Supplementary Material, DS_10.1177_0022034518775036 – NLRP6 Induces Pyroptosis by Activation of Caspase-1 in Gingival Fibroblasts

<p>Supplementary Material, DS_10.1177_0022034518775036 for NLRP6 Induces Pyroptosis by Activation of Caspase-1 in Gingival Fibroblasts by W. Liu, J. Liu, W. Wang, Y. Wang and X. Ouyang in Journal of Dental Research</p

Legislative Documents

Also, variously referred to as: Senate bills; Senate documents; Senate legislative documents; legislative documents; and General Court documents

Additional file 3: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Figure S6. Phylogenetic trees and evolutionary profile of GRAS genes in green plants. (A) Phylogenetic tree showing the evolutionary relationship between plants (marked with different colors) GRASs. Tree topology is a consensus from NJ, UPGMA. Bootstrap values from the original trees higher than 50% are shown (NJ/UPGMA). (B) Evolutionary profile of GRAS subfamilyes’ genes in green plants. (TIF 1487 kb

Additional file 10: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Table S5. The expression data of qRT-PCR. (XLS 36 kb

Additional file 4: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Figure S2. Chromosomal locations of GRAS members in G. hirsutum L.. (TIF 2000 kb

Additional file 11: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Table S3. The public available expression profile of GRAS members. (XLS 123 kb

Additional file 6: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Figure S4. Motifs describution of GRAS members in G. hirsutum L.. development stages in G. hirsutum L.. (TIF 6550 kb

Additional file 2: of Genome-wide analysis of GRAS transcription factor gene family in Gossypium hirsutum L.

Figure S1. Phylogenetic tree built with the UPGMA method. (TIF 8356 kb

Integrated analysis of lncRNAs and mRNA expression profiles reveals the potential role of lncRNAs in different bovine lactation stages

<div>Long non-coding RNAs (lncRNAs) play a critical role in mammary development and breast cancer biology. Despite their important role in the mammary gland, little is known on the roles of lncRNAs in bovine lactation, particularly regarding the molecular processes underlying it. To characterize the role of lncRNAs in bovine lactation, four samples of Holstein cow mammary gland tissue at peak and late lactation stages were examined after biopsy. We then profiled the transcriptome of mammary gland using RNA sequencing technology. Further, functional lncRNA-mRNA co-expression pairs were constructed to infer the function of lncRNAs using a generalized linear model, followed by Gene Ontology (GO) and Kyoto Encyclopedia of Genes and Genomes (KEGG) pathway analyses. Over 1000 putative lncRNAs were identified, 117 of which were differentially expressed between peak and late lactation stages. Bovine lncRNAs were shorter, with fewer exon numbers, and expressed at significantly lower levels than protein-coding genes. Seventy-two differentially expressed lncRNAs (DE-lncRNAs) were co-expressed with 340 different protein-coding genes. The KEGG pathway analysis showed that target mRNAs for DE-lncRNAs were mainly related to lipid and glucose metabolism, including the peroxisome proliferator-activated receptors (PPAR) and 5' adenosine monophosphate-activated protein kinase (AMPK) signaling pathways. Further bioinformatics and integrative analyses revealed that 12 DE-lncRNAs potentially played important roles in bovine lactation. Our findings provide a valuable resource for future bovine transcriptome studies, facilitate the understanding of bovine lactation biology, and offer functional information for cattle lactation.</div><div>Keywords: Long non-coding RNA, bovine lactation, mammary gland, co-expression, RNA sequencing</div

DS_10.1177_0022034518755688 – Supplemental material for Smad6 Methylation Represses NFκB Activation and Periodontal Inflammation

<p>Supplemental material, DS_10.1177_0022034518755688 for Smad6 Methylation Represses NFκB Activation and Periodontal Inflammation by T. Zhang, J. Wu, N. Ungvijanpunya, O. Jackson-Weaver, Y. Gou, J. Feng, T.V. Ho, Y. Shen, J. Liu, S. Richard, J. Jin, G. Hajishengallis, Y. Chai, and J. Xu in Journal of Dental Research</p