Koreferat zu Kirstein/Schäfer - Erzeugt das Verbrauchsgüterkaufrecht Marktversagen?

Das Referat konzentriert sich auf die Frage, ob das Verbrauchsgüterkaufrecht europäischer Provenienz Marktversagen erzeugt. Dabei kommt es zu einem Ergebnis, welches der bisherigen Einschätzung und Bewertung eher widerspricht: Signalling ist weiterhin möglich, und im Ergebnis wird kein Marktversagen bewirkt. Dieser Befund mag für Ökonomen überraschend sein, denn sie stehen der Verbrauchsgüterkaufrichtlinie im Grundansatz skeptisch gegenüber. Unausgesprochen zugrundegelegt sind solcher Skepsis liberale Vorstellungen vom Marktgeschehen, welche die Privatautonomie über alles stellen und Eingriffe in die Privatautonomie von vornherein mit Misstrauen betrachten. Der Befund ist dagegen aus juristischer Sicht nicht kontraintuitiv. Man konnte mit ihm rechnen. Die Realität ist der beste Beleg: Die Märkte für B2C-Verkäufe sind nicht zusammengebrochen. Sie existieren weiter. Dies ist auch kaum verwunderlich. Denn Konsumenten haben weiterhin Bedarf, und Anbieter bemühen sich weiterhin, diesen Bedarf zu decken.

Galvanic corrosion of aluminium–copper model alloys

Galvanic coupling between different α and θ phase-containing model Al–Cu alloys, deposited by magnetron sputtering, has revealed that the anodic α phase did not suffer corrosion and remained in the passive state in sulphate solution. Conversely, sulphate ions induced pitting of the cathodic θ phase. Pitting susceptibility of the cathode increased when the difference between the copper content of the anode and cathode increased. Similar observations were made for all the galvanic couples; further, the higher the copper content of a phase, then the greater its susceptibility to pitting

Electrochemical Behavior of Magnetron-Sputtered Al–Cu Alloy Films in Sulfate Solutions

Model alloys, generated by magnetron sputtering, have been employed to understand the role of copper on the corrosion behavior of aluminum alloys. Binary Al–Cu alloys, with copper contents between 0 and 100 atom %, were synthesized with well-controlled compositions, embracing single-phase alpha and theta alloys together with multiphase alloys. Electrochemical measurements confirmed the stability of the thin alloy films and revealed that the corrosion behavior of the alpha, theta, and eta2 phases differed strongly in the cathodic region. Further, in the anodic region, phases of high copper content suffered pitting in sulfate solutions, while the alpha phase remained passive

Natural Host Genetic Resistance to Lentiviral CNS Disease: A Neuroprotective MHC Class I Allele in SIV-Infected Macaques

Human immunodeficiency virus (HIV) infection frequently causes neurologic disease even with anti-retroviral treatment. Although associations between MHC class I alleles and acquired immunodeficiency syndrome (AIDS) have been reported, the role MHC class I alleles play in restricting development of HIV-induced organ-specific diseases, including neurologic disease, has not been characterized. This study examined the relationship between expression of the MHC class I allele Mane-A*10 and development of lentiviral-induced central nervous system (CNS) disease using a well-characterized simian immunodeficiency (SIV)/pigtailed macaque model. The risk of developing CNS disease (SIV encephalitis) was 2.5 times higher for animals that did not express the MHC class I allele Mane-A*10 (P = 0.002; RR = 2.5). Animals expressing the Mane-A*10 allele had significantly lower amounts of activated macrophages, SIV RNA, and neuronal dysfunction in the CNS than Mane-A*10 negative animals (P<0.001). Mane-A*10 positive animals with the highest CNS viral burdens contained SIV gag escape mutants at the Mane-A*10-restricted KP9 epitope in the CNS whereas wild type KP9 sequences dominated in the brain of Mane-A*10 negative animals with comparable CNS viral burdens. These concordant findings demonstrate that particular MHC class I alleles play major neuroprotective roles in lentiviral-induced CNS disease