12 research outputs found

    We Are Not Your Real Parents: Telling Causal from Confounded using MDL

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    Given data over variables (X1,...,Xm,Y)(X_1,...,X_m, Y) we consider the problem of finding out whether XX jointly causes YY or whether they are all confounded by an unobserved latent variable ZZ. To do so, we take an information-theoretic approach based on Kolmogorov complexity. In a nutshell, we follow the postulate that first encoding the true cause, and then the effects given that cause, results in a shorter description than any other encoding of the observed variables. The ideal score is not computable, and hence we have to approximate it. We propose to do so using the Minimum Description Length (MDL) principle. We compare the MDL scores under the models where XX causes YY and where there exists a latent variables ZZ confounding both XX and YY and show our scores are consistent. To find potential confounders we propose using latent factor modeling, in particular, probabilistic PCA (PPCA). Empirical evaluation on both synthetic and real-world data shows that our method, CoCa, performs very well -- even when the true generating process of the data is far from the assumptions made by the models we use. Moreover, it is robust as its accuracy goes hand in hand with its confidence

    Debiased Recommendation with User Feature Balancing

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    Debiased recommendation has recently attracted increasing attention from both industry and academic communities. Traditional models mostly rely on the inverse propensity score (IPS), which can be hard to estimate and may suffer from the high variance issue. To alleviate these problems, in this article, we propose a novel debiased recommendation framework based on user feature balancing. The general idea is to introduce a projection function to adjust user feature distributions, such that the ideal unbiased learning objective can be upper bounded by a solvable objective purely based on the offline dataset. In the upper bound, the projected user distributions are expected to be equal given different items. From the causal inference perspective, this requirement aims to remove the causal relation from the user to the item, which enables us to achieve unbiased recommendation, bypassing the computation of IPS. To efficiently balance the user distributions upon each item pair, we propose three strategies, including clipping, sampling, and adversarial learning to improve the training process. For more robust optimization, we deploy an explicit model to capture the potential latent confounders in recommendation systems. To the best of our knowledge, this article is the first work on debiased recommendation based on confounder balancing. In the experiments, we compare our framework with many state-of-The-Art methods based on synthetic, semi-synthetic, and real-world datasets. Extensive experiments demonstrate that our model is effective in promoting the recommendation performance

    Using the Literature to Identify Confounders

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    Prior work in causal modeling has focused primarily on learning graph structures and parameters to model data generating processes from observational or experimental data, while the focus of the literature-based discovery paradigm was to identify novel therapeutic hypotheses in publicly available knowledge. The critical contribution of this dissertation is to refashion the literature-based discovery paradigm as a means to populate causal models with relevant covariates to abet causal inference. In particular, this dissertation describes a generalizable framework for mapping from causal propositions in the literature to subgraphs populated by instantiated variables that reflect observational data. The observational data are those derived from electronic health records. The purpose of causal inference is to detect adverse drug event signals. The Principle of the Common Cause is exploited as a heuristic for a defeasible practical logic. The fundamental intuition is that improbable co-occurrences can be “explained away” with reference to a common cause, or confounder. Semantic constraints in literature-based discovery can be leveraged to identify such covariates. Further, the asymmetric semantic constraints of causal propositions map directly to the topology of causal graphs as directed edges. The hypothesis is that causal models conditioned on sets of such covariates will improve upon the performance of purely statistical techniques for detecting adverse drug event signals. By improving upon previous work in purely EHR-based pharmacovigilance, these results establish the utility of this scalable approach to automated causal inference
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