Beckman

Budhiraja

Girgis

Hampl

Humbert

Itoh

Kaneta

Keck

Kubota

Kwan Chang Kim

Maron

McMurtry

Meininger

Miller

Min-Sun Cho

Miyauchi

Nishimura

Palmer

Taraseviciene-Stewart

Todd

Wilkins

Yamaki

Young Mi Hong

Yun Hee Lee

English

PubMed

Crossref

Korean Circulation Journal

Changes of Pulmonary Pathology and Gene Expressions After Simvastatin Treatment in the Monocrotaline-Induced Pulmonary Hypertension Rat Model

A combination of oral sildenafil and beraprost ameliorates pulmonary hypertension in rats.

All hydrophobic HMG-CoA reductase inhibitors induce apoptotic death in rat pulmonary vein endothelial cells.

Apoptotic injury in cultured human hepatocytes induced by HMG-CoA reductase inhibitors.

Attenuation of chronic hypoxic pulmonary hypertension by simvastatin.

Cellular and molecular pathobiology of pulmonary arterial hypertension.

Cellular mechanisms involved in the vascular myogenic response.

Contribution of endogenous endothelin-1 to the progression of cardiopulmonary alterations in rats with monocrotalin-induced pulmonary hypertension. Circ Res

Current perspectives on statins.

Deletion of the eNOS gene has a greater impact on the pulmonary circulation of male than female mice.

Effect of endothelin receptor blokade on monocrotaline-induced pulmonary hypertension in rats.

Endothelial dysfunction in pulmonary hypertension.

Exp Med

Gene expression of endothelin-1 and endothelin receptor A on monocrotalineinduced pulmonary hypertension in rats after bosentan treatment.

Gene expression of nitric oxide synthase and matrix metalloproteinase-2 in monocrotaline-induced pulmonary hypertension in rats after bosentan treatment.

Nitric oxide, superoxide and peroxynitrite: the good, the bad, and the ugly.

Pulmonary toxicity of monocrotaline differs at critical periods of lung development.

Regression of chronic hypoxic pulmonary hypertension by simvastatin.

Respir Crit Care Med

Role of nitric oxide in the pathogenesis of chronic pulmonary hypertension.

Simvastatin as a treatment for pulmonary hypertension trial.

Simvastatin causes endothelial cell apoptosis and attenuates severe pulmonary hypertension.

Simvastatin rescues rats from fatal pulmonary hypertension by inducing apoptosis of neointimal smooth muscle cells.

Simvastatin restores down-regulated GATA-6 expression in pulmonary hypertensive rats. Exp Lung Res

Statin therapy, alone or with rapamycin, does not reverse monocrotaline pulmonary arterial hypertension: the rapamycin-atorvastatin-simvastatin study. Am J Physiol Lung Cell Mol Physiol

The protective effect of simvastatin on monocrotaline-induced pulmonary hypertension in rats.

Vascular endothelial cells synthesize nitric oxide from L-arginine.

http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=3193043

Changes of Pulmonary Pathology and Gene Expressions After Simvastatin Treatment in the Monocrotaline-Induced Pulmonary Hypertension Rat Model

Abstract

Similar works

Full text

Available Versions

Crossref