Tunneling Qubit Operation on a Protected Josephson Junction Array

We discuss a protected quantum computation process based on a hexagon Josephson junction array. Qubits are encoded in the punctured array, which is topologically protected. The degeneracy is related to the number of holes. The topological degeneracy is lightly shifted by tuning the flux through specific hexagons. We also show how to perform single qubit operation and basic quantum gate operations in this system.Comment: 8 pages, 4 figures. The published version in Phys. Rev., A81(2010)01232

5-({[(E)-Benzyl­idene­amino]­oxy}meth­yl)-1,3,4-thia­diazol-2-amine

In the mol­ecule of the title compound, C10H10N4OS, the configuration about the C=N double bond is E. The dihedral angle between the thia­diazole and benzene rings is 81.1 (1)°. In the crystal, mol­ecules are linked by N—H⋯N and C—H⋯O hydrogen bonds to form a two-dimensional network parallel with the bc plane

Quantum correlations in a cluster-like system

We discuss a cluster-like 1D system with triplet interaction. We study the topological properties of this system. We find that the degeneracy depends on the topology of the system, and well protected against external local perturbations. All these facts show that the system is topologically ordered. We also find a string order parameter to characterize the quantum phase transition. Besides, we investigate two-site correlations including entanglement, quantum discord and mutual information. We study the different divergency behaviour of the correlations. The quantum correlation decays exponentially in both topological and magnetic phases, and diverges in reversed power law at the critical point. And we find that in TQPT systems, the global difference of topology induced by dimension can be reflected in local quantum correlations.Comment: 7 pages, 6 figure

(E)-(2-Chloro­benzyl­idene)amino 2-amino-4-chloro­benzoate

In the title compound, C14H10Cl2N2O2, the configuration about the C=N double bond is E and the dihedral angle between the benzene rings is 1.75 (5)°. An intra­molecular N—H⋯O inter­action generates an S(6) ring. In the crystal, mol­ecules are linked by N—H⋯O hydrogen bonds, resulting in [101] chains

2-(1H-Imidazo[4,5-f][1,10]phenanthrolin-2-yl)phenol monohydrate

The asymmetric unit of the title compound, C19H12N4O·H2O, contains one organic molecule and one solvent water mol­ecule, which are connected by N—H⋯O and O—H⋯N hydrogen bonds. In addition, there is one intra­molecular O—H⋯N hydrogen bond. The organic mol­ecule is essentially planar (r.m.s. deviation for all non-H atoms = 0.028 Å)

1-(But-2-enyl­idene)-2-(2-nitro­phen­yl)hydrazine

The mol­ecule of the title Schiff base compound, C10H11N3O2, adopts an E geometry with respect to the C=N double bond. The mol­ecule is roughly planar, with the largest deviation from the mean plane being 0.111 (2) Å, The enyl­idene-hydrazine group is, however, slightly twisted with respect to the phenyl ring, making a dihedral angle of 6.5 (3)°. An intra­molecular N—H⋯O hydrogen bond may be responsible for the planar conformation. An inter­molecular N—H⋯O hydrogen bond links two mol­ecules around an inversion center, building a pseudo dimer

2-Eth­oxy-4-{[(2-nitro­phen­yl)hydrazono]meth­yl}phenol

The title compound, C15H15N3O4, a Schiff base, was obtained from a condensation reaction of 3-eth­oxy-4-hydroxy­benzaldehyde and 2-nitro­phenyl­hydrazine. The mol­ecule is approximately planar, the largest deviation from the mean plane being 0.1449 (16) Å. An intramolecular N—H⋯O inter­action is also present. In the crystal, inter­molecular O—H⋯O hydrogen bonds link the mol­ecules, forming chain parallel to the b axis

Tramadol Pretreatment Enhances Ketamine-Induced Antidepressant Effects and Increases Mammalian Target of Rapamycin in Rat Hippocampus and Prefrontal Cortex

Several lines of evidence have demonstrated that acute administration of ketamine elicits fast-acting antidepressant effects. Moreover, tramadol also has potential antidepressant effects. The aim of this study was to investigate the effects of pretreatment with tramadol on ketamine-induced antidepressant activity and was to determine the expression of mammalian target of rapamycin (mTOR) in rat hippocampus and prefrontal cortex. Rats were intraperitoneally administrated with ketamine at the dose of 10 mg/kg or saline 1 h before the second episode of the forced swimming test (FST). Tramadol or saline was intraperitoneally pretreated 30 min before the former administration of ketamine or saline. The locomotor activity and the immobility time of FST were both measured. After that, rats were sacrificed to determine the expression of mTOR in hippocampus and prefrontal cortex. Tramadol at the dose of 5 mg/kg administrated alone did not elicit the antidepressant effects. More importantly, pretreatment with tramadol enhanced the ketamine-induced antidepressant effects and upregulated the expression of mTOR in rat hippocampus and prefrontal cortex. Pretreatment with tramadol enhances the ketamine-induced antidepressant effects, which is associated with the increased expression of mTOR in rat hippocampus and prefrontal cortex

1-(2-Furylmethyl­ene)-2-(2-nitro­phen­yl)hydrazine

The title Schiff base compound, C11H9N3O3, was obtained from a condensation reaction of furan-2-carbaldehyde and 2-nitro­phenyl­hydrazine. The mol­ecule is roughly planar, the largest deviation from the mean plane defined by all non-H atoms being 0.097 (4). An in ntra­molecular N—H⋯O hydrogen bond might influence the planar conformation of the mol­ecule. In the crystal, weak C—H⋯O hydrogen bonds link the mol­ecules, forming a chain