More is more in language learning:reconsidering the less-is-more hypothesis

The Less-is-More hypothesis was proposed to explain age-of-acquisition effects in first language (L1) acquisition and second language (L2) attainment. We scrutinize different renditions of the hypothesis by examining how learning outcomes are affected by (1) limited cognitive capacity, (2) reduced interference resulting from less prior knowledge, and (3) simplified language input. While there is little-to-no evidence of benefits of limited cognitive capacity, there is ample support for a More-is-More account linking enhanced capacity with better L1- and L2-learning outcomes, and reduced capacity with childhood language disorders. Instead, reduced prior knowledge (relative to adults) may afford children with greater flexibility in inductive inference; this contradicts the idea that children benefit from a more constrained hypothesis space. Finally, studies of childdirected speech (CDS) confirm benefits from less complex input at early stages, but also emphasize how greater lexical and syntactic complexity of the input confers benefits in L1-attainment

What can developmental disorders tell us about the neurocomputational constraints that shape development? the case of Williams syndrome

The uneven cognitive phenotype in the adult outcome of Williams syndrome has led some researchers to make strong claims about the modularity of the brain and the purported genetically determined, innate specification of cognitive modules. Such arguments have particularly been marshaled with respect to language. We challenge this direct generalization from adult phenotypic outcomes to genetic specification and consider instead how genetic disorders provide clues to the constraints on plasticity that shape the outcome of development. We specifically examine behavioral studies, brain imaging, and computational modeling of language in Williams syndrome but contend that our theoretical arguments apply equally to other cognitive domains and other developmental disorders. While acknowledging that selective deficits in normal adult patients might justify claims about cognitive modularity, we question whether similar, seemingly selective deficits found in genetic disorders can be used to argue that such cognitive modules are prespecified in infant brains. Cognitive modules are, in our view, the outcome of development, not its starting point. We note that most work on genetic disorders ignores one vital factor, the actual process of ontogenetic development, and argue that it is vital to view genetic disorders as proceeding under different neurocomputational constraints, not as demonstrations of static modularity

Enlightened Romanticism: Mary Gartside’s colour theory in the age of Moses Harris, Goethe and George Field

The aim of this paper is to evaluate the work of Mary Gartside, a British female colour theorist, active in London between 1781 and 1808. She published three books between 1805 and 1808. In chronological and intellectual terms Gartside can cautiously be regarded an exemplary link between Moses Harris, who published a short but important theory of colour in the second half of the eighteenth century, and J.W. von Goethe’s highly influential Zur Farbenlehre, published in Germany in 1810. Gartside’s colour theory was published privately under the disguise of a traditional water colouring manual, illustrated with stunning abstract colour blots (see example above). Until well into the twentieth century, she remained the only woman known to have published a theory of colour. In contrast to Goethe and other colour theorists in the late 18th and early 19th century Gartside was less inclined to follow the anti-Newtonian attitudes of the Romantic movement

Learning, Arts, and the Brain: The Dana Consortium Report on Arts and Cognition

Reports findings from multiple neuroscientific studies on the impact of arts training on the enhancement of other cognitive capacities, such as reading acquisition, sequence learning, geometrical reasoning, and memory

UNERTAN SYNDROME: A CASE SERIES DEMONSTRTAING HUMAN DEVOLUTION

A large family with six individuals exhibiting the Unertan syndrome (UTS) was identified residing in southern Turkey. All of the individuals had mental impairments and walked on all four extremities. The intra-familial marriages suggested that the UTS is an autosomal recessive disorder. The inferior portions of the cerebellum and vermis were absent as evidenced by MRI and CT scans. The height and head circumference of those affected were within normal ranges. Barany’s test suggested normal vestibular system function. The subjects could not name objects or their close relatives. The males (n = 4) could understand simple questions, answering them with only one or two sounds. The females (n = 2) were superior to the males with respect to language skills and walking, suggesting an association between walking and speaking abilities. One male exhibited three walking patterns at the same time: quadripedal, tiptoe, and scissor walking. Another male used two walking styles: quadripedal and toe-walking. It is emphasized that there are important differences between the UTS and the disequilibrium syndrome (DES). It is suggested that the inability to walk upright in those affected with the UTS may be best explained by a disturbance in lateral-balance mechanisms. An interruption of locomotor development during the transition from quadripedality to bipedality may result in habitual walking on all four extremities and is normal in some children. Since quadripedal gait is an ancestral trait, individuals with the UTS, exhibiting a manifestation of reverse evolution in humans, may be considered an experiment of nature, useful in understanding the mechanisms underlying the transition from quadripedality to bipedality during human evolution. The proposed mutant gene or gene pool playing a role in human quadrupedality may also be responsible for human bipedality at the same time. Herein there is no intent to insult or injure, rather this report is an endeavor to better understand human beings