletal dysplasias and lists several interesting examples of such diseases in order to enable better understanding of the complexity of human musculoskeletal system. Copyright © 2010 K. A. Piróg and M. D. Briggs

A preliminary investigation of materialism and impulsiveness as predictors of technological addictions among young adults

Background and aims: The primary objective of the present research is to investigate the drivers of technological addiction in college students — heavy users of Information and Communication Technology (ICT). The study places cell phone and instant messaging addiction in the broader context of consumption pathologies, investigating the influence of materialism and impulsiveness on these two technologies. Clearly, cell phones serve more than just a utilitarian purpose. Cell phones are used in public and play a vital role in the lives of young adults. The accessibility of new technologies, like cell phones, which have the advantages of portability and an ever increasing array of functions, makes their over-use increasingly likely. Methods: College undergraduates (N = 191) from two U.S. universities completed a paper and pencil survey instrument during class. The questionnaire took approximately 15–20 minutes to complete and contained scales that measured materialism, impulsiveness, and mobile phone and instant messaging addiction. Results: Factor analysis supported the discriminant validity of Ehrenberg, Juckes, White and Walsh's (2008) Mobile Phone and Instant Messaging Addictive Tendencies Scale. The path model indicates that both materialism and impulsiveness impact the two addictive tendencies, and that materialism's direct impact on these addictions has a noticeably larger effect on cell phone use than instant messaging. Conclusions: The present study finds that materialism and impulsiveness drive both a dependence on cell phones and instant messaging. As Griffiths (2012) rightly warns, however, researchers must be aware that one's addiction may not simply be to the cell phone, but to a particular activity or function of the cell phone. The emergence of multi-function smart phones requires that research must dig beneath the technology being used to the activities that draw the user to the particular technology

A dualistic model of primary anal canal adenocarcinoma with distinct cellular origins, etiologies, inflammatory microenvironments and mutational signatures: implications for personalised medicine.

Primary adenocarcinoma of the anal canal is a rare and aggressive gastrointestinal disease with unclear pathogenesis. Because of its rarity, no clear clinical practice guideline has been defined and a targeted therapeutic armamentarium has yet to be developed. The present article aimed at addressing this information gap by in-depth characterising the anal glandular neoplasms at the histologic, immunologic, genomic and epidemiologic levels. In this multi-institutional study, we first examined the histological features displayed by each collected tumour (n = 74) and analysed their etiological relationship with human papillomavirus (HPV) infection. The intratumoural immune cell subsets (CD4, CD8, Foxp3), the expression of immune checkpoints (PD-1, PD-L1), the defect in mismatch repair proteins and the mutation analysis of multiple clinically relevant genes in the gastrointestinal cancer setting were also determined. Finally, the prognostic significance of each clinicopathological variable was assessed. Phenotypic analysis revealed two region-specific subtypes of anal canal adenocarcinoma. The significant differences in the HPV status, density of tumour-infiltrating lymphocytes, expression of immune checkpoints and mutational profile of several targetable genes further supported the separation of these latter neoplasms into two distinct entities. Importantly, anal gland/transitional-type cancers, which poorly respond to standard treatments, displayed less mutations in downstream effectors of the EGFR signalling pathway (i.e., KRAS and NRAS) and demonstrated a significantly higher expression of the immune inhibitory ligand-receptor pair PD-1/PD-L1 compared to their counterparts arising from the colorectal mucosa. Taken together, the findings reported in the present article reveal, for the first time, that glandular neoplasms of the anal canal arise by HPV-dependent or independent pathways. These etiological differences leads to both individual immune profiles and mutational landscapes that can be targeted for therapeutic benefits