56 research outputs found

    CVRecon: Rethinking 3D Geometric Feature Learning For Neural Reconstruction

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    Recent advances in neural reconstruction using posed image sequences have made remarkable progress. However, due to the lack of depth information, existing volumetric-based techniques simply duplicate 2D image features of the object surface along the entire camera ray. We contend this duplication introduces noise in empty and occluded spaces, posing challenges for producing high-quality 3D geometry. Drawing inspiration from traditional multi-view stereo methods, we propose an end-to-end 3D neural reconstruction framework CVRecon, designed to exploit the rich geometric embedding in the cost volumes to facilitate 3D geometric feature learning. Furthermore, we present Ray-contextual Compensated Cost Volume (RCCV), a novel 3D geometric feature representation that encodes view-dependent information with improved integrity and robustness. Through comprehensive experiments, we demonstrate that our approach significantly improves the reconstruction quality in various metrics and recovers clear fine details of the 3D geometries. Our extensive ablation studies provide insights into the development of effective 3D geometric feature learning schemes. Project page: https://cvrecon.ziyue.cool

    Covariance-Based Activity Detection in Cooperative Multi-Cell Massive MIMO: Scaling Law and Efficient Algorithms

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    This paper focuses on the covariance-based activity detection problem in a multi-cell massive multiple-input multiple-output (MIMO) system. In this system, active devices transmit their signature sequences to multiple base stations (BSs), and the BSs cooperatively detect the active devices based on the received signals. While the scaling law for the covariance-based activity detection in the single-cell scenario has been extensively analyzed in the literature, this paper aims to analyze the scaling law for the covariance-based activity detection in the multi-cell massive MIMO system. Specifically, this paper demonstrates a quadratic scaling law in the multi-cell system, under the assumption that the exponent in the classical path-loss model is greater than 2. This finding shows that, in the multi-cell MIMO system, the maximum number of active devices that can be detected correctly in each cell increases quadratically with the length of the signature sequence and decreases logarithmically with the number of cells (as the number of antennas tends to infinity). Moreover, in addition to analyzing the scaling law for the signature sequences randomly and uniformly distributed on a sphere, the paper also establishes the scaling law for signature sequences generated from a finite alphabet, which are easier to generate and store. Moreover, this paper proposes two efficient accelerated coordinate descent (CD) algorithms with a convergence guarantee for solving the device activity detection problem. The first algorithm reduces the complexity of CD by using an inexact coordinate update strategy. The second algorithm avoids unnecessary computations of CD by using an active set selection strategy. Simulation results show that the proposed algorithms exhibit excellent performance in terms of computational efficiency and detection error probability.Comment: 54 pages, 11 figures, submitted for possible publicatio

    Disentangling Object Motion and Occlusion for Unsupervised Multi-frame Monocular Depth

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    Conventional self-supervised monocular depth prediction methods are based on a static environment assumption, which leads to accuracy degradation in dynamic scenes due to the mismatch and occlusion problems introduced by object motions. Existing dynamic-object-focused methods only partially solved the mismatch problem at the training loss level. In this paper, we accordingly propose a novel multi-frame monocular depth prediction method to solve these problems at both the prediction and supervision loss levels. Our method, called DynamicDepth, is a new framework trained via a self-supervised cycle consistent learning scheme. A Dynamic Object Motion Disentanglement (DOMD) module is proposed to disentangle object motions to solve the mismatch problem. Moreover, novel occlusion-aware Cost Volume and Re-projection Loss are designed to alleviate the occlusion effects of object motions. Extensive analyses and experiments on the Cityscapes and KITTI datasets show that our method significantly outperforms the state-of-the-art monocular depth prediction methods, especially in the areas of dynamic objects. Our code will be made publicly available

    Device Activity Detection in mMTC with Low-Resolution ADC: A New Protocol

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    This paper investigates the effect of low-resolution analog-to-digital converters (ADCs) on device activity detection in massive machine-type communications (mMTC). The low-resolution ADCs induce two challenges on the device activity detection compared with the traditional setup with assumption of infinite ADC resolution. First, the codebook design for signal quantization by the low-resolution ADCs is particularly important since a good codebook design can lead to small quantization error on the received signal, which in turn has significant influence on the activity detector performance. To this end, prior information about the received signal power is needed, which depends on the number of active devices KK. This is sharply different from the activity detection problem in traditional setups, in which the knowledge of KK is not required by the BS as a prerequisite. Second, the covariance-based approach achieves good activity detection performance in traditional setups while it is not clear if it can still achieve good performance in this paper. To solve the above challenges, we propose a communication protocol that consists of an estimator for KK and a detector for active device identities: 1) For the estimator, the technical difficulty is that the design of the ADC quantizer and the estimation of KK are closely intertwined and doing one needs the information/execution from the other. We propose a progressive estimator which iteratively performs the estimation of KK and the design of the ADC quantizer; 2) For the activity detector, we propose a custom-designed stochastic gradient descent algorithm to estimate the active device identities. Numerical results demonstrate the effectiveness of the communication protocol.Comment: Submitted to IEEE for possible publicatio

    Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages

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    Although Fas (CD95) is recognized as a death receptor that induces apoptosis, recent studies indicate that the Fas/FasL system can induce pro-inflammatory cytokine production by macrophages independent of conventional caspase-mediated apoptotic signaling. The precise mechanism(s) by which Fas activates macrophage inflammation is unknown. We hypothesized that Fas stimulates rapid release of high mobility group box 1 (HMGB1) that acts in an autocrine and/or paracrine manner to stimulate pro-inflammatory cytokine production via a Toll-like receptor-4 (TLR4)/Interleukin-1 receptor associated kinase-4 (IRAK4)-dependent mechanism. Following Fas activation, HMGB1 was released within 1 hr from viable RAW267.4 cells and primary murine peritoneal macrophages. HMGB1 release was more rapid following Fas activation compared to LPS stimulation. Neutralization of HMGB1 with an inhibitory anti-HMGB1 monoclonal antibody strongly inhibited Fas-induced production of tumor necrosis factor (TNF) and macrophage inflammatory protein-2 (MIP-2). Both Fas-induced HMGB1 release and associated pro-inflammatory cytokine production were significantly decreased from Tlr4-/- and Irak4-/- macrophages, but not Tlr2-/- macrophages. These findings reveal a novel mechanism underlying Fas-mediated pro-inflammatory physiological responses in macrophages. We conclude that Fas activation induces rapid, TLR4/IRAK4-dependent release of HMGB1 that contributes to Fas-mediated pro-inflammatory cytokine production by viable macrophages

    DUA-DA: Distillation-based Unbiased Alignment for Domain Adaptive Object Detection

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    Though feature-alignment based Domain Adaptive Object Detection (DAOD) have achieved remarkable progress, they ignore the source bias issue, i.e. the aligned features are more favorable towards the source domain, leading to a sub-optimal adaptation. Furthermore, the presence of domain shift between the source and target domains exacerbates the problem of inconsistent classification and localization in general detection pipelines. To overcome these challenges, we propose a novel Distillation-based Unbiased Alignment (DUA) framework for DAOD, which can distill the source features towards a more balanced position via a pre-trained teacher model during the training process, alleviating the problem of source bias effectively. In addition, we design a Target-Relevant Object Localization Network (TROLN), which can mine target-related knowledge to produce two classification-free metrics (IoU and centerness). Accordingly, we implement a Domain-aware Consistency Enhancing (DCE) strategy that utilizes these two metrics to further refine classification confidences, achieving a harmonization between classification and localization in cross-domain scenarios. Extensive experiments have been conducted to manifest the effectiveness of this method, which consistently improves the strong baseline by large margins, outperforming existing alignment-based works.Comment: 10pages,5 figure

    Stereo-NEC: Enhancing Stereo Visual-Inertial SLAM Initialization with Normal Epipolar Constraints

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    We propose an accurate and robust initialization approach for stereo visual-inertial SLAM systems. Unlike the current state-of-the-art method, which heavily relies on the accuracy of a pure visual SLAM system to estimate inertial variables without updating camera poses, potentially compromising accuracy and robustness, our approach offers a different solution. We realize the crucial impact of precise gyroscope bias estimation on rotation accuracy. This, in turn, affects trajectory accuracy due to the accumulation of translation errors. To address this, we first independently estimate the gyroscope bias and use it to formulate a maximum a posteriori problem for further refinement. After this refinement, we proceed to update the rotation estimation by performing IMU integration with gyroscope bias removed from gyroscope measurements. We then leverage robust and accurate rotation estimates to enhance translation estimation via 3-DoF bundle adjustment. Moreover, we introduce a novel approach for determining the success of the initialization by evaluating the residual of the normal epipolar constraint. Extensive evaluations on the EuRoC dataset illustrate that our method excels in accuracy and robustness. It outperforms ORB-SLAM3, the current leading stereo visual-inertial initialization method, in terms of absolute trajectory error and relative rotation error, while maintaining competitive computational speed. Notably, even with 5 keyframes for initialization, our method consistently surpasses the state-of-the-art approach using 10 keyframes in rotation accuracy

    PlanarNeRF: Online Learning of Planar Primitives with Neural Radiance Fields

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    Identifying spatially complete planar primitives from visual data is a crucial task in computer vision. Prior methods are largely restricted to either 2D segment recovery or simplifying 3D structures, even with extensive plane annotations. We present PlanarNeRF, a novel framework capable of detecting dense 3D planes through online learning. Drawing upon the neural field representation, PlanarNeRF brings three major contributions. First, it enhances 3D plane detection with concurrent appearance and geometry knowledge. Second, a lightweight plane fitting module is proposed to estimate plane parameters. Third, a novel global memory bank structure with an update mechanism is introduced, ensuring consistent cross-frame correspondence. The flexible architecture of PlanarNeRF allows it to function in both 2D-supervised and self-supervised solutions, in each of which it can effectively learn from sparse training signals, significantly improving training efficiency. Through extensive experiments, we demonstrate the effectiveness of PlanarNeRF in various scenarios and remarkable improvement over existing works

    A novel GLP-1/GIP dual agonist is more effective than liraglutide in reducing inflammation and enhancing GDNF release in the MPTP mouse model of Parkinson's disease

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    Type 2 diabetes mellitus (T2DM) is one of the risk factors for Parkinson's disease (PD). Insulin desensitisation has been observed in the brains of patients, which may promote neurodegeneration. Incretins are a family of growth factors that can re-sensitise insulin signalling. We have previously shown that mimetics of glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) have neuroprotective effects in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropypridine (MPTP) mouse model of PD. Recently, dual GLP-1/GIP receptor agonists have been developed. We therefore tested the novel dual agonist DA3-CH in comparison with the best GLP-1 analogue currently on the market, liraglutide (both drugs 25nmol/kg ip once-daily for 7 days) in the MPTP mouse model of PD (25 mg/kg ip once-daily for 7 days). In the Rotarod and grip strength assessment, DA3-CH was superior to liraglutide in reversing the MPTP–induced motor impairment. Dopamine synthesis as indicated by levels of tyrosine hydroxylase was much reduced by MPTP in the substantia nigra and striatum, and DA3-CH reversed this while liragutide only partially reversed this. The chronic inflammation response as shown in increased levels of activated microglia and astrocytes was reduced by both drugs. Importantly, expression levels of the neuroprotective growth factor Glial Derived Neurotrophic Factor (GDNF) was much enhanced by both DA3-CH and liragutide. The results demonstrate that the combination of GLP-1 and GIP receptor activation is superior to single GLP-1 receptor activation alone. Therefore, new dual agonists may be a promising treatment for PD. The GLP-1 receptor agonist exendin-4 has already shown disease modifying effects in clinical trials in PD patients
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