Engineering data compendium. Human perception and performance. User's guide

The concept underlying the Engineering Data Compendium was the product of a research and development program (Integrated Perceptual Information for Designers project) aimed at facilitating the application of basic research findings in human performance to the design and military crew systems. The principal objective was to develop a workable strategy for: (1) identifying and distilling information of potential value to system design from the existing research literature, and (2) presenting this technical information in a way that would aid its accessibility, interpretability, and applicability by systems designers. The present four volumes of the Engineering Data Compendium represent the first implementation of this strategy. This is the first volume, the User's Guide, containing a description of the program and instructions for its use

Visual Aftereffect Of Texture Density Contingent On Color Of Frame

An aftereffect of perceived texture density contingent on the color of a surrounding region is reported. In a series of experiments, participants were adapted, with fixation, to stimuli in which the relative density of two achromatic texture regions was perfectly correlated with the color presented in a surrounding region. Following adaptation, the perceived relative density of the two regions was contingent on the color of the surrounding region or of the texture elements themselves. For example, if high density on the left was correlated with a blue surround during adaptation (and high density on the right with a yellow surround), then in order for the left and right textures to appear equal in the assessment phase, denser texture was required on the left in the presence of a blue surround (and denser texture on the right in the context of a yellow surround). Contingent aftereffects were found (1) with black-and-white scatter-dot textures, (2) with luminance-balanced textures, and (3) when the texture elements, rather than the surrounds, were colored during assessment. Effect size was decreased when the elements themselves were colored, but also when spatial subportions of the surround were used for the presentation of color. The effect may be mediated by retinal color spreading (Pöppel, 1986) and appears consistent with a local associative account of contingent aftereffects, such as Barlow\u27s (1990) model of modifiable inhibition

The contribution of luminance and chromatic channels to color assimilation

Luminance; Chromatic channelsLuminancia; Canales cromáticosLluminància; Canals cromàticsColor induction is the phenomenon where the physical and the perceived colors of an object differ owing to the color distribution and the spatial configuration of the surrounding objects. Previous works studying this phenomenon on the lsY MacLeod–Boynton color space, show that color assimilation is present only when the magnocellular pathway (i.e., the Y axis) is activated (i.e., when there are luminance differences). Concretely, the authors showed that the effect is mainly induced by the koniocellular pathway (s axis), but not by the parvocellular pathway (l axis), suggesting that when magnocellular pathway is activated it inhibits the koniocellular pathway. In the present work, we study whether parvo-, konio-, and magnocellular pathways may influence on each other through the color induction effect. Our results show that color assimilation does not depend on a chromatic–chromatic interaction, and that chromatic assimilation is driven by the interaction between luminance and chromatic channels (mainly the magno- and the koniocellular pathways). Our results also show that chromatic induction is greatly decreased when all three visual pathways are simultaneously activated, and that chromatic pathways could influence each other through the magnocellular (luminance) pathway. In addition, we observe that chromatic channels can influence the luminance channel, hence inducing a small brightness induction. All these results show that color induction is a highly complex process where interactions between the several visual pathways are yet unknown and should be studied in greater detail.Supported through the research projects: Grant PID2020-115734RB-C21 funded by MCIN/AEI/10.13039/501100011033 and Grant DPI2017-89867-C2-1-R funded by MCIN/AEI/10.13039/501100011033/ and FEDER Una manera de hacer Europa, by the Secretaria d'Universitats i Recerca del Departament d'Empresa i Coneixement de la Generalitat de Catalunya through 2017-SGR-649, and CERCA Programme/Generalitat de Catalunya

Aerospace Medicine and Biology: A continuing bibliography with indexes, supplement 127, April 1974

This special bibliography lists 279 reports, articles, and other documents introduced into the NASA scientific and technical information system in March 1974

Excitatory postsynaptic potentials in rat neocortical neurons in vitro. III. Effects of a quinoxalinedione non-NMDA receptor antagonist

1. Intracellular microelectrodes were used to obtain recordings from neurons in layer II/III of rat frontal cortex. A bipolar electrode positioned in layer IV of the neocortex was used to evoke postsynaptic potentials. Graded series of stimulation were employed to selectively activate different classes of postsynaptic responses. The sensitivity of postsynaptic potentials and iontophoretically applied neurotransmitters to the non-N-methyl-D-asparate (NMDA) antagonist 6-cyano-7-nitroquinoxaline-2,3-dione (CNQX) was examined. 2. As reported previously, low-intensity electrical stimulation of cortical layer IV evoked short-latency early excitatory postsynaptic potentials (eEPSPs) in layer II/III neurons. CNQX reversibly antagonized eEPSPs in a dose-dependent manner. Stimulation at intensities just subthreshold for activation of inhibitory postsynaptic potentials (IPSPs) produced long-latency (10 to 40-ms) EPSPs (late EPSPs or 1EPSPs). CNQX was effective in blocking 1EPSPs. 3. With the use of stimulus intensities at or just below threshold for evoking an action potential, complex synaptic potentials consisting of EPSP-IPSP sequences were observed. Both early, Cl(-)-dependent and late, K(+)-dependent IPSPs were reduced by CNQX. This effect was reversible on washing. This disinhibition could lead to enhanced excitability in the presence of CNQX. 4. Iontophoretic application of quisqualate produced a membrane depolarization with superimposed action potentials, whereas NMDA depolarized the membrane potential and evoked bursts of action potentials. At concentrations up to 5 microM, CNQX selectively antagonized quisqualate responses. NMDA responses were reduced by 10 microM CNQX. D-Serine (0.5-2 mM), an agonist at the glycine regulatory site on the NMDA receptor, reversed the CNQX depression of NMDA responses