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The Role of Type 1 and Type 2 5′-Deiodinase in the Pathophysiology of the 3,5,3′-Triiodothyronine Toxicosis of McCune-Albright Syndrome
Authors
Aaron Chidakel
Albright
+44 more
Amino
Bartha
Beth A. Brillante
Bianco
Bianco
Brtko
Canettieri
Collins
Collins
Collins
Collins
Coppotelli
Curcio-Morelli
Dentice
Feuillan
Feuillan
Francesco S. Celi
Gereben
Giuseppe Coppotelli
Jakobs
Kim
Kim
Laurberg
Laurberg
Leonard
Lumbroso
Maia
Marilyn Kelly
Mastorakos
McCune
Michael T. Collins
Michiels
Murakami
Natasha Cherman
Penelope P. Feuillan
Ringel
Salvatore
Schwindinger
Segni
St. Germain
Thomas Shawker
Visser
Weetman
Weinstein
Publication date
Publisher
The Endocrine Society
Doi
Cite
View
on
PubMed
Abstract
Context: McCune-Albright syndrome (MAS) is caused by mutations in GNAS (most often R201C or R201H) leading to constitutive cAMP signaling and multiple endocrine dysfunctions, including morphological and functional thyroid involvement
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Last time updated on 11/12/2019