The continuity of normal velocity at hte matching interface for and .

<p>Left: Real part, Right: Imaginary part.</p

The continuity of pressure at the matching interface for and .

<p>Left: Real part, Right: Imaginary part.</p

Power balance versus non-dimensional height for fundamental mode incidence.

<p>Left: Clamped edge conditions, Right: Pin-jointed edge conditions.</p

Power balance versus frequency for fundamental mode incidence.

<p>Left: Clamped edge conditions, Right: Pin-jointed edge conditions.</p

Non-dimensional geometry of wave-guide.

<p>Non-dimensional geometry of wave-guide.</p

Table_1_NADH-Mediated Gene Expression in Streptococcus pneumoniae and Role of Rex as a Transcriptional Repressor of the Rex-Regulon.PDF

<p>Nicotinamide adenine dinucleotides (NAD(H)) play a vital role in various biological processes, including keeping the cellular redox balance. In this study, we investigate the regulatory responses of Streptococcus pneumoniae D39 to NADH and characterize the role of the Rex protein as a transcriptional repressor of the gapN, fba, pncB, adhB2, gap, and adhE genes. Transcriptomic analysis was used to observe the response of S. pneumoniae D39 to NADH. Our microarray studies revealed elevated expression of various genes/operons involved in transport and biosynthesis of niacin (gapN, fba, pncB, adhB2, gap, and adhE). Promoter lacZ-fusion assays and microarray studies with the rex mutant revealed the role of Rex as a transcriptional repressor of gapN, fba, pncB, adhB2, gap, and adhE involved in niacin uptake and biosynthesis, in the presence of NADH. We predict the operator site (5′-TTGTKAWAAWWTTCACAA-3′) of Rex in the regulatory regions of Rex-regulated genes that was subsequently validated by promoter mutational experiments.</p

Power balance versus frequency for secondary mode incidence.

<p>Left: Clamped edge conditions, Right: Pin-jointed edge conditions.</p

Power balance versus non-dimensional height for secondary mode incidence.

<p>Left: Clamped edge conditions, Right: Pin-jointed edge conditions.</p

Table_2_NADH-Mediated Gene Expression in Streptococcus pneumoniae and Role of Rex as a Transcriptional Repressor of the Rex-Regulon.PDF

<p>Nicotinamide adenine dinucleotides (NAD(H)) play a vital role in various biological processes, including keeping the cellular redox balance. In this study, we investigate the regulatory responses of Streptococcus pneumoniae D39 to NADH and characterize the role of the Rex protein as a transcriptional repressor of the gapN, fba, pncB, adhB2, gap, and adhE genes. Transcriptomic analysis was used to observe the response of S. pneumoniae D39 to NADH. Our microarray studies revealed elevated expression of various genes/operons involved in transport and biosynthesis of niacin (gapN, fba, pncB, adhB2, gap, and adhE). Promoter lacZ-fusion assays and microarray studies with the rex mutant revealed the role of Rex as a transcriptional repressor of gapN, fba, pncB, adhB2, gap, and adhE involved in niacin uptake and biosynthesis, in the presence of NADH. We predict the operator site (5′-TTGTKAWAAWWTTCACAA-3′) of Rex in the regulatory regions of Rex-regulated genes that was subsequently validated by promoter mutational experiments.</p

List of strains and plasmids used in this study.

<p>List of strains and plasmids used in this study.</p