20 research outputs found
Surgical management of foreign body obstruction of the small and large colons in 29 equids (2004–2016)
Standing flank laparotomy for the treatment of small colon impactions in 15 ponies and one horse
Continuous fluid infusion per rectum compared with intravenous and nasogastric fluid administration in horses
Alterações morfológicas de tecido laminar do casco e parâmetros clínicos e laboratoriais de equinos com síndrome cólica letal Morphologic alterations of the hoof lamellar tissue, and clinic and laboratorial analyses of horses with lethal colic syndrome
As afecções gastrintestinais dos cavalos são agravadas por complicações como a laminite, cuja etiopatogenia está relacionada à degradação da membrana basal do tecido laminar por metaloproteinases (MMPs). A ativação das MMPs pode ocorrer devido à liberação local de citocinas inflamatórias ou enzimas provenientes de leucócitos infiltrados no tecido laminar. O objetivo deste trabalho foi avaliar as alterações morfológicas do tecido laminar de equinos com síndrome cólica letal e sua provável associação com parâmetros clínicos e laboratoriais. Observou-se intensa destruição da arquitetura laminar, principalmente nos animais com alterações físicas e laboratoriais mais acentuadas, como tempo de preenchimento capilar prolongado (TPC), membranas mucosas congestas, taquicardia, hemoconcentração e redução nas contagens de plaquetas e leucócitos. Os resultados sinalizam o provável momento do desenvolvimento de lesões do tecido laminar em equinos com síndrome cólica, no qual é possível adotar medidas preventivas contra a laminite.<br>The gastrointestinal diseases of horses are aggravated by complications such as laminitis. The laminitis etiopathogeny are connected with lamellar basement membrane degradation by matrix metalloproteinases (MMPs). Inflammatory cytokines and leukocytes enzymes can active MMPs. The object of this study was to evaluate morphological changes on lamellar tissue of horses with colic syndrome and its association with clinical and laboratorial parameters. It was observed intensive destruction of lamellar architecture, mainly on animals with severe physical and laboratorial alterations, such as delayed capillary refill time, congested mucous membrane, tachycardia, hemoconcentration and low count of platelet and leukocytes. The results sign to the most likely moment of development of lamellar tissue injuries in horses with colic syndrome, which can be adopted preventive measures against laminitis
GALEX AND PAN-STARRS1 DISCOVERY OF SN IIP 2010aq: THE FIRST FEW DAYS AFTER SHOCK BREAKOUT IN A RED SUPERGIANT STAR
We present the early UV and optical light curve of Type IIP supernova (SN)
2010aq at z=0.0862, and compare it to analytical models for thermal emission
following SN shock breakout in a red supergiant star. SN 2010aq was discovered
in joint monitoring between the Galaxy Evolution Explorer (GALEX) Time Domain
Survey (TDS) in the NUV and the Pan-STARRS1 Medium Deep Survey (PS1 MDS) in the
g, r, i, and z bands. The GALEX and Pan-STARRS1 observations detect the SN less
than 1 day after shock breakout, measure a diluted blackbody temperature of
31,000 +/- 6,000 K 1 day later, and follow the rise in the UV/optical light
curve over the next 2 days caused by the expansion and cooling of the SN
ejecta. The high signal-to-noise ratio of the simultaneous UV and optical
photometry allows us to fit for a progenitor star radius of 700 +/- 200 R_sun,
the size of a red supergiant star. An excess in UV emission two weeks after
shock breakout compared to SNe well fitted by model atmosphere-code synthetic
spectra with solar metallicity, is best explained by suppressed line blanketing
due to a lower metallicity progenitor star in SN 2010aq. Continued monitoring
of PS1 MDS fields by the GALEX TDS will increase the sample of early UV
detections of Type II SNe by an order of magnitude, and probe the diversity of
SN progenitor star properties.Comment: 4 pages, 3 color figures. Accepted for publication in ApJ Letters,
corrections from proofs adde