Fringe platforms: An analysis of contesting alternatives to the mainstream social media platforms in a platformized public sphere

Social media companies are ubiquitous in our social lives and public debate. They provide spaces for discussion and grant us access to journalism. In his 1962 Strukturwandel der Öffentlichkeit, Jürgen Habermas described how the public sphere was transformed through the introduction of modern communication systems. With the advent of social media platforms, the public sphere has transformed again through ‘platformization’. Platformization is the process by which Big Tech companies infiltrate infrastructures, economic processes and governmental frameworks of entire public sectors, structuring them around their own practices and logics. This dissertation studies the contemporary platformized public sphere, not by focusing at the center of the public sphere, but by looking at the edges of the platform ecology, where radical or counter platform technology are situated. I do this through the concept of ‘fringe platforms’, which are defined as; alternative platform services that are established as an explicit critique of the ideological premises and practices of mainstream platform services, which strive to cause a shift in the norms of the platform ecology they contest by offering an ideologically different technology. One such platform is alt-right microblogging service Gab.com, which was subjected to a process of 'deplatformization' in 2018, when its user base was implicated in white supremacist terrorism. Deplatformization refers to tech companies’ efforts to reduce toxic content by pushing back controversial platforms and their communities to the edges of the ecosystem by denying them access to the basic infrastructural services required to function online. By studying Gab through three case studies this dissertation poses the following research questions: What is the role of fringe social media platforms in a platformized public sphere? What hierarchies and shifts in power do they signify? And how can they inform us about the platform ecosystem? In the first case study, I explore Gab as an ecosystem, and conclude that the study of fringe platforms entails a more explicit role in the analyses for a platform’s self-positioning and narrative, as well as a shift in focus from a platform as an ecosystem towards a lens that takes into account the (infra)structural consequences of a platform as part of an ecosystem of services. In the second and third case study, I oblige to this conclusion and examine Gab as part of the platform ecosystem, shifting the analytical lens to the power dynamics and infrastructures of the platformized public sphere. There, I conclude that deplatformization demonstrates how the power and influence of private technology platforms reaches far beyond their own boundaries, which reveals platform power as infrastructural and rule-setting power. In the conclusion chapter, I argue that the aforementioned fringe lens is useful, not only for the analysis of fringe platforms, but also for the platformized public sphere as a whole, as it makes the structures and infrastructures of the platformized public sphere visible; highlights power and discourse; focuses on dynamics, conflict and breakdown; and incorporates the dominant and democratically productive as well as the marginal and illiberal, in its analyses

Dynamical propagation and Roe algebras of warped spaces

Given a non-singular action $\Gamma \curvearrowright (X,\mu)$, we define the $*$-algebra $\mathbb C_{\rm fp}[\Gamma \curvearrowright X]$ of operators of finite dynamical propagation associated with this action. This assignment is completely canonical and only depends on the measure class of $\mu$. We prove that the algebraic crossed product $L^{\infty}X \rtimes_{\rm alg} \Gamma$ surjects onto $\mathbb C_{\rm fp}[\Gamma \curvearrowright X]$ and that this surjection is a $\ast$-isomorphism whenever the action is essentially free. As a consequence, we canonically characterize ergodicity and strong ergodicity of the action in terms of structural properties of $\mathbb C_{\rm fp}[\Gamma \curvearrowright X]$ and its closure. We also use these techniques to describe the Roe algebra of a warped space in terms of the Roe algebra of the (non-warped) space and the group action. We apply this result to Roe algebras of warped cones.Comment: 26 page

Deplatformization and the governance of the platform ecosystem

This article analyzes deplatformization as an implied governance strategy by major tech companies to detoxify the platform ecosystem of radical content while consolidating their power as designers, operators, and governors of that same ecosystem. Deplatformization is different from deplatforming: it entails a systemic effort to push back encroaching radical right-wing platforms to the fringes of the ecosystem by denying them the infrastructural services needed to function online. We identify several deplatformization strategies, using Gab as an example of a platform that survived its relegation and which subsequently tried to build an alternative at the edge of the mainstream ecosystem. Evaluating deplatformization in terms of governance, the question that arises is who is responsible for cleansing the ecosystem: corporations, states, civil society actors, or all three combined? Understanding the implied governance of deplatformization is imperative to assess the higher stakes in future debates concerning Internet governability

The Importance of Human FcγRI in Mediating Protection to Malaria

The success of passive immunization suggests that antibody-based therapies will be effective at controlling malaria. We describe the development of fully human antibodies specific for Plasmodium falciparum by antibody repertoire cloning from phage display libraries generated from immune Gambian adults. Although these novel reagents bind with strong affinity to malaria parasites, it remains unclear if in vitro assays are predictive of functional immunity in humans, due to the lack of suitable animal models permissive for P. falciparum. A potentially useful solution described herein allows the antimalarial efficacy of human antibodies to be determined using rodent malaria parasites transgenic for P. falciparum antigens in mice also transgenic for human Fc-receptors. These human IgG1s cured animals of an otherwise lethal malaria infection, and protection was crucially dependent on human FcγRI. This important finding documents the capacity of FcγRI to mediate potent antimalaria immunity and supports the development of FcγRI-directed therapy for human malaria

Mapping genomic loci implicates genes and synaptic biology in schizophrenia

Schizophrenia has a heritability of 60-80%1, much of which is attributable to common risk alleles. Here, in a two-stage genome-wide association study of up to 76,755 individuals with schizophrenia and 243,649 control individuals, we report common variant associations at 287 distinct genomic loci. Associations were concentrated in genes that are expressed in excitatory and inhibitory neurons of the central nervous system, but not in other tissues or cell types. Using fine-mapping and functional genomic data, we identify 120 genes (106 protein-coding) that are likely to underpin associations at some of these loci, including 16 genes with credible causal non-synonymous or untranslated region variation. We also implicate fundamental processes related to neuronal function, including synaptic organization, differentiation and transmission. Fine-mapped candidates were enriched for genes associated with rare disruptive coding variants in people with schizophrenia, including the glutamate receptor subunit GRIN2A and transcription factor SP4, and were also enriched for genes implicated by such variants in neurodevelopmental disorders. We identify biological processes relevant to schizophrenia pathophysiology; show convergence of common and rare variant associations in schizophrenia and neurodevelopmental disorders; and provide a resource of prioritized genes and variants to advance mechanistic studies

Mapping genomic loci prioritises genes and implicates synaptic biology in schizophrenia

Schizophrenia has a heritability of 60–80%1, much of which is attributable to common risk alleles. Here, in a two-stage genome-wide association study of up to 76,755 individuals with schizophrenia and 243,649 control individuals, we report common variant associations at 287 distinct genomic loci. Associations were concentrated in genes that are expressed in excitatory and inhibitory neurons of the central nervous system, but not in other tissues or cell types. Using fine-mapping and functional genomic data, we identify 120 genes (106 protein-coding) that are likely to underpin associations at some of these loci, including 16 genes with credible causal non-synonymous or untranslated region variation. We also implicate fundamental processes related to neuronal function, including synaptic organization, differentiation and transmission. Fine-mapped candidates were enriched for genes associated with rare disruptive coding variants in people with schizophrenia, including the glutamate receptor subunit GRIN2A and transcription factor SP4, and were also enriched for genes implicated by such variants in neurodevelopmental disorders. We identify biological processes relevant to schizophrenia pathophysiology; show convergence of common and rare variant associations in schizophrenia and neurodevelopmental disorders; and provide a resource of prioritized genes and variants to advance mechanistic studies